Comprehensive behavioral characterization of an APP/PS-1 double knock-in mouse model of Alzheimer's disease - Wikidata - awgldk - TriplyDB

Comprehensive behavioral characterization of an APP/PS-1 double knock-in mouse model of Alzheimer's disease

Comprehensive behavioral characterization of an APP/PS-1 double knock-in mouse model of Alzheimer's disease

http://www.wikidata.org/entity/Q36997908

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Behavioral assays with mouse models of Alzheimer's disease: practical considerations and guidelines.Galantamine slows down plaque formation and behavioral decline in the 5XFAD mouse model of Alzheimer's disease.REAC technology modifies pathological neuroinflammation and motor behaviour in an Alzheimer's disease mouse model Fingolimod Limits Acute Aβ Neurotoxicity and Promotes Synaptic Versus Extrasynaptic NMDA Receptor Functionality in Hippocampal Neurons Elevated emotional contagion in a mouse model of Alzheimer's disease is associated with increased synchronization in the insula and amygdala Hypertension, cerebrovascular impairment, and cognitive decline in aged AβPP/PS1 mice The Effect of Apolipoprotein E ε4 (APOE ε4) on Visuospatial Working Memory in Healthy Elderly and Amnestic Mild Cognitive Impairment Patients: An Event-Related Potentials Study.A novel monoclonal antibody against the N-terminus of Aβ1-42 reduces plaques and improves cognition in a mouse model of Alzheimer's disease.Using mice to model Alzheimer's dementia: an overview of the clinical disease and the preclinical behavioral changes in 10 mouse models Il10 deficiency rebalances innate immunity to mitigate Alzheimer-like pathology.Generation and behavior characterization of CaMKIIβ knockout mice.Closed head injury in an age-related Alzheimer mouse model leads to an altered neuroinflammatory response and persistent cognitive impairment.Long-term treadmill exercise improves spatial memory of male APPswe/PS1dE9 mice by regulation of BDNF expression and microglia activation.Effects of BACE1 haploinsufficiency on APP processing and Aβ concentrations in male and female 5XFAD Alzheimer mice at different disease stages Amelioration of cognitive impairments in APPswe/PS1dE9 mice is associated with metabolites alteration induced by total salvianolic acid.Behavioural Phenotyping of APPswe/PS1δE9 Mice: Age-Rrelated Changes and Effect of Long-Term Paroxetine Treatment.Clinically relevant intronic splicing enhancer mutation in myelin proteolipid protein leads to progressive microglia and astrocyte activation in white and gray matter regions of the brain NAAG Peptidase Inhibitors Act via mGluR3: Animal Models of Memory, Alzheimer's, and Ethanol Intoxication.Attenuation of traumatic brain injury-induced cognitive impairment in mice by targeting increased cytokine levels with a small molecule experimental therapeutic.A Novel Small Molecule Modulator of Amyloid Pathology.Characterization of AD-like phenotype in aged APPSwe/PS1dE9 mice.A comparative evaluation of a novel vaccine in APP/PS1 mouse models of Alzheimer's disease.Microvascular ultrastructural changes precede cognitive impairment in the murine APPswe/PS1dE9 model of Alzheimer's disease.Practical considerations for choosing a mouse model of Alzheimer's disease.Cognitive Impairment After Sleep Deprivation Rescued by Transcranial Magnetic Stimulation Application in Octodon degus.Chronic cannabidiol treatment improves social and object recognition in double transgenic APPswe/PS1∆E9 mice.Colivelin Ameliorates Impairments in Cognitive Behaviors and Synaptic Plasticity in APP/PS1 Transgenic Mice.Mouse models of neurodegenerative disease: preclinical imaging and neurovascular component.Longitudinal investigation of neuroinflammation and metabolite profiles in the APPswe ×PS1Δe9 transgenic mouse model of Alzheimer's disease.Effects and Mechanism of Huannao Yicong Decoction Extract on the Ethology of Transgenic APP/PS1 Mice.Deuterated polyunsaturated fatty acids reduce brain lipid peroxidation and hippocampal amyloid β-peptide levels, without discernable behavioral effects in an APP/PS1 mutant transgenic mouse model of Alzheimer's disease.Tocotrienol-Rich Fraction of Palm Oil Improves Behavioral Impairments and Regulates Metabolic Pathways in AβPP/PS1 Mice.SMPD3 deficiency perturbs neuronal proteostasis and causes progressive cognitive impairment.Association between Alzheimer's disease pathogenesis and early demyelination and oligodendrocyte dysfunction.

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Comprehensive behavioral characterization of an APP/PS-1 double knock-in mouse model of Alzheimer's disease

http://www.wikidata.org/entity/Q36997908

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2013 nî lūn-bûn

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Comprehensive behavioral chara ...... e model of Alzheimer's disease

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Linda J Van Eldik

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Scott J Webster

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P2860

DHA improves cognition and prevents dysfunction of entorhinal cortex neurons in 3xTg-AD mice

Correlative Memory Deficits, Abeta Elevation, and Amyloid Plaques in Transgenic Mice

Alzheimer-type neuropathology in transgenic mice overexpressing V717F β-amyloid precursor protein

Endoproteolysis of presenilin 1 and accumulation of processed derivatives in vivo

ApoE-directed therapeutics rapidly clear β-amyloid and reverse deficits in AD mouse models

Passive immunotherapy against Abeta in aged APP-transgenic mice reverses cognitive deficits and depletes parenchymal amyloid deposits in spite of increased vascular amyloid and microhemorrhage

Phospholipase A2 reduction ameliorates cognitive deficits in a mouse model of Alzheimer's disease

Ferulic acid is a nutraceutical β-secretase modulator that improves behavioral impairment and alzheimer-like pathology in transgenic mice

Triple-transgenic model of Alzheimer's disease with plaques and tangles: intracellular Abeta and synaptic dysfunction

High-level neuronal expression of abeta 1-42 in wild-type human amyloid protein precursor transgenic mice: synaptotoxicity without plaque formation

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