Matrix metalloproteinase-2 or -9 deletions protect against hemorrhagic transformation during early stage of cerebral ischemia and reperfusion.
about
Critical role of matrix metalloprotease-9 in chronic high fat diet-induced cerebral vascular remodelling and increase of ischaemic brain injury in mice†.17β-Estradiol Ameliorates Tight Junction Disruption via Repression of MMP Transcription.Hemostasis and alterations of the central nervous systemPlasmin-dependent modulation of the blood-brain barrier: a major consideration during tPA-induced thrombolysis?Hemorrhagic transformation after cerebral infarction: current concepts and challenges.Hemorrhagic Transformation after Tissue Plasminogen Activator Reperfusion Therapy for Ischemic Stroke: Mechanisms, Models, and Biomarkers.Matrix Metalloproteinase-2 Knockout and Heterozygote Mice Are Protected from Hydronephrosis and Kidney Fibrosis after Unilateral Ureteral Obstruction.The serum levels of MMP-9, MMP-2 and vWF in patients with low doses of urokinase peritoneal dialysis decreased uremia complicated with cerebral infarctionConnective tissue growth factor regulates retinal neovascularization through p53 protein-dependent transactivation of the matrix metalloproteinase (MMP)-2 geneTargets of vascular protection in acute ischemic stroke differ in type 2 diabetes.Matrix metalloproteinases and blood-brain barrier disruption in acute ischemic stroke.Genetic mouse models to study blood-brain barrier development and functionMatrix metalloproteinase-2 deletions protect against hemorrhagic transformation after 1 h of cerebral ischemia and 23 h of reperfusion.Progranulin deficiency promotes post-ischemic blood-brain barrier disruption2-Chloroethanol Induced Upregulation of Matrix Metalloproteinase-2 in Primary Cultured Rat Astrocytes Via MAPK Signal Pathways.Hemorrhagic transformation after ischemic stroke in animals and humansMatrix Metalloproteinase in Blood-Brain Barrier Breakdown in Dementia.New approach to identify ischemic stroke patients at risk to develop hemorrhagic transformation.High matrix metalloproteinase-9 expression induces angiogenesis and basement membrane degradation in stroke-prone spontaneously hypertensive rats after cerebral infarction.Post-ischemic treatment with azithromycin protects ganglion cells against retinal ischemia/reperfusion injury in the rat.Dual role of mitochondria in producing melatonin and driving GPCR signaling to block cytochrome c release.Cerebral Ischemic Reperfusion Injury Following Recanalization of Large Vessel Occlusions.Dexamethasone suppresses JMJD3 gene activation via a putative negative glucocorticoid response element and maintains integrity of tight junctions in brain microvascular endothelial cells.Plasma matrix metalloproteinase-9 levels predict intensive care unit mortality early after severe traumatic brain injury.Attenuation of urokinase activity during experimental ischaemia protects the cerebral barrier from damage through regulation of matrix metalloproteinase-2 and NAD(P)H oxidase.
P2860
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P2860
Matrix metalloproteinase-2 or -9 deletions protect against hemorrhagic transformation during early stage of cerebral ischemia and reperfusion.
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
2012年论文
@zh
2012年论文
@zh-cn
name
Matrix metalloproteinase-2 or ...... bral ischemia and reperfusion.
@ast
Matrix metalloproteinase-2 or ...... bral ischemia and reperfusion.
@en
type
label
Matrix metalloproteinase-2 or ...... bral ischemia and reperfusion.
@ast
Matrix metalloproteinase-2 or ...... bral ischemia and reperfusion.
@en
prefLabel
Matrix metalloproteinase-2 or ...... bral ischemia and reperfusion.
@ast
Matrix metalloproteinase-2 or ...... bral ischemia and reperfusion.
@en
P2093
P2860
P1433
P1476
Matrix metalloproteinase-2 or ...... bral ischemia and reperfusion.
@en
P2093
P2860
P304
P356
10.1016/J.NEUROSCIENCE.2012.03.036
P407
P577
2012-04-19T00:00:00Z