Host cell virus entry mechanisms enhance anti-JCV-antibody switch in natalizumab-treated multiple sclerosis patients.

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Host cell virus entry mechanisms enhance anti-JCV-antibody switch in natalizumab-treated multiple sclerosis patients.

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http://www.wikidata.org/entity/Q38868240

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2016 nî lūn-bûn

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2016年の論文

@ja

2016年論文

@yue

2016年論文

@zh-hant

2016年論文

@zh-hk

2016年論文

@zh-mo

2016年論文

@zh-tw

2016年论文

@wuu

2016年论文

@zh

2016年论文

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Host cell virus entry mechanis ...... d multiple sclerosis patients.

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Host cell virus entry mechanis ...... d multiple sclerosis patients.

@en

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Host cell virus entry mechanis ...... d multiple sclerosis patients.

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P1476

Host cell virus entry mechanis ...... ed multiple sclerosis patients

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P2093

Anat Achiron

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David Magalashvili

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Mark Dolev

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Rina Zilkha-Falb

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Yael Stern

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P2860

The huntingtin interacting protein HIP1 is a clathrin and alpha-adaptin-binding protein involved in receptor-mediated endocytosis

Molecular biology, epidemiology, and pathogenesis of progressive multifocal leukoencephalopathy, the JC virus-induced demyelinating disease of the human brain

New insights on human polyomavirus JC and pathogenesis of progressive multifocal leukoencephalopathy

The human polyomavirus, JCV, uses serotonin receptors to infect cells

A randomized, placebo-controlled trial of natalizumab for relapsing multiple sclerosis

A general framework for weighted gene co-expression network analysis

Transcription factor Spi-B binds unique sequences present in the tandem repeat promoter/enhancer of JC virus and supports viral activity

Risk of natalizumab-associated progressive multifocal leukoencephalopathy.

A JC virus-induced signal is required for infection of glial cells by a clathrin- and eps15-dependent pathway

The Ets protein Spi-B is expressed exclusively in B cells and T cells during development.

P2888

s13365-016-0445-4

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736-746

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scholarly article

P356

10.1007/S13365-016-0445-4

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P433

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P478

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P50

Michael Gurevich

Gadi Miron

P577

2016-05-11T00:00:00Z

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P698

27170332

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