The NMDA antagonist MK-801 disrupts reconsolidation of a cocaine-associated memory for conditioned place preference but not for self-administration in rats.
about
Noradrenergic regulation of fear and drug-associated memory reconsolidationAnisomycin in the medial prefrontal cortex reduces reconsolidation of cocaine-associated memories in the rat self-administration modelDisrupting astrocyte-neuron lactate transfer persistently reduces conditioned responses to cocaine.Photoperiodic suppression of drug reinstatementContribution of an SFK-Mediated Signaling Pathway in the Dorsal Hippocampus to Cocaine-Memory Reconsolidation in Rats.Glucocorticoid receptor antagonism disrupts the reconsolidation of social reward-related memories in ratsThe Hepatocyte Growth Factor/c-Met Antagonist, Divalinal-Angiotensin IV, Blocks the Acquisition of Methamphetamine Dependent Conditioned Place Preference in Rats.Reconsolidation of a cocaine-associated stimulus requires amygdalar protein kinase A.Activation of exchange protein activated by cAMP in the rat basolateral amygdala impairs reconsolidation of a memory associated with self-administered cocaine.Learning to forget: manipulating extinction and reconsolidation processes to treat addictionEarly methylphenidate exposure enhances cocaine self-administration but not cocaine-induced conditioned place preference in young adult ratsBaclofen facilitates the extinction of methamphetamine-induced conditioned place preference in rats.Aberrant learning and memory in addiction.Cocaine and HIV-1 interplay: molecular mechanisms of action and addiction.Brain Circuits of Methamphetamine Place Reinforcement Learning: The Role of the Hippocampus-VTA Loop.Gabapentin potentiates sensitivity to the interoceptive effects of alcohol and increases alcohol self-administration in ratsExtracellular signal-regulated kinase in the basolateral amygdala, but not the nucleus accumbens core, is critical for context-response-cocaine memory reconsolidation in rats.Addiction: a drug-induced disorder of memory reconsolidation.The Nucleus Accumbens: Mechanisms of Addiction across Drug Classes Reflect the Importance of Glutamate HomeostasisIncreasing the GluN2A/GluN2B Ratio in Neurons of the Mouse Basal and Lateral Amygdala Inhibits the Modification of an Existing Fear Memory Trace.Involvement of amygdalar protein kinase A, but not calcium/calmodulin-dependent protein kinase II, in the reconsolidation of cocaine-related contextual memories in rats.Basolateral amygdala involvement in memory reconsolidation processes that facilitate drug context-induced cocaine seeking.Dorsal hippocampal regulation of memory reconsolidation processes that facilitate drug context-induced cocaine-seeking behavior in ratsMechanisms governing the reactivation-dependent destabilization of memories and their role in extinction.Common influences of non-competitive NMDA receptor antagonists on the consolidation and reconsolidation of cocaine-cue memory.Reconsolidation of drug memories.Memory reconsolidation in aversive and appetitive settings.NMDAR dependent intracellular responses associated with cocaine conditioned place preference behavior.Reduction in Responding for Sucrose and Cocaine Reinforcement by Disruption of Memory Reconsolidation(1,2,3).On the transition from reconsolidation to extinction of contextual fear memories.Double dissociation of the requirement for GluN2B- and GluN2A-containing NMDA receptors in the destabilization and restabilization of a reconsolidating memory.The effect of propranolol and midazolam on the reconsolidation of a morphine place preference in chronically treated rats.Endocannabinoids underlie reconsolidation of hedonic memories in Wistar rats.Infralimbic GluN2A-Containing NMDA Receptors Modulate Reconsolidation of Cocaine Self-Administration Memory.NMDA receptor glycine modulatory site in the ventral tegmental area regulates the acquisition, retrieval, and reconsolidation of cocaine reward memory.Cocaine conditioned place preference: unexpected suppression of preference due to testing combined with strong conditioning.Neuronal metabolomics by ion mobility mass spectrometry in cocaine self-administering rats after early and late withdrawal.Saccharin fading is not required for the acquisition of alcohol self-administration, and can alter the dynamics of cue-alcohol memory reconsolidation.The effect of postretrieval extinction of nicotine pavlovian memories in rats trained to self-administer nicotine.
