The skeletal L-type Ca(2+) current is a major contributor to excitation-coupled Ca(2+) entry.
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Ca2+-dependent modulation of voltage-gated Ca2+ channelsMalignant hyperthermia: a reviewCa(V)1.1: The atypical prototypical voltage-gated Ca²⁺ channelStore-operated Ca2+ entry in muscle physiology and diseasesAlternating bipolar field stimulation identifies muscle fibers with defective excitability but maintained local Ca(2+) signals and contraction.The calmodulin-binding, short linear motif, NSCaTE is conserved in L-type channel ancestors of vertebrate Cav1.2 and Cav1.3 channelsTriclosan impairs excitation-contraction coupling and Ca2+ dynamics in striated muscleMinding the calcium store: Ryanodine receptor activation as a convergent mechanism of PCB toxicityCalcium homeostasis in myogenic differentiation factor 1 (MyoD)-transformed, virally-transduced, skin-derived equine myotubesDHPR alpha1S subunit controls skeletal muscle mass and morphogenesis.Rapid Ca2+ flux through the transverse tubular membrane, activated by individual action potentials in mammalian skeletal muscle.Differential effects of RGK proteins on L-type channel function in adult mouse skeletal muscleA malignant hyperthermia-inducing mutation in RYR1 (R163C): alterations in Ca2+ entry, release, and retrograde signaling to the DHPR.A malignant hyperthermia-inducing mutation in RYR1 (R163C): consequent alterations in the functional properties of DHPR channelsStore-operated Ca2+ entry in malignant hyperthermia-susceptible human skeletal muscle.The cardiac alpha(1C) subunit can support excitation-triggered Ca2+ entry in dysgenic and dyspedic myotubesMice null for calsequestrin 1 exhibit deficits in functional performance and sarcoplasmic reticulum calcium handling.Simulations of calcium channel block by trivalent cations: Gd(3+) competes with permeant ions for the selectivity filter.RGK protein-mediated impairment of slow depolarization- dependent Ca2+ entry into developing myotubes.The Sick and the Weak: Neuropathies/Myopathies in the Critically Ill.Accelerated activation of SOCE current in myotubes from two mouse models of anesthetic- and heat-induced sudden death.Alterations of excitation-contraction coupling and excitation coupled Ca(2+) entry in human myotubes carrying CAV3 mutations linked to rippling muscleCa(2+) permeation and/or binding to CaV1.1 fine-tunes skeletal muscle Ca(2+) signaling to sustain muscle function.Dissecting out the complex Ca2+-mediated phenylephrine-induced contractions of mouse aortic segments.Role of Na⁺/Ca²⁺ exchanger in Ca²⁺ homeostasis in rat suprachiasmatic nucleus neurons.Properties of Na+ currents conducted by a skeletal muscle L-type Ca2+ channel pore mutant (SkEIIIK).Levels of Ca(V)1.2 L-Type Ca(2+) Channels Peak in the First Two Weeks in Rat Hippocampus Whereas Ca(V)1.3 Channels Steadily Increase through Development.Identification of L- and T-type Ca2+ channels in rat cerebral arteries: role in myogenic tone developmentDistinct Components of Retrograde Ca(V)1.1-RyR1 Coupling Revealed by a Lethal Mutation in RyR1.Dantrolene-induced inhibition of skeletal L-type Ca2+ current requires RyR1 expression.Functional characterization of orbicularis oculi and extraocular musclesProgressive impairment of CaV1.1 function in the skeletal muscle of mice expressing a mutant type 1 Cu/Zn superoxide dismutase (G93A) linked to amyotrophic lateral sclerosis.Checking your SOCCs and feet: the molecular mechanisms of Ca2+ entry in skeletal muscle.Calcium entry in skeletal muscle.Role of ryanodine receptor subtypes in initiation and formation of calcium sparks in arterial smooth muscle: comparison with striated muscle.The role of store-operated calcium influx in skeletal muscle signaling.Reciprocal dihydropyridine and ryanodine receptor interactions in skeletal muscle activation.Store-operated Ca(2+) entry (SOCE) contributes to normal skeletal muscle contractility in young but not in aged skeletal muscle.Voltage-Induced Ca²⁺ Release in Postganglionic Sympathetic Neurons in Adult Mice.Muscle weakness in myotonic dystrophy associated with misregulated splicing and altered gating of Ca(V)1.1 calcium channel.
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The skeletal L-type Ca(2+) current is a major contributor to excitation-coupled Ca(2+) entry.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on January 2009
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
The skeletal L-type Ca
@nl
The skeletal L-type Ca(2+) cur ...... citation-coupled Ca(2+) entry.
@en
type
label
The skeletal L-type Ca
@nl
The skeletal L-type Ca(2+) cur ...... citation-coupled Ca(2+) entry.
@en
prefLabel
The skeletal L-type Ca
@nl
The skeletal L-type Ca(2+) cur ...... citation-coupled Ca(2+) entry.
@en
P2093
P2860
P356
P1476
The skeletal L-type Ca(2+) cur ...... citation-coupled Ca(2+) entry.
@en
P2093
Isaac N Pessah
Kurt G Beam
Roger A Bannister
P2860
P356
10.1085/JGP.200810105
P577
2009-01-01T00:00:00Z