Derepressed hyphal growth and reduced virulence in a VH1 family-related protein phosphatase mutant of the human pathogen Candida albicans.
about
Candida glabrata: new tools and technologies-expanding the toolkitBiochemical properties and possible roles of ectophosphatase activities in fungiRegulatory roles of phosphorylation in model and pathogenic fungiIdentification of Candida albicans genes induced during thrush offers insight into pathogenesis.Insertional mutagenesis enables cleistothecial formation in a non-mating strain of Histoplasma capsulatum.The G protein-coupled receptor Gpr1 and the Galpha protein Gpa2 act through the cAMP-protein kinase A pathway to induce morphogenesis in Candida albicans.Roles of the Candida albicans mitogen-activated protein kinase homolog, Cek1p, in hyphal development and systemic candidiasisIron-responsive chromatin remodelling and MAPK signalling enhance adhesion in Candida albicans.Signaling through adenylyl cyclase is essential for hyphal growth and virulence in the pathogenic fungus Candida albicans.Repression of hyphal proteinase expression by the mitogen-activated protein (MAP) kinase phosphatase Cpp1p of Candida albicans is independent of the MAP kinase Cek1p.Attenuation of virulence and changes in morphology in Candida albicans by disruption of the N-acetylglucosamine catabolic pathwaySignal transduction cascades regulating fungal development and virulence.Pph3 dephosphorylation of Rad53 is required for cell recovery from MMS-induced DNA damage in Candida albicans.Candida albicans cell-type switching and functional plasticity in the mammalian host.Linking fungal morphogenesis with virulence.Ppg1, a PP2A-type protein phosphatase, controls filament extension and virulence in Candida albicans.TUP1, CPH1 and EFG1 make independent contributions to filamentation in candida albicans.Candida albicans OPI1 regulates filamentous growth and virulence in vaginal infections, but not inositol biosynthesis.Calmodulin binding to Dfi1p promotes invasiveness of Candida albicansMolecular genetic and genomic approaches to the study of medically important fungiA Candida albicans cell wall-linked protein promotes invasive filamentation into semi-solid medium.Purification and characterization of an autoregulatory substance capable of regulating the morphological transition in Candida albicansCandida albicans Cek1 mitogen-activated protein kinase signaling enhances fungicidal activity of salivary histatin 5Critical role of DNA checkpoints in mediating genotoxic-stress-induced filamentous growth in Candida albicans.Hbr1 Activates and Represses Hyphal Growth in Candida albicans and Regulates Fungal Morphogenesis under Embedded Conditions.Identification of the putative protein phosphatase gene PTC1 as a virulence-related gene using a silkworm model of Candida albicans infectionEctopic expression of URA3 can influence the virulence phenotypes and proteome of Candida albicans but can be overcome by targeted reintegration of URA3 at the RPS10 locus.Role of the mitogen-activated protein kinase Hog1p in morphogenesis and virulence of Candida albicansHWP1 functions in the morphological development of Candida albicans downstream of EFG1, TUP1, and RBF1.Morphogenesis, adhesive properties, and antifungal resistance depend on the Pmt6 protein mannosyltransferase in the fungal pathogen candida albicans.Defective hyphal development and avirulence caused by a deletion of the SSK1 response regulator gene in Candida albicans.The Sho1 adaptor protein links oxidative stress to morphogenesis and cell wall biosynthesis in the fungal pathogen Candida albicans.Differential susceptibility of mitogen-activated protein kinase pathway mutants to oxidative-mediated killing by phagocytes in the fungal pathogen Candida albicans.The Cek1 and Hog1 mitogen-activated protein kinases play complementary roles in cell wall biogenesis and chlamydospore formation in the fungal pathogen Candida albicans.Msb2 signaling mucin controls activation of Cek1 mitogen-activated protein kinase in Candida albicans.Ras links cellular morphogenesis to virulence by regulation of the MAP kinase and cAMP signalling pathways in the pathogenic fungus Candida albicans.The serine/threonine protein phosphatase SIT4 modulates yeast-to-hypha morphogenesis and virulence in Candida albicans.The dual specificity phosphatase Rok1 negatively regulates mating and pathogenicity in Ustilago maydis.Rad6p represses yeast-hypha morphogenesis in the human fungal pathogen Candida albicans.Over-expression of Candida albicans mitochondrial ribosomal protein S9 (MrpS9p) disturbs mitochondrial function in Saccharomyces cerevisiae.
P2860
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P2860
Derepressed hyphal growth and reduced virulence in a VH1 family-related protein phosphatase mutant of the human pathogen Candida albicans.
description
1997 nî lūn-bûn
@nan
1997年の論文
@ja
1997年論文
@yue
1997年論文
@zh-hant
1997年論文
@zh-hk
1997年論文
@zh-mo
1997年論文
@zh-tw
1997年论文
@wuu
1997年论文
@zh
1997年论文
@zh-cn
name
Derepressed hyphal growth and ...... man pathogen Candida albicans.
@en
type
label
Derepressed hyphal growth and ...... man pathogen Candida albicans.
@en
prefLabel
Derepressed hyphal growth and ...... man pathogen Candida albicans.
@en
P2093
P2860
P356
P1476
Derepressed hyphal growth and ...... man pathogen Candida albicans.
@en
P2093
Röllinghoff M
Schröppel K
Whiteway M
P2860
P304
P356
10.1091/MBC.8.12.2539
P577
1997-12-01T00:00:00Z