Sharpening the ends for repair: mechanisms and regulation of DNA resection. - Wikidata - awgldk - TriplyDB

Sharpening the ends for repair: mechanisms and regulation of DNA resection.

Sharpening the ends for repair: mechanisms and regulation of DNA resection.

http://www.wikidata.org/entity/Q37058602

about

Dna2 initiates resection at clean DNA double-strand breaks.A special issue on the DNA damage response and genomic instability.

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P2860

Sharpening the ends for repair: mechanisms and regulation of DNA resection.

http://www.wikidata.org/entity/Q37058602

description

article científic

@ca

article scientifique

@fr

articolo scientifico

@it

artigo científico

@pt

bilimsel makale

@tr

scientific article published on 12 May 2016

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Sharpening the ends for repair: mechanisms and regulation of DNA resection.

@en

Sharpening the ends for repair: mechanisms and regulation of DNA resection.

@nl

type

label

Sharpening the ends for repair: mechanisms and regulation of DNA resection.

@en

Sharpening the ends for repair: mechanisms and regulation of DNA resection.

@nl

prefLabel

Sharpening the ends for repair: mechanisms and regulation of DNA resection.

@en

Sharpening the ends for repair: mechanisms and regulation of DNA resection.

@nl

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Acta Biochimica et Biophysica Sinica

P1476

Sharpening the ends for repair: mechanisms and regulation of DNA resection.

@en

P2093

Sharad C Paudyal

http://www.w3.org/2001/XMLSchema#string

Zhongsheng You

http://www.w3.org/2001/XMLSchema#string

P2860

The RAD2 domain of human exonuclease 1 exhibits 5' to 3' exonuclease and flap structure-specific endonuclease activities

The mechanism of double-strand DNA break repair by the nonhomologous DNA end-joining pathway

BLM-DNA2-RPA-MRN and EXO1-BLM-RPA-MRN constitute two DNA end resection machineries for human DNA break repair

The yeast Fun30 and human SMARCAD1 chromatin remodellers promote DNA end resection

Human SIRT6 promotes DNA end resection through CtIP deacetylation

DNA end resection by CtIP and exonuclease 1 prevents genomic instability

ATM and ATR substrate analysis reveals extensive protein networks responsive to DNA damage

DNA damage-induced cell cycle checkpoint control requires CtIP, a phosphorylation-dependent binding partner of BRCA1 C-terminal domains

The 3' to 5' exonuclease activity of Mre 11 facilitates repair of DNA double-strand breaks

N terminus of CtIP is critical for homologous recombination-mediated double-strand break repair

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647-657

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scholarly article

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10.1093/ABBS/GMW043

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7

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48

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2016-05-12T00:00:00Z

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27174871

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4930117

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