Sulfonylurea receptor-dependent and -independent pathways mediate vasodilation induced by ATP-sensitive K+ channel openers
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Diazoxide promotes oligodendrocyte precursor cell proliferation and myelinationCaveolin-1 assembles type 1 inositol 1,4,5-trisphosphate receptors and canonical transient receptor potential 3 channels into a functional signaling complex in arterial smooth muscle cellsDirect activation of β-cell KATP channels with a novel xanthine derivative.The mitochondrial complex II and ATP-sensitive potassium channel interaction: quantitation of the channel in heart mitochondria.Diversity of mitochondria-dependent dilator mechanisms in vascular smooth muscle of cerebral arteries from normal and insulin-resistant rats.Persistent hyperinsulinemic hypoglycemia of infancy: constitutive activation of the mTOR pathway with associated exocrine-islet transdifferentiation and therapeutic implicationsEugenol dilates rat cerebral arteries by inhibiting smooth muscle cell voltage-dependent calcium channels.Hydrogen sulfide and cerebral microvascular tone in newborn pigs.Reassessment of risk genotypes (GRN, TMEM106B, and ABCC9 variants) associated with hippocampal sclerosis of aging pathologyCa(V)1.2 channel N-terminal splice variants modulate functional surface expression in resistance size artery smooth muscle cellsHydrogen sulfide dilates cerebral arterioles by activating smooth muscle cell plasma membrane KATP channelsVasodilation induced by oxygen/glucose deprivation is attenuated in cerebral arteries of SUR2 null miceAlternative splicing of Cav1.2 channel exons in smooth muscle cells of resistance-size arteries generates currents with unique electrophysiological properties.The complex II inhibitor atpenin A5 protects against cardiac ischemia-reperfusion injury via activation of mitochondrial KATP channelsPharmacology of mitochondrial potassium channels: dark side of the field.Isoform-selective physical coupling of TRPC3 channels to IP3 receptors in smooth muscle cells regulates arterial contractility.Transcriptional upregulation of α2δ-1 elevates arterial smooth muscle cell voltage-dependent Ca2+ channel surface expression and cerebrovascular constriction in genetic hypertension.Smooth muscle cell alpha2delta-1 subunits are essential for vasoregulation by CaV1.2 channels.Hydrogen sulfide activates Ca²⁺ sparks to induce cerebral arteriole dilatation.Smooth muscle cell transient receptor potential polycystin-2 (TRPP2) channels contribute to the myogenic response in cerebral arteries.The voltage-dependent L-type Ca2+ (CaV1.2) channel C-terminus fragment is a bi-modal vasodilator.The expression of KATP channel subunits in alpha-synuclein-transfected MES23.5 cells.
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P2860
Sulfonylurea receptor-dependent and -independent pathways mediate vasodilation induced by ATP-sensitive K+ channel openers
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 29 May 2008
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Sulfonylurea receptor-dependen ...... P-sensitive K+ channel openers
@en
Sulfonylurea receptor-dependen ...... -sensitive K+ channel openers.
@nl
type
label
Sulfonylurea receptor-dependen ...... P-sensitive K+ channel openers
@en
Sulfonylurea receptor-dependen ...... -sensitive K+ channel openers.
@nl
prefLabel
Sulfonylurea receptor-dependen ...... P-sensitive K+ channel openers
@en
Sulfonylurea receptor-dependen ...... -sensitive K+ channel openers.
@nl
P2860
P356
P1476
Sulfonylurea receptor-dependen ...... P-sensitive K+ channel openers
@en
P2093
Elizabeth M McNally
Jonathan H Jaggar
P2860
P304
P356
10.1124/MOL.108.048165
P577
2008-05-29T00:00:00Z