Chronic NMDA administration increases neuroinflammatory markers in rat frontal cortex: cross-talk between excitotoxicity and neuroinflammation. - Wikidata - awgldk - TriplyDB

Chronic NMDA administration increases neuroinflammatory markers in rat frontal cortex: cross-talk between excitotoxicity and neuroinflammation.

Chronic NMDA administration increases neuroinflammatory markers in rat frontal cortex: cross-talk between excitotoxicity and neuroinflammation.

http://www.wikidata.org/entity/Q36953315

about

Bipolar disorder and mechanisms of action of mood stabilizers Implications of glial nitric oxide in neurodegenerative diseases Lithium and the other mood stabilizers effective in bipolar disorder target the rat brain arachidonic acid cascade Growth factors in synaptic function.Chronic NMDA administration to rats increases brain pro-apoptotic factors while decreasing anti-Apoptotic factors and causes cell death.Altered expression of apoptotic factors and synaptic markers in postmortem brain from bipolar disorder patients.Mood-stabilizers target the brain arachidonic acid cascade.Increased excitotoxicity and neuroinflammatory markers in postmortem frontal cortex from bipolar disorder patients.Extracellular-derived calcium does not initiate in vivo neurotransmission involving docosahexaenoic acid.Imaging upregulated brain arachidonic acid metabolism in HIV-1 transgenic rats Upregulated expression of brain enzymatic markers of arachidonic and docosahexaenoic acid metabolism in a rat model of the metabolic syndrome Valproate uncompetitively inhibits arachidonic acid acylation by rat acyl-CoA synthetase 4: relevance to valproate's efficacy against bipolar disorder Altered arachidonic acid cascade enzymes in postmortem brain from bipolar disorder patients Imaging brain signal transduction and metabolism via arachidonic and docosahexaenoic acid in animals and humans.Dietary n-6 polyunsaturated fatty acid deprivation increases docosahexaenoic acid metabolism in rat brain.Epigenetic modifications in frontal cortex from Alzheimer's disease and bipolar disorder patients.Neuroinflammation and synaptic loss Propylisopropylacetic acid (PIA), a constitutional isomer of valproic acid, uncompetitively inhibits arachidonic acid acylation by rat acyl-CoA synthetase 4: a potential drug for bipolar disorder Intracellular- and extracellular-derived Ca(2+) influence phospholipase A(2)-mediated fatty acid release from brain phospholipids.Targeting NADPH oxidase and phospholipases A2 in Alzheimer's disease.Curcumin-loaded embryonic stem cell exosomes restored neurovascular unit following ischemia-reperfusion injury.Do omega-6 and trans fatty acids play a role in complex regional pain syndrome? A pilot study.Glutamate homeostasis in the adult rat prefrontal cortex is altered by cortical docosahexaenoic acid accrual during adolescence: An in vivo1H MRS study.Neuroprotection by mefenamic acid against D-serine: involvement of oxidative stress, inflammation and apoptosis.

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P2860

Chronic NMDA administration increases neuroinflammatory markers in rat frontal cortex: cross-talk between excitotoxicity and neuroinflammation.

http://www.wikidata.org/entity/Q36953315

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Hyung-Wook Kim

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Jagadeesh S Rao

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Yunyoung C Chang

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P2860

Chronic brain inflammation leads to a decline in hippocampal NMDA-R1 receptors

5'-flanking region surrounding a human cytosolic phospholipase A2 gene

Diversity of group types, regulation, and function of phospholipase A2

Analysis of the convulsant-potentiating effects of lithium in rats

Regulation of NMDA receptor trafficking by amyloid-beta

Chronic administration of carbamazepine down-regulates AP-2 DNA-binding activity and AP-2alpha protein expression in rat frontal cortex

Decrease in the AP-2 DNA-binding activity and in the protein expression of AP-2 alpha and AP-2 beta in frontal cortex of rats treated with lithium for 6 weeks

Brain lesions, obesity, and other disturbances in mice treated with monosodium glutamate.

Cell-specific expression of the mouse glial fibrillary acidic protein gene: identification of the cis- and trans-acting promoter elements for astrocyte-specific expression.

Early events in the trafficking of N-methyl-D-aspartate (NMDA) receptors.

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