IL-12 and type-I IFN synergize for IFN-gamma production by CD4 T cells, whereas neither are required for IFN-gamma production by CD8 T cells after Listeria monocytogenes infection
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Listeria monocytogenes: a model pathogen to study antigen-specific memory CD8 T cell responsesMurine model of Clostridium difficile infection with aged gnotobiotic C57BL/6 mice and a BI/NAP1 strain.Differential regulation of effector- and central-memory responses to Toxoplasma gondii Infection by IL-12 revealed by tracking of Tgd057-specific CD8+ T cells.Type I interferons are not critical for skin allograft rejection or the generation of donor-specific CD8+ memory T cells.Naturally occurring altered peptide ligands control Salmonella-specific CD4+ T cell proliferation, IFN-gamma production, and protective potency.Lipopolysaccharide potentiates effector T cell accumulation into nonlymphoid tissues through TRIFCD4+ T cells are not required for the induction of dengue virus-specific CD8+ T cell or antibody responses but contribute to protection after vaccination.Perinatal Listeria monocytogenes susceptibility despite preconceptual priming and maintenance of pathogen-specific CD8(+) T cells during pregnancy.Foxp3+ regulatory T cells impede the priming of protective CD8+ T cells.Temporal regulation of rapamycin on memory CTL programming by IL-12CXCR3 blockade protects against Listeria monocytogenes infection-induced fetal wastage.Pathogen-induced inflammatory environment controls effector and memory CD8+ T cell differentiation.IFN-alpha beta and self-MHC divert CD8 T cells into a distinct differentiation pathway characterized by rapid acquisition of effector functionsHIV-1 replication activates CD4+ T cells with specificities for persistent herpes virusesInnate IFN-γ is essential for programmed death ligand-1-mediated T cell stimulation following Listeria monocytogenes infection.Heterogeneity and cell-fate decisions in effector and memory CD8+ T cell differentiation during viral infectionAge-related changes in CD8 T cell homeostasis and immunity to infectionTLR9 and STING agonists synergistically induce innate and adaptive type-II IFN.Specific depletion reveals a novel role for neutrophil-mediated protection in the liver during Listeria monocytogenes infectionFunctional genomics identifies type I interferon pathway as central for host defense against Candida albicansType I Interferons Regulate the Magnitude and Functionality of Mouse Polyomavirus-Specific CD8 T Cells in a Virus Strain-Dependent MannerNicotine inhibits memory CTL programmingPrevention of allograft tolerance by bacterial infection with Listeria monocytogenes.Antigen-free adjuvant assists late effector CD4 T cells to transit to memory in lymphopenic hosts.PDL-1 blockade impedes T cell expansion and protective immunity primed by attenuated Listeria monocytogenes.Deviation from a strong Th1-dominated to a modest Th17-dominated CD4 T cell response in the absence of IL-12p40 and type I IFNs sustains protective CD8 T cells.Reciprocal responsiveness to interleukin-12 and interferon-alpha specifies human CD8+ effector versus central memory T-cell fatesRegulatory T cells and the immune pathogenesis of prenatal infection.IL-23 promotes the production of IL-17 by antigen-specific CD8 T cells in the absence of IL-12 and type-I interferons.Epigenetic control of T-helper-cell differentiation.Type I IFN induces binding of STAT1 to Bcl6: divergent roles of STAT family transcription factors in the T follicular helper cell genetic programTranscription factor STAT3 and type I interferons are corepressive insulators for differentiation of follicular helper and T helper 1 cellsRegulation of effector and memory T-cell functions by type I interferon.Cell biology and immunology of Listeria monocytogenes infections: novel insights.Rapid culling of the CD4+ T cell repertoire in the transition from effector to memory.Differential Inhibitory Receptor Expression on T Cells Delineates Functional Capacities in Chronic Viral Infection.Generation of a Listeria vaccine strain by enhanced caspase-1 activation.Effector functions of memory CTLs can be affected by signals received during reactivation.MyD88 and interferon-alpha/beta are differentially required for dendritic cell maturation but dispensable for development of protective memory against Listeria.The immune characterization of interferon-β responses in tuberculosis patients.
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IL-12 and type-I IFN synergize for IFN-gamma production by CD4 T cells, whereas neither are required for IFN-gamma production by CD8 T cells after Listeria monocytogenes infection
description
article científic
@ca
article scientifique
@fr
articolo scientifico
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artigo científico
@pt
bilimsel makale
@tr
scientific article published on April 2007
@en
vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
IL-12 and type-I IFN synergize ...... steria monocytogenes infection
@en
IL-12 and type-I IFN synergize ...... teria monocytogenes infection.
@nl
type
label
IL-12 and type-I IFN synergize ...... steria monocytogenes infection
@en
IL-12 and type-I IFN synergize ...... teria monocytogenes infection.
@nl
prefLabel
IL-12 and type-I IFN synergize ...... steria monocytogenes infection
@en
IL-12 and type-I IFN synergize ...... teria monocytogenes infection.
@nl
P2093
P2860
P1476
IL-12 and type-I IFN synergize ...... steria monocytogenes infection
@en
P2093
Ganesh A Kolumam
Kaja Murali-Krishna
Nural N Orgun
Sing Sing Way
P2860
P304
P356
10.4049/JIMMUNOL.178.7.4498
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P577
2007-04-01T00:00:00Z