Toll-like receptor-3 activation increases the vulnerability of the neonatal brain to hypoxia-ischemia.
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Polyinosinic-polycytidylic acid has therapeutic effects against cerebral ischemia/reperfusion injury through the downregulation of TLR4 signaling via TLR3Neonatal Hypoxia Ischaemia: Mechanisms, Models, and Therapeutic Challenges.Prevalence of bloodstream pathogens is higher in neonatal encephalopathy cases vs. controls using a novel panel of real-time PCR assaysExendin-4 reduces ischemic brain injury in normal and aged type 2 diabetic mice and promotes microglial M2 polarization.Cellular mechanisms of toll-like receptor-3 activation in the thalamus are associated with white matter injury in the developing brainModeling Ischemia in the Immature Brain: How Translational Are Animal Models?The role of inflammation in perinatal brain injury.Is the ferret a suitable species for studying perinatal brain injury?TIR-Domain-Containing Adapter-Inducing Interferon-β (TRIF) Is Essential for MPTP-Induced Dopaminergic Neuroprotection via Microglial Cell M1/M2 Modulation.Toll-like receptors 2, -3 and -4 prime microglia but not astrocytes across central nervous system regions for ATP-dependent interleukin-1β release.Microglia toxicity in preterm brain injury.Maternal immune activation and abnormal brain development across CNS disorders.Inflammation-induced sensitization of the brain in term infants.Interplay Between Exosomes, microRNAs and Toll-Like Receptors in Brain Disorders.Perinatal hypoxia-ischemia reduces α 7 nicotinic receptor expression and selective α 7 nicotinic receptor stimulation suppresses inflammation and promotes microglial Mox phenotypePerinatal risk factors for neonatal encephalopathy: an unmatched case-control study.Systemic activation of Toll-like receptor 2 suppresses mitochondrial respiration and exacerbates hypoxic-ischemic injury in the developing brain.Role of Perinatal Inflammation in Neonatal Arterial Ischemic Stroke.Oxidative stress diseases unique to the perinatal period: A window into the developing innate immune response.Maternal immune activation in neurodevelopmental disorders.Tollip is a critical mediator of cerebral ischaemia-reperfusion injury.Neuroprotective exendin-4 enhances hypothermia therapy in a model of hypoxic-ischaemic encephalopathyFas-ligand and interleukin-6 in the cerebrospinal fluid are early predictors of hypoxic-ischemic encephalopathy and long-term outcomes after birth asphyxia in term infants
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P2860
Toll-like receptor-3 activation increases the vulnerability of the neonatal brain to hypoxia-ischemia.
description
2013 nî lūn-bûn
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2013年の論文
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name
Toll-like receptor-3 activatio ...... tal brain to hypoxia-ischemia.
@en
Toll-like receptor-3 activatio ...... tal brain to hypoxia-ischemia.
@nl
type
label
Toll-like receptor-3 activatio ...... tal brain to hypoxia-ischemia.
@en
Toll-like receptor-3 activatio ...... tal brain to hypoxia-ischemia.
@nl
prefLabel
Toll-like receptor-3 activatio ...... tal brain to hypoxia-ischemia.
@en
Toll-like receptor-3 activatio ...... tal brain to hypoxia-ischemia.
@nl
P2093
P2860
P50
P1476
Toll-like receptor-3 activatio ...... tal brain to hypoxia-ischemia.
@en
P2093
Amin Mottahedin
Frances Northington
Linnea Stridh
Raul Chavez Valdez
P2860
P304
12041-12051
P356
10.1523/JNEUROSCI.0673-13.2013
P407
P577
2013-07-01T00:00:00Z