Large RNase T1-resistant oligonucleotides encoding p15E and the U3 region of the long terminal repeat distinguish two biological classes of mink cell focus-forming type C viruses of inbred mice.
about
Virological events leading to spontaneous AKR thymomasThe four classes of endogenous murine leukemia virus: structural relationships and potential for recombinationAn increase in disease latency is associated with a host-dependent selection for recombinant murine leukemia viruses with substitutions in the p15E (TM) gene.Origin of pathogenic determinants of recombinant murine leukemia viruses: analysis of Bxv-1-related xenotropic viruses from CWD mice.Influence of enhancer sequences on thymotropism and leukemogenicity of mink cell focus-forming viruses.Mechanism of selection of class II recombinant murine leukemia viruses in the highly leukemic strain CWD.AKR ecotropic murine leukemia virus SL3-3 forms envelope gene recombinants in vivoAssociation of recombinant murine leukemia viruses of the class II genotype with spontaneous lymphomas in CWD mice.Role of recombinant ecotropic and polytropic viruses in the development of spontaneous thymic lymphomas in HRS/J miceThe E47 transcription factor binds to the enhancer sequences of recombinant murine leukemia viruses and influences enhancer functionThe mouse H-2A region influences the envelope gene structure of tumor-associated murine leukemia viruses.Comparison of two host cell range variants of feline immunodeficiency virus.Characterization of endogenous and recombinant proviral elements of a highly tumorigenic AKR cell line.Mink cell focus-forming murine leukemia virus infection induces apoptosis of thymic lymphocytes.Defective virus is associated with induction of murine retrovirus-induced immunodeficiency syndrome.Origins of the endogenous and infectious laboratory mouse gammaretroviruses.Stages in development of mink cell focus-inducing (MCF) virus-accelerated leukemia in AKR mice.Characterization of epitopes defining two major subclasses of polytropic murine leukemia viruses (MuLVs) which are differentially expressed in mice infected with different ecotropic MuLVsA direct demonstration of recombination between an injected virus and endogenous viral sequences, resulting in the generation of mink cell focus-inducing viruses in AKR miceAlignment of U3 region sequences of mammalian type C viruses: identification of highly conserved motifs and implications for enhancer designPoint mutations in the Moloney murine leukemia virus enhancer identify a lymphoid-specific viral core motif and 1,3-phorbol myristate acetate-inducible element.Macrophages are the first thymic cells to express polytropic retrovirus in AKR mouse leukemogenesisGenetic identification of endogenous polytropic proviruses by using recombinant inbred mice.Nuclear factors that bind to the enhancer region of nondefective Friend murine leukemia virus.Normal expression of polymorphic endogenous retroviral RNA containing segments identical to mink cell focus-forming virusExpression of mink cell focus-forming murine leukemia virus-related transcripts in AKR miceClass II polytropic murine leukemia viruses (MuLVs) of AKR/J mice: possible role in the generation of class I oncogenic polytropic MuLVs.Leukemia induction by a new strain of Friend mink cell focus-inducing virus: synergistic effect of Friend ecotropic murine leukemia virusA 3' end fragment encompassing the transcriptional enhancers of nondefective Friend virus confers erythroleukemogenicity on Moloney leukemia virus.The tandem direct repeats within the long terminal repeat of murine leukemia viruses are the primary determinant of their leukemogenic potential.Proviral genome of radiation leukemia virus: molecular cloning of biologically active proviral DNA and nucleotide sequence of its long terminal repeatConstruction of recombinants between molecular clones of murine retrovirus MCF 247 and Akv: determinant of an in vitro host range property that maps in the long terminal repeatAt least four viral genes contribute to the leukemogenicity of murine retrovirus MCF 247 in AKR miceThe high leukemogenic potential of Gross passage A murine leukemia virus maps in the region of the genome corresponding to the long terminal repeat and to the 3' end of env.Thymic epithelial genotype influences the production of recombinant leukemogenic retroviruses in mice.Localization of the leukemogenic determinants of SL3-3, an ecotropic, XC-positive murine leukemia virus of AKR mouse origin.Nucleotide sequence of the gp70 gene of murine retrovirus MCF 247.Evidence for a new form of retroviral env transcript in leukemic and normal mouse lymphoid cells.Generation of mink cell focus-forming viruses by Friend murine leukemia virus: recombination with specific endogenous proviral sequences.Free and integrated recombinant murine leukemia virus DNAs appear in preleukemic thymuses of AKR/J mice.
P2860
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P2860
Large RNase T1-resistant oligonucleotides encoding p15E and the U3 region of the long terminal repeat distinguish two biological classes of mink cell focus-forming type C viruses of inbred mice.
description
1983 nî lūn-bûn
@nan
1983年の論文
@ja
1983年論文
@yue
1983年論文
@zh-hant
1983年論文
@zh-hk
1983年論文
@zh-mo
1983年論文
@zh-tw
1983年论文
@wuu
1983年论文
@zh
1983年论文
@zh-cn
name
Large RNase T1-resistant oligo ...... type C viruses of inbred mice.
@en
type
label
Large RNase T1-resistant oligo ...... type C viruses of inbred mice.
@en
prefLabel
Large RNase T1-resistant oligo ...... type C viruses of inbred mice.
@en
P2093
P2860
P1433
P1476
Large RNase T1-resistant oligo ...... type C viruses of inbred mice
@en
P2093
J W Hartley
N H Hopkins
P2860
P304
P407
P577
1983-01-01T00:00:00Z