Smad gene expression in pulmonary fibroblasts: indications for defective ECM repair in COPD
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A gene expression signature of emphysema-related lung destruction and its reversal by the tripeptide GHKDifferential expression of tissue repair genes in the pathogenesis of chronic obstructive pulmonary disease.Altered fibroblast proteoglycan production in COPD.Activation of WNT/β-catenin signaling in pulmonary fibroblasts by TGF-β₁ is increased in chronic obstructive pulmonary disease.Variability in the precursor proteins of collagen I and III in different stages of COPDDefective alterations in the collagen network to prostacyclin in COPD lung fibroblasts.Inhaled steroids modulate extracellular matrix composition in bronchial biopsies of COPD patients: a randomized, controlled trial.Mechanisms of lung development: contribution to adult lung disease and relevance to chronic obstructive pulmonary diseaseSensitivity of heterozygous α1,6-fucosyltransferase knock-out mice to cigarette smoke-induced emphysema: implication of aberrant transforming growth factor-β signaling and matrix metalloproteinase gene expressionGlycogen synthase kinase-3 (GSK-3) regulates TGF-β₁-induced differentiation of pulmonary fibroblasts.Fibulin-1 regulates the pathogenesis of tissue remodeling in respiratory diseasesUpdate in chronic obstructive pulmonary disease 2009.Fibrotic response of tissue remodeling in COPD.A brief review of chronic obstructive pulmonary diseaseRole of transcription factors in the pathogenesis of asthma and COPD.Differential gene expression profiles of peripheral blood mononuclear cells in childhood asthma.Identification of transforming growth factor-beta-regulated microRNAs and the microRNA-targetomes in primary lung fibroblastsCombined Pulmonary Fibrosis and Emphysema in Scleroderma-Related Lung Disease Has a Major Confounding Effect on Lung Physiology and Screening for Pulmonary Hypertension.Role of Smad3 and p38 Signalling in Cigarette Smoke-induced CFTR and BK dysfunction in Primary Human Bronchial Airway Epithelial Cells.Differential effects of fluticasone on extracellular matrix production by airway and parenchymal fibroblasts in severe COPD.Transforming growth factor (TGF) β1 and Smad signalling pathways: A likely key to EMT-associated COPD pathogenesis.Smoking-related emphysema is associated with idiopathic pulmonary fibrosis and rheumatoid lung.Melatonin suppresses fibrotic responses induced by cigarette smoke via downregulation of TGF-β1.TGF-β Signaling Pathways in Different Compartments of the Lower Airways of Patients With Stable COPD.Prevalence and Effects of Emphysema in Never-Smokers with Rheumatoid Arthritis Interstitial Lung Disease.Mesenchymal stem cell-conditioned media recovers lung fibroblasts from cigarette smoke-induced damage.Palifermin induces alveolar maintenance programs in emphysematous mice.
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Smad gene expression in pulmonary fibroblasts: indications for defective ECM repair in COPD
description
article científic
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article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 16 December 2008
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Smad gene expression in pulmon ...... r defective ECM repair in COPD
@en
Smad gene expression in pulmon ...... defective ECM repair in COPD.
@nl
type
label
Smad gene expression in pulmon ...... r defective ECM repair in COPD
@en
Smad gene expression in pulmon ...... defective ECM repair in COPD.
@nl
prefLabel
Smad gene expression in pulmon ...... r defective ECM repair in COPD
@en
Smad gene expression in pulmon ...... defective ECM repair in COPD.
@nl
P2093
P2860
P356
P1433
P1476
Smad gene expression in pulmon ...... r defective ECM repair in COPD
@en
P2093
Andre Zandvoort
Jacobien A Noordhoek
Johannes T W M Vos
Marnix R Jonker
P2860
P2888
P356
10.1186/1465-9921-9-83
P577
2008-12-16T00:00:00Z
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P6179
1015783514