P2860
Q27023378-CAF48FB2-CB8F-45EC-A9F7-FA75DBBB82FFQ27305155-39F02753-33DF-4613-83A0-9DCF32F7E1D4Q27319794-2E1E1A49-76C3-47D7-97D3-897DD0C5C7BCQ28575977-D67FB8EA-C3DE-454A-B060-308E8C6AEC1BQ30697174-7B41A595-17A0-47BC-B4FA-E8DF17526919Q33551307-B3EF53F9-7893-437F-8A34-6E636466E2D7Q33771966-2DC54F38-17F7-4244-B08C-92D015EAB5ACQ33897247-DF48EAB4-A704-4C08-98F7-257039F51BB7Q34258049-E07DBA69-C474-423D-A4A1-EE58FDA050D7Q34277629-A8CA74A1-8730-4145-8E45-9C4E28EBCAA3Q34787716-E9CAFB1E-0B53-4717-A08D-8E45A0C71825Q34794146-641A0337-6EE0-412E-A3A9-93A95F31762FQ35112659-48EFE8AC-4746-41B3-B97E-AA6DB5C201B9Q35528967-35C6F983-45BC-478B-AD04-B75836F8E489Q35937208-5EE906C5-2E27-4D5F-A226-4C6C221E12BDQ36872261-716C78CB-1131-4E82-AFE1-1AF497182A8AQ36901159-24FDF609-D9F1-4458-8DDC-9929E918AA37Q36917021-71CA17E7-B742-4510-A8FD-088A90AD2AD4Q37062547-AC429D1A-1185-424E-9F1B-20C99290D52AQ37236018-992422FA-5342-4C70-9270-7C752D43B37EQ37366144-A10C1D57-BC0A-48C8-911D-BB35F1C023B4Q37380938-27ED15FA-A427-4665-BB49-DD8C386E1AAAQ37380945-7BB9945D-1959-445E-9D84-37B5C8703E9BQ37414015-F2DE571F-0CE4-4BC6-87CF-12C877A2B4A6Q37568031-96FD8840-879C-4DE1-98BF-91CD66FAF41BQ37985308-7DA232B6-BC85-43F3-91CF-B5207B9AB971Q38140857-4DC1BEF6-B1EB-4ADE-9906-43412A009A45Q38804849-AF9CE0DE-DA56-4C9D-9F96-F41324FB8877Q41190880-9691C2BE-2A6E-4647-B2DF-822F5F59097FQ41589613-F58CF434-E21A-4133-AA95-E4E2BDF01BCBQ41821754-4C34146B-450F-4633-819F-51D2FC04D387Q42853652-E1F9E878-17DC-4F25-BAF6-9277EA64F726Q44738063-AE2B75E6-FB85-43AE-9D35-E3799A88AB6FQ47347121-EEF8AA94-3500-431A-B041-FCCCB9635C84Q48211785-5DF2E690-173B-4F23-A4A3-72A62974C38FQ48273174-2DB9E375-7968-41DE-AF48-C8CB4298C66EQ48797991-AE63DA55-5F3F-41CD-8F4B-42D508C3AFD1Q50108083-F7FC4A52-B3A8-43D8-A87E-E64C460575F6Q50647954-355683E8-53A7-48AC-AB4A-3C9AB7967C6D
P2860
The NMDA antagonist MK-801 disrupts reconsolidation of a cocaine-associated memory for conditioned place preference but not for self-administration in rats.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 02 December 2008
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
The NMDA antagonist MK-801 dis ...... r self-administration in rats.
@en
The NMDA antagonist MK-801 dis ...... r self-administration in rats.
@nl
type
label
The NMDA antagonist MK-801 dis ...... r self-administration in rats.
@en
The NMDA antagonist MK-801 dis ...... r self-administration in rats.
@nl
prefLabel
The NMDA antagonist MK-801 dis ...... r self-administration in rats.
@en
The NMDA antagonist MK-801 dis ...... r self-administration in rats.
@nl
P2093
P2860
P356
P1433
P1476
The NMDA antagonist MK-801 dis ...... r self-administration in rats.
@en
P2093
Barbara A Sorg
Brian R Lee
Travis E Brown
P2860
P304
P356
10.1101/LM.1152808
P577
2008-12-02T00:00:00Z