Thyroid Hormone Metabolism Defects in a Mouse Model of SBP2 Deficiency.

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Thyroid Hormone Metabolism Defects in a Mouse Model of SBP2 Deficiency.

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2017 nî lūn-bûn

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2017年の論文

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年學術文章

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Thyroid Hormone Metabolism Defects in a Mouse Model of SBP2 Deficiency.

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Thyroid Hormone Metabolism Defects in a Mouse Model of SBP2 Deficiency.

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Thyroid Hormone Metabolism Defects in a Mouse Model of SBP2 Deficiency.

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Thyroid Hormone Metabolism Defects in a Mouse Model of SBP2 Deficiency.

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Thyroid Hormone Metabolism Defects in a Mouse Model of SBP2 Deficiency.

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Thyroid Hormone Metabolism Defects in a Mouse Model of SBP2 Deficiency.

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P1476

Thyroid Hormone Metabolism Defects in a Mouse Model of SBP2 Deficiency.

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P2093

Alexandra M Dumitrescu

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Benjamin Follman

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Haruki Fujisawa

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Jiao Fu

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Xiao-Hui Liao

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P2860

A rapid and sensitive method for the quantitation of microgram quantities of protein utilizing the principle of protein-dye binding

Resistance to thyroid hormone caused by two mutant thyroid hormone receptors beta, R243Q and R243W, with marked impairment of function that cannot be explained by altered in vitro 3,5,3'-triiodothyroinine binding affinity

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The RNA-binding protein Secisbp2 differentially modulates UGA codon reassignment and RNA decay.

Biochemistry, cellular and molecular biology, and physiological roles of the iodothyronine selenodeiodinases.

Mutations in the selenocysteine insertion sequence-binding protein 2 gene lead to a multisystem selenoprotein deficiency disorder in humans.

Mutations in SECISBP2 result in abnormal thyroid hormone metabolism.

Critical role of types 2 and 3 deiodinases in the negative regulation of gene expression by T₃in the mouse cerebral cortex.

Changes in thyroid status during perinatal development of MCT8-deficient male mice.

P304

4317-4330

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P31

scholarly article

P356

10.1210/EN.2017-00618

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P407

English

P433

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P478

158

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P577

2017-09-28T00:00:00Z

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P698

29029094

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P932

5711384

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