JC virus antibody status underestimates infection rates.
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Stratification and monitoring of natalizumab-associated progressive multifocal leukoencephalopathy risk: recommendations from an expert groupThe importance of mouse models to define immunovirologic determinants of progressive multifocal leukoencephalopathyImmune System Involvement in the Pathogenesis of JC Virus Induced PML: What is Learned from Studies of Patients with Underlying Diseases and Therapies as Risk FactorsViral miRNAs in plasma and urine divulge JC polyomavirus infection.Antibodies reacting with JCPyV_VP2 _167-15mer as a novel serological marker for JC polyomavirus infection.JC virus antibody index in natalizumab-treated patients: correlations with John Cunningham virus DNA and C-reactive protein levelAnti-JC virus antibody levels in serum or plasma further define risk of natalizumab-associated progressive multifocal leukoencephalopathy.Late Occurrence of PML in a Patient Treated for Lymphoma with Immunomodulatory Chemotherapies, Bendamustine, Rituximab, and Ibritumomab Tiuxetan.JC polyomavirus infection is strongly controlled by human leucocyte antigen class II variantsOpportunistic DNA Recombination With Epstein-Barr Virus at Sites of Control Region Rearrangements Mediating JC Virus Neurovirulence.Diagnostic assays for polyomavirus JC and progressive multifocal leukoencephalopathy5-HT2 receptors facilitate JC polyomavirus entry.First-line natalizumab in multiple sclerosis: rationale, patient selection, benefits and risks.Immune surveillance and response to JC virus infection and PML.The human JC polyomavirus (JCPyV): virological background and clinical implications.Requirement for safety monitoring for approved multiple sclerosis therapies: an overview.Progressive multifocal leukoencephalopathy in multiple sclerosis.Management of disease-modifying treatments in neurological autoimmune diseases of the central nervous system.Natalizumab: risk stratification of individual patients with multiple sclerosis.Sorting out the risks in progressive multifocal leukoencephalopathy.A risk classification for immunosuppressive treatment-associated progressive multifocal leukoencephalopathy.Treatment-Related Progressive Multifocal Leukoencephalopathy in Multiple Sclerosis: A Comprehensive Review of Current Evidence and Future Needs.Host cell virus entry mechanisms enhance anti-JCV-antibody switch in natalizumab-treated multiple sclerosis patients.Natalizumab treatment of multiple sclerosis: new insights.Polyomavirus detection in multiple sclerosis patients under natalizumab therapy: Profile and frequency of urinary shedding.Diagnosis and Treatment of Progressive Multifocal Leukoencephalopathy Associated with Multiple Sclerosis Therapies.Anti-JCV serology during natalizumab treatment: Review and meta-analysis of 17 independent patient cohorts analyzing anti-John Cunningham polyoma virus sero-conversion rates under natalizumab treatment and differences between technical and biologicaAnti-JC virus seroprevalence in a Finnish MS cohort.Natalizumab-treated patients at high risk for PML persistently excrete JC polyomavirus.Therapy with natalizumab is associated with high JCV seroconversion and rising JCV index values.Markers of JC virus infection in patients with multiple sclerosis under natalizumab therapy.Inter- and intralaboratory comparison of JC polyomavirus antibody testing using two different virus-like particle-based assays.JC virus urinary excretion and seroprevalence in natalizumab-treated multiple sclerosis patients.Analysis of CD4+ CD8+ double-positive T cells in blood, cerebrospinal fluid and multiple sclerosis lesions.Nearly one-half of Brazilian patients with multiple sclerosis using natalizumab are DNA-JC virus positive.Modeling probability of additional cases of natalizumab-associated JCV sero-negative progressive multifocal leukoencephalopathy.Finger pointing to JC virus: a tale of two indexes.Two demyelinating diseases in the brain of a single patient, PML and MS: how to minimize 'one' while treating the 'other'."All the soarings of my mind begin in my blood:" central nervous system complication of Waldenström macroglobulinemia.JC Virus-DNA Detection Is Associated with CD8 Effector Accumulation in Peripheral Blood of Patients with Multiple Sclerosis under Natalizumab Treatment, Independently from JC Virus Serostatus.
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JC virus antibody status underestimates infection rates.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on July 2013
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
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name
JC virus antibody status underestimates infection rates.
@en
JC virus antibody status underestimates infection rates.
@nl
type
label
JC virus antibody status underestimates infection rates.
@en
JC virus antibody status underestimates infection rates.
@nl
prefLabel
JC virus antibody status underestimates infection rates.
@en
JC virus antibody status underestimates infection rates.
@nl
P2093
P2860
P356
P1433
P1476
JC virus antibody status underestimates infection rates.
@en
P2093
Craig S Miller
Joseph R Berger
Julie Gurwell
Nubia Vega
Robert J Danaher
Sidney A Houff
P2860
P356
10.1002/ANA.23893
P577
2013-07-01T00:00:00Z