Brain-derived neurotrophic factor effects on oligodendrocyte progenitors of the basal forebrain are mediated through trkB and the MAP kinase pathway.
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Myelin management by the 18.5-kDa and 21.5-kDa classic myelin basic protein isoformsFSD-C10, a Fasudil derivative, promotes neuroregeneration through indirect and direct mechanismsNudging oligodendrocyte intrinsic signaling to remyelinate and repair: Estrogen receptor ligand effects.Estrogen receptor β ligand therapy activates PI3K/Akt/mTOR signaling in oligodendrocytes and promotes remyelination in a mouse model of multiple sclerosis.A bimodal influence of thyroid hormone on cerebellum oligodendrocyte differentiation.Astrocyte-derived BDNF supports myelin protein synthesis after cuprizone-induced demyelination.BDNF+/- mice exhibit deficits in oligodendrocyte lineage cells of the basal forebrain.Experimental Autoimmune Encephalomyelitis (EAE)-Induced Elevated Expression of the E1 Isoform of Methyl CpG Binding Protein 2 (MeCP2E1): Implications in Multiple Sclerosis (MS)-Induced Neurological Disability and Associated Myelin DamageGenetic ablation of steroid receptor coactivator-3 promotes PPAR-beta-mediated alternative activation of microglia in experimental autoimmune encephalomyelitis.Distribution and phenotype of TrkB oligodendrocyte lineage cells in the adult rat spinal cord.A selective role for ARMS/Kidins220 scaffold protein in spatial memory and trophic support of entorhinal and frontal cortical neuronsThe thrombin receptor is a critical extracellular switch controlling myelination.Erk1/2 MAPK and mTOR signaling sequentially regulates progression through distinct stages of oligodendrocyte differentiation.Astrocytes Promote Oligodendrogenesis after White Matter Damage via Brain-Derived Neurotrophic Factor.PSA-NCAM positive neural progenitors stably expressing BDNF promote functional recovery in a mouse model of spinal cord injury.ERK1/ERK2 MAPK signaling is required to increase myelin thickness independent of oligodendrocyte differentiation and initiation of myelination.Brain-derived neurotrophic factor deficiency restricts proliferation of oligodendrocyte progenitors following cuprizone-induced demyelinationNucleus-localized 21.5-kDa myelin basic protein promotes oligodendrocyte proliferation and enhances neurite outgrowth in coculture, unlike the plasma membrane-associated 18.5-kDa isoformThe antiaging protein Klotho enhances oligodendrocyte maturation and myelination of the CNS.Sustained activation of ERK1/2 MAPK in oligodendrocytes and schwann cells enhances myelin growth and stimulates oligodendrocyte progenitor expansion.Regulation of myelin genes implicated in psychiatric disorders by functional activity in axons.Promoting oligodendrogenesis and myelin repair using the multiple sclerosis medication glatiramer acetate.Crosstalk between cerebral endothelium and oligodendrocyte.Tissue-type plasminogen activator exerts EGF-like chemokinetic effects on oligodendrocytes in white matter (re)myelination.Neuroglialpharmacology: myelination as a shared mechanism of action of psychotropic treatments.Signaling mechanisms regulating myelination in the central nervous system.Stem cells for brain repair in neonatal hypoxia-ischemia.Transcriptional Regulation of Brain-Derived Neurotrophic Factor (BDNF) by Methyl CpG Binding Protein 2 (MeCP2): a Novel Mechanism for Re-Myelination and/or Myelin Repair Involved in the Treatment of Multiple Sclerosis (MS).Levels of BDNF impact oligodendrocyte lineage cells following a cuprizone lesion.Monosialoganglioside 1 may alleviate neurotoxicity induced by propofol combined with remifentanil in neural stem cells.TAPP1 inhibits the differentiation of oligodendrocyte precursor cells via suppressing the Mek/Erk pathwayTransplantation of Induced Pluripotent Stem Cell-Derived Neural Stem Cells Mediate Functional Recovery Following Thoracic Spinal Cord Injury Through Remyelination of Axons.Extracellular signal-regulated kinase 1/2 signaling promotes oligodendrocyte myelination in vitro.Neural stem cells modified to express BDNF antagonize trimethyltin-induced neurotoxicity through PI3K/Akt and MAP kinase pathways.Brain-derived neurotrophic factor stimulates proliferation and differentiation of neural stem cells, possibly by triggering the Wnt/β-catenin signaling pathway.Bad wrap: Myelin and myelin plasticity in health and disease.Neuregulin-1 promotes remyelination and fosters a pro-regenerative inflammatory response in focal demyelinating lesions of the spinal cord.Microenvironmental regulation of oligodendrocyte replacement and remyelination in spinal cord injury.
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P2860
Brain-derived neurotrophic factor effects on oligodendrocyte progenitors of the basal forebrain are mediated through trkB and the MAP kinase pathway.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on January 2009
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Brain-derived neurotrophic fac ...... kB and the MAP kinase pathway.
@en
Brain-derived neurotrophic fac ...... kB and the MAP kinase pathway.
@nl
type
label
Brain-derived neurotrophic fac ...... kB and the MAP kinase pathway.
@en
Brain-derived neurotrophic fac ...... kB and the MAP kinase pathway.
@nl
prefLabel
Brain-derived neurotrophic fac ...... kB and the MAP kinase pathway.
@en
Brain-derived neurotrophic fac ...... kB and the MAP kinase pathway.
@nl
P2093
P2860
P356
P1476
Brain-derived neurotrophic fac ...... kB and the MAP kinase pathway.
@en
P2093
Ashlee Van't Veer
Cheryl F Dreyfus
Deborah R Boetig
Melissa R Wood
Tanya Z Fischer
Yangzhou Du
P2860
P356
10.1002/JNR.21841
P577
2009-01-01T00:00:00Z