Zag expression during aging suppresses proliferation after kidney injury. - Wikidata - awgldk - TriplyDB

Zag expression during aging suppresses proliferation after kidney injury.

Zag expression during aging suppresses proliferation after kidney injury.

http://www.wikidata.org/entity/Q36985793

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The aging kidney: increased susceptibility to nephrotoxicity Remedial applications of silencing ribonucleic acids and modalities for its delivery to the kidneys--a review Mechanisms and consequences of injury and repair in older organ transplants Telomere shortening reduces regenerative capacity after acute kidney injury.α(E)-catenin regulates BMP-7 expression and migration in renal epithelial cells The relationship between renal function and plasma concentration of the cachectic factor zinc-alpha2-glycoprotein (ZAG) in adult patients with chronic kidney disease Acute kidney injury in elderly persons.A role for the age-dependent loss of α(E)-catenin in regulation of N-cadherin expression and cell migration Enhanced ZAG production by subcutaneous adipose tissue is linked to weight loss in gastrointestinal cancer patients Zinc-alpha2-glycoprotein in patients with acute and chronic kidney disease The duration of postoperative acute kidney injury is an additional parameter predicting long-term survival in diabetic veterans.Increased cellular senescence and vascular rarefaction exacerbate the progression of kidney fibrosis in aged mice following transient ischemic injury In vivo and in vitro analysis of age-associated changes and somatic cellular senescence in renal epithelial cells.Bβ(15-42) attenuates the effect of ischemia-reperfusion injury in renal transplantation The Newest Hypothesis about Vitiligo: Most of the Suggested Pathogeneses of Vitiligo Can Be Attributed to Lack of One Factor, Zinc-α2-Glycoprotein Zinc-α2-Glycoprotein Exerts Antifibrotic Effects in Kidney and Heart.Chitinase-like protein Brp-39/YKL-40 modulates the renal response to ischemic injury and predicts delayed allograft function.Autophagy Induces Prosenescent Changes in Proximal Tubular S3 Segments GDF11 improves tubular regeneration after acute kidney injury in elderly mice.Met activation is required for early cytoprotection after ischemic kidney injury.Pathophysiology of acute kidney injury Twist2 Is Upregulated in Early Stages of Repair Following Acute Kidney Injury.Zinc-α2-glycoprotein: an adipokine modulator of body fat mass?Renal fibrosis: insight from proteomics in animal models and human disease.New insights on molecular mechanisms of renal aging.Progress in the development of animal models of acute kidney injury and its impact on drug discovery.Mechanisms of maladaptive repair after AKI leading to accelerated kidney ageing and CKD.Renal Aging: Causes and Consequences.The impact of autophagy on the development of senescence in primary tubular epithelial cells.Adipokine zinc-alpha-2-glycoprotein as a novel urinary biomarker presents earlier than microalbuminuria in diabetic nephropathy Ischemia-reperfusion model of acute kidney injury and post injury fibrosis in mice.Renal tubular Notch signaling triggers a prosenescent state after acute kidney injury.Role of fibrinogen in acute ischemic kidney injury.Age-related decline in label-retaining tubular cells: implication for reduced regenerative capacity after injury in the aging kidney.Circulating Spexin Levels Negatively Correlate With Age, BMI, Fasting Glucose, and Triglycerides in Healthy Adult Women.

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P2860

Zag expression during aging suppresses proliferation after kidney injury.

http://www.wikidata.org/entity/Q36985793

description

2008 nî lūn-bûn

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2008年の論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

@zh-tw

2008年论文

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2008年论文

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2008年论文

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name

Zag expression during aging suppresses proliferation after kidney injury.

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type

label

Zag expression during aging suppresses proliferation after kidney injury.

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prefLabel

Zag expression during aging suppresses proliferation after kidney injury.

@en

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P1433

Journal of the American Society of Nephrology

P1476

Zag expression during aging suppresses proliferation after kidney injury.

@en

P2093

Arnaud Marlier

http://www.w3.org/2001/XMLSchema#string

Lloyd G Cantley

http://www.w3.org/2001/XMLSchema#string

Roland Schmitt

http://www.w3.org/2001/XMLSchema#string

P2860

Characterization of zinc-alpha(2)-glycoprotein as a cell adhesion molecule that inhibits the proliferation of an oral tumor cell line

Acute renal failure: definitions, diagnosis, pathogenesis, and therapy

Analysis of Relative Gene Expression Data Using Real-Time Quantitative PCR and the 2−ΔΔCT Method

Crystal structure of human ZAG, a fat-depleting factor related to MHC molecules

Preparation and properties of Zn-alpha 2-glycoprotein of normal human plasma

Increased susceptibility of aging kidney to ischemic injury: identification of candidate genes changed during aging, but corrected by caloric restriction

Characterization of ZAG protein expression in prostate cancer using a semi-automated microscope system.

Localization of proliferating cell nuclear antigen, vimentin, c-Fos, and clusterin in the postischemic kidney. Evidence for a heterogenous genetic response among nephron segments, and a large pool of mitotically active and dedifferentiated cells.

Bone marrow stem cells contribute to repair of the ischemically injured renal tubule

Gene expression profiling identifies clinically relevant subtypes of prostate cancer.

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2375-2383

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scholarly article

P356

10.1681/ASN.2008010035

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12

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19

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2008-09-24T00:00:00Z

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18815245

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2588107

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