The rapid decrease in astrocyte-associated dystroglycan expression by focal cerebral ischemia is protease-dependent.
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Immunohistochemical distribution of basement membrane proteins in the human inner ear from older subjectsAging and the neurovascular unitA high throughput screen identifies chemical modulators of the laminin-induced clustering of dystroglycan and aquaporin-4 in primary astrocytesComponents of the basal lamina and dystrophin-dystroglycan complex in the neurointermediate lobe of rat pituitary gland: different localizations of beta-dystroglycan, dystrobrevins, alpha1-syntrophin, and aquaporin-4.Hemostasis and alterations of the central nervous systemRelationship of neurovascular elements to neuron injury during ischemia.In the hypoxic central nervous system, endothelial cell proliferation is followed by astrocyte activation, proliferation, and increased expression of the alpha 6 beta 4 integrin and dystroglycanInterendothelial claudin-5 expression depends on cerebral endothelial cell-matrix adhesion by β(1)-integrinsMicroglial cell activation is a source of metalloproteinase generation during hemorrhagic transformation.Deletion of astroglial connexins weakens the blood-brain barrier.Dabigatran abrogates brain endothelial cell permeability in response to thrombin.Extracellular matrix and matrix receptors in blood-brain barrier formation and stroke.Transplanted stem cell-secreted vascular endothelial growth factor effects poststroke recovery, inflammation, and vascular repair.Immunologic privilege in the central nervous system and the blood-brain barrier.Disruption of dystroglycan-laminin interactions modulates water uptake by astrocytesLaminins containing the β2 and γ3 chains regulate astrocyte migration and angiogenesis in the retina.Upregulation of fibronectin and the α5β1 and αvβ3 integrins on blood vessels within the cerebral ischemic penumbra.A novel and simple method for culturing pericytes from mouse brain.Virchow's triad: the vascular basis of cerebral injuryVascular amyloid alters astrocytic water and potassium channels in mouse models and humans with Alzheimer's diseaseInflammation and the neurovascular unit in the setting of focal cerebral ischemia.Endothelial β4 integrin is predominantly expressed in arterioles, where it promotes vascular remodeling in the hypoxic brainA novel method to establish microglia-free astrocyte cultures: comparison of matrix metalloproteinase expression profiles in pure cultures of astrocytes and microgliaThe neurovascular unit in the setting of stroke.14,15-epoxyeicosatrienoic acid promotes production of brain derived neurotrophic factor from astrocytes and exerts neuroprotective effects during ischaemic injury.Perlecan domain V modulates astrogliosis in vitro and after focal cerebral ischemia through multiple receptors and increased nerve growth factor release.Loss of astrocyte polarization upon transient focal brain ischemia as a possible mechanism to counteract early edema formation.Endothelial α6β4 integrin protects during experimental autoimmune encephalomyelitis-induced neuroinflammation by maintaining vascular integrity and tight junction protein expression.Postmortem immunohistochemical alterations following cerebral lesions: A possible pathohistological importance of the β-dystroglycan immunoreactivity.β1-integrin-matrix interactions modulate cerebral microvessel endothelial cell tight junction expression and permeability.At low doses ethanol maintains blood-brain barrier (BBB) integrity after hypoxia and reoxygenation: a brain slice study.The temporal expression patterns of fibronectin and its receptors-α5β1 and αvβ3 integrins on blood vessels after cerebral ischemia.
P2860
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P2860
The rapid decrease in astrocyte-associated dystroglycan expression by focal cerebral ischemia is protease-dependent.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
2007年论文
@zh
2007年论文
@zh-cn
name
The rapid decrease in astrocyt ...... schemia is protease-dependent.
@en
type
label
The rapid decrease in astrocyt ...... schemia is protease-dependent.
@en
prefLabel
The rapid decrease in astrocyt ...... schemia is protease-dependent.
@en
P2093
P2860
P356
P1476
The rapid decrease in astrocyt ...... schemia is protease-dependent.
@en
P2093
Gregory J del Zoppo
Maria Spatz
Richard Milner
Stephanie Hung
Xiaoyun Wang
P2860
P304
P356
10.1038/SJ.JCBFM.9600585
P577
2007-11-21T00:00:00Z