Enhanced ethanol catabolism in orphan nuclear receptor SHP-null mice.
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Animal Models of Alcoholic Liver Disease: Pathogenesis and Clinical RelevanceMetabolomics Analysis Revealed Distinct Cyclic Changes of Metabolites Altered by Chronic Ethanol-Plus-Binge and Shp Deficiency.Small heterodimer partner deficiency exacerbates binge drinking‑induced liver injury via modulation of natural killer T cell and neutrophil infiltration.Hairy and enhancer of split 6 prevents hepatic lipid accumulation through inhibition of Pparg2 expression.
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Enhanced ethanol catabolism in orphan nuclear receptor SHP-null mice.
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2016 nî lūn-bûn
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2016年の論文
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2016年論文
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2016年論文
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2016年論文
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2016年論文
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2016年論文
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name
Enhanced ethanol catabolism in orphan nuclear receptor SHP-null mice.
@en
type
label
Enhanced ethanol catabolism in orphan nuclear receptor SHP-null mice.
@en
prefLabel
Enhanced ethanol catabolism in orphan nuclear receptor SHP-null mice.
@en
P2093
P2860
P356
P1476
Enhanced ethanol catabolism in orphan nuclear receptor SHP-null mice.
@en
P2093
Jung Eun Park
Mikang Lee
Ryan Mifflin
Yoon Kwang Lee
P2860
P304
P356
10.1152/AJPGI.00343.2015
P577
2016-03-11T00:00:00Z