Transient potential receptor melastatin-2 (Trpm2) does not influence murine MLL-AF9-driven AML leukemogenesis or in vitro response to chemotherapy.

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Transient potential receptor melastatin-2 (Trpm2) does not influence murine MLL-AF9-driven AML leukemogenesis or in vitro response to chemotherapy.

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article científic

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artigo científico

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bilimsel makale

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scientific article published on 23 March 2016

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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Transient potential receptor m ...... itro response to chemotherapy.

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Transient potential receptor melastatin-2

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Transient potential receptor m ...... itro response to chemotherapy.

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Transient potential receptor melastatin-2

@nl

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Transient potential receptor m ...... itro response to chemotherapy.

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Transient potential receptor melastatin-2

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Transient potential receptor m ...... vitro response to chemotherapy

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Anne-Laure Perraud

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Jessica N Haladyna

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Simone S Riedel

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Taylor Pastuer

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P2860

Molecular cloning of a novel putative Ca2+ channel protein (TRPC7) highly expressed in brain

ADP-ribose gating of the calcium-permeable LTRPC2 channel revealed by Nudix motif homology

TRPM2-mediated Ca2+influx induces chemokine production in monocytes that aggravates inflammatory neutrophil infiltration

Four Ca2+ ions activate TRPM2 channels by binding in deep crevices near the pore but intracellularly of the gate

Potent inhibition of DOT1L as treatment of MLL-fusion leukemia

Gene Expression Commons: an open platform for absolute gene expression profiling

Myeloid malignancies with chromosome 5q deletions acquire a dependency on an intrachromosomal NF-κB gene network

A splice variant of the human ion channel TRPM2 modulates neuroblastoma tumor growth through hypoxia-inducible factor (HIF)-1/2α.

Transient Receptor Potential Melastatin 2 (TRPM2) ion channel is required for innate immunity against Listeria monocytogenes.

Inhibition of the transient receptor potential melastatin-2 channel causes increased DNA damage and decreased proliferation in breast adenocarcinoma cells

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596-602.e3

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scholarly article

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10.1016/J.EXPHEM.2016.03.006

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Kathrin M. Bernt

Kathrin M Bernt

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2016-03-24T00:00:00Z

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27033163

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4914470

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