Notch4-dependent antagonism of canonical TGF-β1 signaling defines unique temporal fluctuations of SMAD3 activity in sheared proximal tubular epithelial cells.
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The physical basis of renal fibrosis: effects of altered hydrodynamic forces on kidney homeostasis.Notch in fibrosis and as a target of anti-fibrotic therapy.Fluid shear stress-induced TGF-β/ALK5 signaling in renal epithelial cells is modulated by MEK1/2.Comprehensive transcriptome analysis of fluid shear stress altered gene expression in renal epithelial cells.NOTCH4 Intracellular Domain Regulates Transcription
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Notch4-dependent antagonism of canonical TGF-β1 signaling defines unique temporal fluctuations of SMAD3 activity in sheared proximal tubular epithelial cells.
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 10 April 2013
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Notch4-dependent antagonism of ...... imal tubular epithelial cells.
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Notch4-dependent antagonism of ...... imal tubular epithelial cells.
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type
label
Notch4-dependent antagonism of ...... imal tubular epithelial cells.
@en
Notch4-dependent antagonism of ...... imal tubular epithelial cells.
@nl
prefLabel
Notch4-dependent antagonism of ...... imal tubular epithelial cells.
@en
Notch4-dependent antagonism of ...... imal tubular epithelial cells.
@nl
P2860
P1476
Notch4-dependent antagonism of ...... imal tubular epithelial cells.
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P2093
Bryan M Grabias
Konstantinos Konstantopoulos
P2860
P304
P356
10.1152/AJPRENAL.00594.2012
P577
2013-04-10T00:00:00Z