Changes in thyroid status during perinatal development of MCT8-deficient male mice. - Wikidata - awgldk - TriplyDB

Changes in thyroid status during perinatal development of MCT8-deficient male mice.

Changes in thyroid status during perinatal development of MCT8-deficient male mice.

http://www.wikidata.org/entity/Q36946053

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Altered behavioral performance and live imaging of circuit-specific neural deficiencies in a zebrafish model for psychomotor retardation Cerebral cortex hyperthyroidism of newborn mct8-deficient mice transiently suppressed by lat2 inactivation.A novel mutation in the Albumin gene (R218S) causing familial dysalbuminemic hyperthyroxinemia in a family of Bangladeshi extraction.Placenta passage of the thyroid hormone analog DITPA to male wild-type and Mct8-deficient mice.Increased oxidative metabolism and neurotransmitter cycling in the brain of mice lacking the thyroid hormone transporter SLC16A2 (MCT8)Desensitization and Incomplete Recovery of Hepatic Target Genes After Chronic Thyroid Hormone Treatment and Withdrawal in Male Adult Mice Thyroid Hormone Signaling in Male Mouse Skeletal Muscle Is Largely Independent of D2 in Myocytes.The Thyroid Hormone Analog DITPA Ameliorates Metabolic Parameters of Male Mice With Mct8 Deficiency Coupling between Nutrient Availability and Thyroid Hormone Activation The Type 3 Deiodinase Is a Critical Determinant of Appropriate Thyroid Hormone Action in the Developing Testis.Mct8-deficient mice have increased energy expenditure and reduced fat mass that is abrogated by normalization of serum T3 levels.Pharmacological treatment and BBB-targeted genetic therapy for MCT8-dependent hypomyelination in zebrafish.American Thyroid Association Guide to investigating thyroid hormone economy and action in rodent and cell models.Thyroid hormones, t3 and t4, in the brain.Thyroid hormone action: astrocyte-neuron communication.Thyroid hormone transporters--functions and clinical implications.Role of the Thyroid System in Myelination and Neural Connectivity.Thyroid Hormone Economy in the Perinatal Mouse Brain: Implications for Cerebral Cortex Development.Adeno Associated Virus 9-Based Gene Therapy Delivers a Functional Monocarboxylate Transporter 8, Improving Thyroid Hormone Availability to the Brain of Mct8-Deficient Mice.Thyroid Hormone Availability and Action during Brain Development in Rodents.MCT8 Deficiency in Male Mice Mitigates the Phenotypic Abnormalities Associated With the Absence of a Functional Type 3 Deiodinase Oncogene-induced senescence and its evasion in a mouse model of thyroid neoplasia.An Essential Physiological Role for MCT8 in Bone in Male Mice.Fetal Exposure to High Maternal Thyroid Hormone Levels Causes Central Resistance to Thyroid Hormone in Adult Humans and Mice.GLIS3 is indispensable for TSH/TSHR-dependent thyroid hormone biosynthesis and follicular cell proliferation.Thyroid Hormone Metabolism Defects in a Mouse Model of SBP2 Deficiency.TRH Action Is Impaired in Pituitaries of Male IGSF1-Deficient Mice.The Value of Comprehensive Thyroid Function Testing and Family History for Early Diagnosis of MCT8 Deficiency.Changes in Hepatic TRβ Protein Expression, Lipogenic Gene Expression, and Long-Chain Acylcarnitine Levels During Chronic Hyperthyroidism and Triiodothyronine Withdrawal in a Mouse Model.Deafness and loss of cochlear hair cells in the absence of thyroid hormone transporters Slc16a2 (Mct8) and Slc16a10 (Mct10).

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P2860

Changes in thyroid status during perinatal development of MCT8-deficient male mice.

http://www.wikidata.org/entity/Q36946053

description

2013 nî lūn-bûn

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2013年の論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

@zh-tw

2013年论文

@wuu

2013年论文

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2013年论文

@zh-cn

name

Changes in thyroid status during perinatal development of MCT8-deficient male mice.

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type

label

Changes in thyroid status during perinatal development of MCT8-deficient male mice.

@en

prefLabel

Changes in thyroid status during perinatal development of MCT8-deficient male mice.

@en

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Changes in thyroid status during perinatal development of MCT8-deficient male mice.

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P2093

Alexandra M Dumitrescu

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Alfonso Massimiliano Ferrara

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Juan Bernal

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Pilar Gil-Ibáñez

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Roy E Weiss

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Samuel Refetoff

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Teresa Marcinkowski

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Xiao-Hui Liao

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P2860

A novel syndrome combining thyroid and neurological abnormalities is associated with mutations in a monocarboxylate transporter gene

Thyroid hormone-regulated mouse cerebral cortex genes are differentially dependent on the source of the hormone: a study in monocarboxylate transporter-8- and deiodinase-2-deficient mice.

Pregnant rat uterus expresses high levels of the type 3 iodothyronine deiodinase

Mice deficient in MCT8 reveal a mechanism regulating thyroid hormone secretion.

Biochemistry, cellular and molecular biology, and physiological roles of the iodothyronine selenodeiodinases.

Effects of ligand and thyroid hormone receptor isoforms on hepatic gene expression profiles of thyroid hormone receptor knockout mice.

Association between mutations in a thyroid hormone transporter and severe X-linked psychomotor retardation.

Thyroid hormones and brain development.

The role of thyroid hormone in fetal neurodevelopment.

Distinct roles of deiodinases on the phenotype of Mct8 defect: a comparison of eight different mouse genotypes.

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2533-2541

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scholarly article

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10.1210/EN.2012-2031

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7

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154

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2013-05-21T00:00:00Z

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23696569

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3689279

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