Reactive oxygen species-induced activation of p90 ribosomal S6 kinase prolongs cardiac repolarization through inhibiting outward K+ channel activity.
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Reactive Oxygen Species, Endoplasmic Reticulum Stress and Mitochondrial Dysfunction: The Link with Cardiac ArrhythmogenesisMolecular and Electrophysiological Mechanisms Underlying Cardiac Arrhythmogenesis in Diabetes MellitusRSK3: A regulator of pathological cardiac remodelingNovel role for p90 ribosomal S6 kinase in the regulation of cardiac myofilament phosphorylationMolecular determinants of cardiac transient outward potassium current (I(to)) expression and regulationBone marrow mesenchymal stem cells protected post-infarcted myocardium against arrhythmias via reversing potassium channels remodelling.Deletion of FoxO1 leads to shortening of QRS by increasing Na(+) channel activity through enhanced expression of both cardiac NaV1.5 and β3 subunitReactive oxygen species suppress cardiac NaV1.5 expression through Foxo1.Pyruvate-fortified resuscitation stabilizes cardiac electrical activity and energy metabolism during hypovolemia.Xanthine oxidase inhibition alleviates the cardiac complications of insulin resistance: effect on low grade inflammation and the angiotensin system.Na+/H+ exchanger isoform 1 induced cardiomyocyte hypertrophy involves activation of p90 ribosomal s6 kinase.p90RSK targets the ERK5-CHIP ubiquitin E3 ligase activity in diabetic hearts and promotes cardiac apoptosis and dysfunction.Mechanistic Investigation of the Arrhythmogenic Role of Oxidized CaMKII in the HeartOxidized LDL induces FAK-dependent RSK signaling to drive NF-κB activation and VCAM-1 expressionFunctional Expression Profile of Voltage-Gated K(+) Channel Subunits in Rat Small Mesenteric Arteries.Melanocyte-like cells in the heart and pulmonary veins contribute to atrial arrhythmia triggers.Role of autocrine/paracrine mechanisms in response to myocardial strain.Modulation of voltage-gated ion channels by sialylation.Cardiac potassium channel subtypes: new roles in repolarization and arrhythmia.Subclinical vascular endothelial dysfunctions and myocardial changes with type 1 diabetes mellitus in children and adolescents.Reactive oxygen species-activated Ca/calmodulin kinase IIδ is required for late I(Na) augmentation leading to cellular Na and Ca overload.Novel insights into the cellular basis of atrial fibrillation.Protein kinase A is central for forward transport of two-pore domain potassium channels K2P3.1 and K2P9.1.Zingerone alleviates the delayed ventricular repolarization and AV conduction in diabetes: Effect on cardiac fibrosis and inflammation.
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P2860
Reactive oxygen species-induced activation of p90 ribosomal S6 kinase prolongs cardiac repolarization through inhibiting outward K+ channel activity.
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 03 July 2008
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Reactive oxygen species-induce ...... g outward K+ channel activity.
@en
Reactive oxygen species-induce ...... g outward K+ channel activity.
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type
label
Reactive oxygen species-induce ...... g outward K+ channel activity.
@en
Reactive oxygen species-induce ...... g outward K+ channel activity.
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prefLabel
Reactive oxygen species-induce ...... g outward K+ channel activity.
@en
Reactive oxygen species-induce ...... g outward K+ channel activity.
@nl
P2093
P2860
P1433
P1476
Reactive oxygen species-induce ...... g outward K+ channel activity.
@en
P2093
Carolyn McClain
Haodong Xu
Jack Taunton
Jun-ichi Abe
Sheng Ping Xu
Tetsuro Shishido
Thomas M Spangenberg
P2860
P304
P356
10.1161/CIRCRESAHA.107.166678
P577
2008-07-03T00:00:00Z