Interaction of the Lyme disease spirochete Borrelia burgdorferi with brain parenchyma elicits inflammatory mediators from glial cells as well as glial and neuronal apoptosis
about
Cytokines and chemokines at the crossroads of neuroinflammation, neurodegeneration, and neuropathic painLyme disease and pediatric autoimmune neuropsychiatric disorders associated with streptococcal infections (PANDAS): an overviewHerpes Simplex Virus Type 1 and Other Pathogens are Key Causative Factors in Sporadic Alzheimer's DiseaseMicroglia are mediators of Borrelia burgdorferi-induced apoptosis in SH-SY5Y neuronal cellsEstablished Population of Blacklegged Ticks with High Infection Prevalence for the Lyme Disease Bacterium, Borrelia burgdorferi Sensu Lato, on Corkscrew Island, Kenora District, OntarioPrevalence of the Lyme Disease Spirochete, Borrelia burgdorferi, in Blacklegged Ticks, Ixodes scapularis at Hamilton-Wentworth, OntarioAnti-inflammatory effects of dexamethasone and meloxicam on Borrelia burgdorferi-induced inflammation in neuronal cultures of dorsal root ganglia and myelinating cells of the peripheral nervous systemReceptor tyrosine kinases play a significant role in human oligodendrocyte inflammation and cell death associated with the Lyme disease bacterium Borrelia burgdorferi.Microglia in infectious diseases of the central nervous system.Regulated Production of CXCL13 within the Central Nervous System.Type-I interferons suppress microglial production of the lymphoid chemokine, CXCL13.Activated microglia contribute to neuronal apoptosis in Toxoplasmic encephalitis.Survey on cattle ticks in Nur, north of Iran.Inflammatory mediator release from primary rhesus microglia in response to Borrelia burgdorferi results from the activation of several receptors and pathwaysInflammation in the pathogenesis of lyme neuroborreliosisInterleukin-10 alters effector functions of multiple genes induced by Borrelia burgdorferi in macrophages to regulate Lyme disease inflammationA possible role for inflammation in mediating apoptosis of oligodendrocytes as induced by the Lyme disease spirochete Borrelia burgdorferi.MicroRNA and mRNA Transcriptome Profiling in Primary Human Astrocytes Infected with Borrelia burgdorferiReview: apoptotic mechanisms in bacterial infections of the central nervous system.Cerebrospinal fluid proteome of patients with acute Lyme diseaseAntagonist of the neurokinin-1 receptor curbs neuroinflammation in ex vivo and in vitro models of Lyme neuroborreliosis.Acetate supplementation reduces microglia activation and brain interleukin-1β levels in a rat model of Lyme neuroborreliosisCXCL13 drives spinal astrocyte activation and neuropathic pain via CXCR5.Targeting CXCL13 During NeuroinflammationThe Lyme disease spirochete Borrelia burgdorferi induces inflammation and apoptosis in cells from dorsal root gangliaThe multifaceted responses of primary human astrocytes and brain microvascular endothelial cells to the Lyme disease spirochete, Borrelia burgdorferi.Bacterial Amyloid and DNA are Important Constituents of Senile Plaques: Further Evidence of the Spirochetal and Biofilm Nature of Senile PlaquesPossible role of glial cells in the onset and progression of Lyme neuroborreliosis.Immunogenetic Markers Definition in Latvian Patients with Lyme Borreliosis and Lyme Neuroborreliosis.The Lyme Disease Pathogen Borrelia burgdorferi Infects Murine Bone and Induces Trabecular Bone Loss.Effects of dexamethasone and meloxicam on Borrelia burgdorferi-induced inflammation in glial and neuronal cells of the central nervous system.The MEK/ERK pathway is the primary conduit for Borrelia burgdorferi-induced inflammation and P53-mediated apoptosis in oligodendrocytes.Detection of Lyme Disease Bacterium, Borrelia burgdorferi sensu lato, in Blacklegged Ticks Collected in the Grand River Valley, Ontario, Canada.The Putative Role of Viruses, Bacteria, and Chronic Fungal Biotoxin Exposure in the Genesis of Intractable Fatigue Accompanied by Cognitive and Physical Disability.Application of "Omics" Technologies for Diagnosis and Pathogenesis of Neurological Infections.Inflammasome Involvement in Alzheimer's Disease.Retrospective Evaluation of Horses Diagnosed with Neuroborreliosis on Postmortem Examination: 16 Cases (2004-2015).Oral inflammation and infection, and chronic medical diseases: implications for the elderly.Repetitive hypoxic preconditioning induces an immunosuppressed B cell phenotype during endogenous protection from stroke.Glial Cell-Elicited Activation of Brain Microvasculature in Response to Brucella abortus Infection Requires ASC Inflammasome-Dependent IL-1β Production.
P2860
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P2860
Interaction of the Lyme disease spirochete Borrelia burgdorferi with brain parenchyma elicits inflammatory mediators from glial cells as well as glial and neuronal apoptosis
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
2008年论文
@zh
2008年论文
@zh-cn
name
Interaction of the Lyme diseas ...... s glial and neuronal apoptosis
@en
type
label
Interaction of the Lyme diseas ...... s glial and neuronal apoptosis
@en
prefLabel
Interaction of the Lyme diseas ...... s glial and neuronal apoptosis
@en
P2093
P2860
P1476
Interaction of the Lyme diseas ...... s glial and neuronal apoptosis
@en
P2093
Andrew A Lackner
Deepak Kaushal
Geeta Ramesh
Jason Dufour
Juan T Borda
Mario T Philipp
Ramesh Ramamoorthy
P2860
P304
P356
10.2353/AJPATH.2008.080483
P407
P577
2008-10-02T00:00:00Z