Depletion of alveolar macrophages during influenza infection facilitates bacterial superinfections
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The Association between Invasive Group A Streptococcal Diseases and Viral Respiratory Tract InfectionsThe role of respiratory viruses in the etiology of bacterial pneumonia: An ecological perspectiveThe role of airway macrophages in apoptotic cell clearance following acute and chronic lung inflammationInfluenza and Bacterial Superinfection: Illuminating the Immunologic Mechanisms of DiseaseNon-invasive Imaging of Sendai Virus Infection in Pharmacologically Immunocompromised Mice: NK and T Cells, but not Neutrophils, Promote Viral Clearance after Therapy with Cyclophosphamide and DexamethasoneThe immunology of influenza virus-associated bacterial pneumoniaPostviral Complications: Bacterial PneumoniaLimited Efficacy of Antibacterial Vaccination Against Secondary Serotype 3 Pneumococcal Pneumonia Following Influenza Infection.Influenza A induces dysfunctional immunity and death in MeCP2-overexpressing mice.Protecting against post-influenza bacterial pneumonia by increasing phagocyte recruitment and ROS production.Asthma increases susceptibility to heterologous but not homologous secondary influenzaInfluenza vaccines to control influenza-associated bacterial infection: where do we stand?Influenza-associated bacterial pneumonia; managing and controlling infection on two fronts.Acute Lung Injury Results from Innate Sensing of Viruses by an ER Stress PathwaySingle-Dose CpG Immunization Protects Against a Heterosubtypic Challenge and Generates Antigen-Specific Memory T Cells.Collectins, H-ficolin and LL-37 reduce influence viral replication in human monocytes and modulate virus-induced cytokine production.Pneumolysin expression by streptococcus pneumoniae protects colonized mice from influenza virus-induced diseaseRole of alveolar macrophages in chronic obstructive pulmonary disease.Coinfection with Streptococcus pneumoniae modulates the B cell response to influenza virusAlveolar macrophages contribute to the pathogenesis of human metapneumovirus infection while protecting against respiratory syncytial virus infectionCoinfection with Streptococcus pneumoniae negatively modulates the size and composition of the ongoing influenza-specific CD8⁺ T cell responseA leukocyte score to improve clinical outcome predictions in bacteremic pneumococcal pneumonia in adults.Importance of bacterial replication and alveolar macrophage-independent clearance mechanisms during early lung infection with Streptococcus pneumoniae.Delivery of GM-CSF to Protect against Influenza Pneumonia.Plasma gelsolin improves lung host defense against pneumonia by enhancing macrophage NOS3 function.Type I Interferon Induced Epigenetic Regulation of Macrophages Suppresses Innate and Adaptive Immunity in Acute Respiratory Viral InfectionTRAIL+ monocytes and monocyte-related cells cause lung damage and thereby increase susceptibility to influenza-Streptococcus pneumoniae coinfection.Pulmonary immunostimulation with MALP-2 in influenza virus-infected mice increases survival after pneumococcal superinfectionImpact of Type I and III Interferons on Respiratory Superinfections Due to Multidrug-Resistant Pathogens.Host genetics of severe influenza: from mouse Mx1 to human IRF7Neonatal Pulmonary Macrophage Depletion Coupled to Defective Mucus Clearance Increases Susceptibility to Pneumonia and Alters Pulmonary Immune Responses.Invasive Aspergillosis Associated With Severe Influenza Infections.Influenza A Virus Infection Predisposes Hosts to Secondary Infection with Different Streptococcus pneumoniae Serotypes with Similar Outcome but Serotype-Specific Manifestation.Viral and bacterial co-infection in severe pneumonia triggers innate immune responses and specifically enhances IP-10: a translational study.A Critical, Nonlinear Threshold Dictates Bacterial Invasion and Initial Kinetics During Influenza.Hierarchical effects of pro-inflammatory cytokines on the post-influenza susceptibility to pneumococcal coinfection.Influenza Virus Overcomes Cellular Blocks To Productively Replicate, Impacting Macrophage FunctionLanthionine Synthetase C-Like 2 Modulates Immune Responses to Influenza Virus Infection.Virus-Bacteria Interactions: An Emerging Topic in Human Infection.M1-like monocytes are a major immunological determinant of severity in previously healthy adults with life-threatening influenza.
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Depletion of alveolar macrophages during influenza infection facilitates bacterial superinfections
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
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scientific article published on 26 June 2013
@en
vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Depletion of alveolar macropha ...... ates bacterial superinfections
@en
Depletion of alveolar macropha ...... tes bacterial superinfections.
@nl
type
label
Depletion of alveolar macropha ...... ates bacterial superinfections
@en
Depletion of alveolar macropha ...... tes bacterial superinfections.
@nl
prefLabel
Depletion of alveolar macropha ...... ates bacterial superinfections
@en
Depletion of alveolar macropha ...... tes bacterial superinfections.
@nl
P2860
P356
P1476
Depletion of alveolar macropha ...... ates bacterial superinfections
@en
P2093
Hazem E Ghoneim
Paul G Thomas
P2860
P304
P356
10.4049/JIMMUNOL.1300014
P407
P577
2013-06-26T00:00:00Z