Deletion of the type-1 interferon receptor in APPSWE/PS1ΔE9 mice preserves cognitive function and alters glial phenotype - Wikidata - awgldk - TriplyDB

Deletion of the type-1 interferon receptor in APPSWE/PS1ΔE9 mice preserves cognitive function and alters glial phenotype

Deletion of the type-1 interferon receptor in APPSWE/PS1ΔE9 mice preserves cognitive function and alters glial phenotype

http://www.wikidata.org/entity/Q37084675

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Down-Regulated Drebrin Aggravates Cognitive Impairments in a Mouse Model of Alzheimer's Disease.Immunoproteasome deficiency alters microglial cytokine response and improves cognitive deficits in Alzheimer's disease-like APPPS1 mice.Type-I interferon pathway in neuroinflammation and neurodegeneration: focus on Alzheimer's disease.Essential Dietary Bioactive Lipids in Neuroinflammatory Diseases.Effective expression of Drebrin in hippocampus improves cognitive function and alleviates lesions of Alzheimer's disease in APP (swe)/PS1 (ΔE9) mice.Type-I interferons mediate the neuroinflammatory response and neurotoxicity induced by rotenone.Microbial Immuno-Communication in Neurodegenerative Diseases Type I interferon pathway in CNS homeostasis and neurological disorders.Antibiotic-induced perturbations in microbial diversity during post-natal development alters amyloid pathology in an aged APPSWE/PS1ΔE9 murine model of Alzheimer's disease.Exosomal biomarkers in Down syndrome and Alzheimer's disease.Type-I interferons in Parkinson's disease: innate inflammatory response drives fate of neurons in model of degenerative brain disorder: An editorial comment on 'Type-I interferons mediate the neuroinflammatory response and neurotoxicity induced by r

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Deletion of the type-1 interferon receptor in APPSWE/PS1ΔE9 mice preserves cognitive function and alters glial phenotype

http://www.wikidata.org/entity/Q37084675

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 11 July 2016

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Deletion of the type-1 interfe ...... ion and alters glial phenotype

@en

Deletion of the type-1 interfe ...... on and alters glial phenotype.

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type

label

Deletion of the type-1 interfe ...... ion and alters glial phenotype

@en

Deletion of the type-1 interfe ...... on and alters glial phenotype.

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prefLabel

Deletion of the type-1 interfe ...... ion and alters glial phenotype

@en

Deletion of the type-1 interfe ...... on and alters glial phenotype.

@nl

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Acta neuropathologica communications

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Deletion of the type-1 interfe ...... ion and alters glial phenotype

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Juliet M Taylor

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Kate M Brody

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Moses Zhang

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Myles R Minter

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Sandy R Shultz

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Zachery Moore

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A family of cytokine-inducible inhibitors of signalling

Innate immune recognition of viral infection

Induction of suppressor of cytokine signaling-3 by herpes simplex virus type 1 contributes to inhibition of the interferon signaling pathway

NLRP3 is activated in Alzheimer's disease and contributes to pathology in APP/PS1 mice

A rapid and sensitive method for the quantitation of microgram quantities of protein utilizing the principle of protein-dye binding

Analysis of Relative Gene Expression Data Using Real-Time Quantitative PCR and the 2−ΔΔCT Method

Regulation of type I interferon responses

Inconsistencies and controversies surrounding the amyloid hypothesis of Alzheimer's disease

Endogenous murine Aβ increases amyloid deposition in APP23 but not in APPPS1 transgenic mice.

Mutant presenilins specifically elevate the levels of the 42 residue beta-amyloid peptide in vivo: evidence for augmentation of a 42-specific gamma secretase

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10.1186/S40478-016-0341-4

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