Immunosuppression after traumatic or ischemic CNS damage: it is neuroprotective and illuminates the role of microglial cells.
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Rational modulation of the innate immune system for neuroprotection in ischemic strokeCaloric restriction suppresses microglial activation and prevents neuroapoptosis following cortical injury in ratsmTOR inhibition suppresses established epilepsy in a mouse model of cortical dysplasia.Acute neuroimmune modulation attenuates the development of anxiety-like freezing behavior in an animal model of traumatic brain injuryReversal of established traumatic brain injury-induced, anxiety-like behavior in rats after delayed, post-injury neuroimmune suppressionInfluence of stochastic gene expression on the cell survival rheostat after traumatic brain injuryEffect of Chronic Administration of Low Dose Rapamycin on Development and Immunity in Young RatsProfile of minocycline neuroprotection in bilirubin-induced auditory system dysfunctionATP and NO dually control migration of microglia to nerve lesions.Hyperoxic reperfusion after global cerebral ischemia promotes inflammation and long-term hippocampal neuronal deathGenetic analysis of the role of tumor necrosis factor receptors in functional outcome after traumatic brain injury in mice.Role of Akt and mammalian target of rapamycin in functional outcome after concussive brain injury in miceCannabinoid receptor type-2 stimulation, blockade, and deletion alter the vascular inflammatory responses to traumatic brain injury.The cytokine response to human traumatic brain injury: temporal profiles and evidence for cerebral parenchymal production.Rapamycin reverses status epilepticus-induced memory deficits and dendritic damage.Protective Effects of Quercetin on Mitochondrial Biogenesis in Experimental Traumatic Brain Injury via the Nrf2 Signaling Pathway.Anti-inflammatory Activity of 1-docosanoyl Cafferate Isolated from Rhus verniciflua in LPS-stimulated BV2 Microglial CellsHyaluronic acid-based hydrogel enhances neuronal survival in spinal cord slice cultures from postnatal mice.Effect of titanium dioxide nanoparticles on zebrafish embryos and developing retinaRapamycin treatment improves neuron viability in an in vitro model of stroke.Minocycline treatment inhibits microglial activation and alters spinal levels of endocannabinoids in a rat model of neuropathic pain.Potential of immunosuppressive agents in cerebral ischaemia.Effect of exosomes derived from multipluripotent mesenchymal stromal cells on functional recovery and neurovascular plasticity in rats after traumatic brain injuryThe pathophysiology of repetitive concussive traumatic brain injury in experimental models; new developments and open questionsMinocycline and aspirin in the treatment of bipolar depression: a protocol for a proof-of-concept, randomised, double-blind, placebo-controlled, 2x2 clinical trial3,4,5-Trihydroxycinnamic Acid Inhibits LPS-Induced iNOS Expression by Suppressing NF-κB Activation in BV2 Microglial CellsThe role of microglia in the neurogenesis of zebrafish retinaTime dependent neuroprotection of mycophenolate mofetil: effects on temporal dynamics in glial proliferation, apoptosis, and scar formation.Neural progenitor cell proliferation in the hypothalamus is involved in acquired heat tolerance in long-term heat-acclimated rats.Neuroprotective effect of Shenqi Fuzheng injection pretreatment in aged rats with cerebral ischemia/reperfusion injuryB-cell maturation antigen, a proliferation-inducing ligand, and B-cell activating factor are candidate mediators of spinal cord injury-induced autoimmunityMultipotent mesenchymal stromal cells decrease transforming growth factor β1 expression in microglia/macrophages and down-regulate plasminogen activator inhibitor 1 expression in astrocytes after stroke.3,4,5-Trihydroxycinnamic Acid Inhibits Lipopolysaccharide-Induced Inflammatory Response through the Activation of Nrf2 Pathway in BV2 Microglial Cells.Increased activation of Iba1+ microglia in pediatric epilepsy patients with Rasmussen's encephalitis compared with cortical dysplasia and tuberous sclerosis complex.The role of macrophages in optic nerve regeneration.Modulation of brain hemichannels and gap junction channels by pro-inflammatory agents and their possible role in neurodegenerationIPAF inflammasome is involved in interleukin-1β production from astrocytes, induced by palmitate; implications for Alzheimer's Disease.Post-ischemic brain damage: pathophysiology and role of inflammatory mediators.Neurotrophic natural products: chemistry and biology.Inflammation in neurodegenerative diseases.
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P2860
Immunosuppression after traumatic or ischemic CNS damage: it is neuroprotective and illuminates the role of microglial cells.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 23 December 2007
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Immunosuppression after trauma ...... the role of microglial cells.
@en
Immunosuppression after trauma ...... the role of microglial cells.
@nl
type
label
Immunosuppression after trauma ...... the role of microglial cells.
@en
Immunosuppression after trauma ...... the role of microglial cells.
@nl
prefLabel
Immunosuppression after trauma ...... the role of microglial cells.
@en
Immunosuppression after trauma ...... the role of microglial cells.
@nl
P1476
Immunosuppression after trauma ...... the role of microglial cells.
@en
P2093
Nils P Hailer
P304
P356
10.1016/J.PNEUROBIO.2007.12.001
P577
2007-12-23T00:00:00Z