Reperfusion activates metalloproteinases that contribute to neurovascular injury
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Administration of S-methyl-L-thiocitrulline protects against brain injuries after intracerebral hemorrhage.Peroxynitrite decomposition catalyst prevents matrix metalloproteinase activation and neurovascular injury after prolonged cerebral ischemia in rats.Brain distribution and elimination of recombinant human TIMP-1 after cerebral ischemia and reperfusion in rats.Heterogeneity in the rat brain vasculature revealed by quantitative confocal analysis of endothelial barrier antigen and P-glycoprotein expression.Engaging neuroscience to advance translational research in brain barrier biology.Matrix metalloproteinase-2 or -9 deletions protect against hemorrhagic transformation during early stage of cerebral ischemia and reperfusion.Acute and chronic vascular responses to experimental focal arterial stroke in the neonate rat.Mechanical reperfusion is associated with post-ischemic hemorrhage in rat brain.Matrix metalloproteinase-2 deletions protect against hemorrhagic transformation after 1 h of cerebral ischemia and 23 h of reperfusion.Bone marrow-derived cells are the major source of MMP-9 contributing to blood-brain barrier dysfunction and infarct formation after ischemic stroke in mice.Hemorrhagic transformation after ischemic stroke in animals and humansMatrix Metalloproteinase in Blood-Brain Barrier Breakdown in Dementia.Outcome Differences between Intra-Arterial Iso- and Low-Osmolality Iodinated Radiographic Contrast Media in the Interventional Management of Stroke III Trial.Endothelial barrier antigen-immunoreactivity is conversely associated with blood-brain barrier dysfunction after embolic stroke in ratsHemoglobin-induced oxidative stress contributes to matrix metalloproteinase activation and blood-brain barrier dysfunction in vivoCerebral Ischemic Reperfusion Injury Following Recanalization of Large Vessel Occlusions.The protective effect of berberine against neuronal damage by inhibiting matrix metalloproteinase-9 and laminin degradation in experimental autoimmune encephalomyelitis.Plasma 3-nitrotyrosine estimates the reperfusion-induced cerebrovascular stress, whereas matrix metalloproteinases mainly reflect plasma activity: a study in patients treated with thrombolysis or endovascular recanalization.Negative correlation of CD34+ cells with blood-brain barrier permeability following traumatic brain injury in a rat model.Decreased infarct volume and intracranial hemorrhage associated with intra-arterial nonionic iso-osmolar contrast material in an MCA occlusion/reperfusion model.A lipoxin A4 analog ameliorates blood-brain barrier dysfunction and reduces MMP-9 expression in a rat model of focal cerebral ischemia-reperfusion injury.Hypothermia and brain inflammation after cardiac arrest
P2860
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P2860
Reperfusion activates metalloproteinases that contribute to neurovascular injury
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
2007年论文
@zh
2007年论文
@zh-cn
name
Reperfusion activates metalloproteinases that contribute to neurovascular injury
@en
type
label
Reperfusion activates metalloproteinases that contribute to neurovascular injury
@en
prefLabel
Reperfusion activates metalloproteinases that contribute to neurovascular injury
@en
P2093
P2860
P1476
Reperfusion activates metalloproteinases that contribute to neurovascular injury
@en
P2093
Frank R Sharp
Gail J Pyne-Geithman
Joseph F Clark
Joseph P Broderick
Kenneth R Wagner
Pooja Khatri
Ruiqiong Ran
Thomas Tomsick
P2860
P304
P356
10.1016/J.EXPNEUROL.2007.12.003
P407
P577
2007-12-15T00:00:00Z