Cognitive and neuroinflammatory consequences of mild repeated stress are exacerbated in aged mice. - Wikidata - awgldk - TriplyDB

Cognitive and neuroinflammatory consequences of mild repeated stress are exacerbated in aged mice.

Cognitive and neuroinflammatory consequences of mild repeated stress are exacerbated in aged mice.

http://www.wikidata.org/entity/Q36966884

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Aging-related changes in neuroimmune-endocrine function: implications for hippocampal-dependent cognition Damaging effects of a high-fat diet to the brain and cognition: a review of proposed mechanisms Responses to environmental enrichment differ with sex and genotype in a transgenic mouse model of Huntington's disease The yin and yang of microglia Neuroinflammation in the normal aging hippocampus Neuroinflammation and cerebrovascular disease in old age: a translational medicine perspective Exercise induces age-dependent changes on epigenetic parameters in rat hippocampus: a preliminary study Maintaining brain health by monitoring inflammatory processes: a mechanism to promote successful aging 11beta-hydroxysteroid dehydrogenase type 1 expression is increased in the aged mouse hippocampus and parietal cortex and causes memory impairments.Old Maids: Aging and Its Impact on Microglia Function.Neuroinflammatory challenges compromise neuronal function in the aging brain: Postoperative cognitive delirium and Alzheimer's disease.Age-related defects in sensorimotor activity, spatial learning, and memory in C57BL/6 mice.Memory impairments in healthy aging: Role of aging-induced microglial sensitization.Strength and vulnerability integration: a model of emotional well-being across adulthood.Immunity, aging, and geriatric depression The inflammation hypothesis in geriatric depression Concurrent hippocampal induction of MHC II pathway components and glial activation with advanced aging is not correlated with cognitive impairment.Chronic stress exacerbates tau pathology, neurodegeneration, and cognitive performance through a corticotropin-releasing factor receptor-dependent mechanism in a transgenic mouse model of tauopathy Dietary broccoli mildly improves neuroinflammation in aged mice but does not reduce lipopolysaccharide-induced sickness behavior A lifespan approach to neuroinflammatory and cognitive disorders: a critical role for glia.Stress-induced impairments in prefrontal-mediated behaviors and the role of the N-methyl-D-aspartate receptor.Intracisternal interleukin-1 receptor antagonist prevents postoperative cognitive decline and neuroinflammatory response in aged rats.Cognitive dysfunction with aging and the role of inflammation Microglial aging in the healthy CNS: phenotypes, drivers, and rejuvenation Behavioral model for assessing cognitive decline.Microglia during development and aging.Effects of an alpha7 nicotinic receptor agonist and stress on spatial memory in an animal model of Alzheimer's disease.Age-dependent exacerbation of white matter stroke outcomes: a role for oxidative damage and inflammatory mediators.Neuroinflammation associated with aging sensitizes the brain to the effects of infection or stress.Sustained expression of interleukin-1beta in mouse hippocampus impairs spatial memory Systemic TNF-α produces acute cognitive dysfunction and exaggerated sickness behavior when superimposed upon progressive neurodegeneration.An Integrated Review of Psychological Stress in Parkinson's Disease: Biological Mechanisms and Symptom and Health Outcomes Soyo-san reduces depressive-like behavior and proinflammatory cytokines in ovariectomized female rats.Dysregulated neuronal-microglial cross-talk during aging, stress and inflammation.Review: microglia of the aged brain: primed to be activated and resistant to regulation.Cerebral dysfunction after coronary artery bypass surgery.Feeding the beast: can microglia in the senescent brain be regulated by diet?Neuroinflammation: the devil is in the details.Does the Interplay Between Aging and Neuroinflammation Modulate Alzheimer's Disease Clinical Phenotypes? A Clinico-Pathological Perspective.Luteolin inhibits microglia and alters hippocampal-dependent spatial working memory in aged mice.

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P2860

Cognitive and neuroinflammatory consequences of mild repeated stress are exacerbated in aged mice.

http://www.wikidata.org/entity/Q36966884

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2008 nî lūn-bûn

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2008年论文

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Cognitive and neuroinflammator ...... are exacerbated in aged mice.

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Cognitive and neuroinflammator ...... are exacerbated in aged mice.

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Cognitive and neuroinflammator ...... are exacerbated in aged mice.

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J.PSYNEUEN.2008.02.013

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Psychoneuroendocrinology

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Cognitive and neuroinflammator ...... s are exacerbated in aged mice

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J B Buchanan

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J Chen

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N L Sparkman

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P2860

Associations of salivary cortisol with cognitive function in the Baltimore memory study

Twenty years of research on cytokine-induced sickness behavior

What does epidemiology tell us about risk factors for herpes zoster?

mRNA up-regulation of MHC II and pivotal pro-inflammatory genes in normal brain aging

Memory and the hippocampus: a synthesis from findings with rats, monkeys, and humans

Neuroinflammation and disruption in working memory in aged mice after acute stimulation of the peripheral innate immune system.

Stress, age, and immune function: toward a lifespan approach.

Stress, learned helplessness, and brain interleukin-1 beta.

Spatial memory performances of aged rats in the water maze predict levels of hippocampal neurogenesis

Clinical relevance of age-related immune dysfunction.

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755-765

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scholarly article

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10.1016/J.PSYNEUEN.2008.02.013

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6

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33

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Rodney W. Johnson

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2008-04-14T00:00:00Z

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