T cell-mediated immunity in the lung: a Cryptococcus neoformans pulmonary infection model using SCID and athymic nude mice.
about
Host immunity to Cryptococcus neoformansRole of sphingomyelin synthase in controlling the antimicrobial activity of neutrophils against Cryptococcus neoformansInvolvement of CD14, toll-like receptors 2 and 4, and MyD88 in the host response to the fungal pathogen Cryptococcus neoformans in vivoCryptococcus neoformans enters the endolysosomal pathway of dendritic cells and is killed by lysosomal components.Insights into the mechanisms of protective immunity against Cryptococcus neoformans infection using a mouse model of pulmonary cryptococcosisScavenger receptor A modulates the immune response to pulmonary Cryptococcus neoformans infection.Differential host susceptibility to intracerebral infections with Candida albicans and Cryptococcus neoformans.Accumulation of CD11b+ lung dendritic cells in response to fungal infection results from the CCR2-mediated recruitment and differentiation of Ly-6Chigh monocytes.Microevolution of a standard strain of Cryptococcus neoformans resulting in differences in virulence and other phenotypes.Decreased resistance to primary intravenous Cryptococcus neoformans infection in aged mice despite adequate resistance to intravenous rechallenge.Intranasal vaccination induces protective immunity against intranasal infection with virulent Francisella tularensis biovar ACharacterization of IL-22 and antimicrobial peptide production in mice protected against pulmonary Cryptococcus neoformans infection.Role of IL-17A on resolution of pulmonary C. neoformans infectionUrokinase-type plasminogen activator in inflammatory cell recruitment and host defense against Pneumocystis carinii in mice.Requirement for CD4(+) T lymphocytes in host resistance against Cryptococcus neoformans in the central nervous system of immunized mice.Phagocytosis and protein processing are required for presentation of Cryptococcus neoformans mitogen to T lymphocytesBoth Th1 and Th2 cytokines affect the ability of monoclonal antibodies to protect mice against Cryptococcus neoformans.TLR9 signaling is required for generation of the adaptive immune protection in Cryptococcus neoformans-infected lungs.Cryptococcus neoformans modulates extracellular killing by neutrophils.Primary dendritic cells phagocytose Cryptococcus neoformans via mannose receptors and Fcgamma receptor II for presentation to T lymphocytesImmune modulation mediated by cryptococcal laccase promotes pulmonary growth and brain dissemination of virulent Cryptococcus neoformans in mice.Regulation of cytokine production during the expression phase of the anticryptococcal delayed-type hypersensitivity response.Anticryptococcal resistance in the mouse brain: beneficial effects of local administration of heat-inactivated yeast cells.Pathogenesis of pulmonary Cryptococcus neoformans infection in the ratIn vivo role of dendritic cells in a murine model of pulmonary cryptococcosisChemokine receptor 2-mediated accumulation of fungicidal exudate macrophages in mice that clear cryptococcal lung infection.Prior exposure to live Mycobacterium bovis BCG decreases Cryptococcus neoformans-induced lung eosinophilia in a gamma interferon-dependent mannerEffects of immunization with Cryptococcus neoformans cells or cryptococcal culture filtrate antigen on direct anticryptococcal activities of murine T lymphocytesRole of tumor necrosis factor and gamma interferon in acquired resistance to Cryptococcus neoformans in the central nervous system of mice.A 77-kilodalton protein of Cryptococcus neoformans, a member of the heat shock protein 70 family, is a major antigen detected in the sera of mice with pulmonary cryptococcosis.Genome-Wide Transcription Study of Cryptococcus neoformans H99 Clinical Strain versus Environmental Strains.Isolation and purification of antigenic components of CryptococcusEarly induction of CCL7 downstream of TLR9 signaling promotes the development of robust immunity to cryptococcal infection.The relative susceptibility of mouse strains to pulmonary Cryptococcus neoformans infection is associated with pleiotropic differences in the immune response.Phenotypic switching of Cryptococcus neoformans occurs in vivo and influences the outcome of infectionT cells cooperate with passive antibody to modify Cryptococcus neoformans infection in mice.Vaccine and immunotherapeutic approaches for the prevention of cryptococcosis: lessons learned from animal models.CD4+ T cells cause multinucleated giant cells to form around Cryptococcus neoformans and confine the yeast within the primary site of infection in the respiratory tractThe Cnes2 locus on mouse chromosome 17 regulates host defense against cryptococcal infection through pleiotropic effects on host immunityMechanisms of dendritic cell lysosomal killing of Cryptococcus.
P2860
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P2860
T cell-mediated immunity in the lung: a Cryptococcus neoformans pulmonary infection model using SCID and athymic nude mice.
description
1991 nî lūn-bûn
@nan
1991年の論文
@ja
1991年論文
@yue
1991年論文
@zh-hant
1991年論文
@zh-hk
1991年論文
@zh-mo
1991年論文
@zh-tw
1991年论文
@wuu
1991年论文
@zh
1991年论文
@zh-cn
name
T cell-mediated immunity in th ...... ng SCID and athymic nude mice.
@en
type
label
T cell-mediated immunity in th ...... ng SCID and athymic nude mice.
@en
prefLabel
T cell-mediated immunity in th ...... ng SCID and athymic nude mice.
@en
P2093
P2860
P1476
T cell-mediated immunity in th ...... ng SCID and athymic nude mice.
@en
P2093
G B Huffnagle
M F Lipscomb
P2860
P304
P407
P577
1991-04-01T00:00:00Z