about
Targeted disruption of SPI3/Serpinb6 does not result in developmental or growth defects, leukocyte dysfunction, or susceptibility to strokeCOPD and stroke: are systemic inflammation and oxidative stress the missing links?Weight-bearing locomotion in the developing opossum, Monodelphis domestica following spinal transection: remodeling of neuronal circuits caudal to lesionThe association of metalloendopeptidase EC 3.4.24.15 at the extracellular surface of the AtT-20 cell plasma membraneFibroblasts derived from Gpx1 knockout mice display senescent-like features and are susceptible to H2O2-mediated cell deathDiminished Akt phosphorylation in neurons lacking glutathione peroxidase-1 (Gpx1) leads to increased susceptibility to oxidative stress-induced cell deathIncreased infarct size and exacerbated apoptosis in the glutathione peroxidase-1 (Gpx-1) knockout mouse brain in response to ischemia/reperfusion injuryGlutathione peroxidase-1 primes pro-inflammatory cytokine production after LPS challenge in vivo.Absence of glutathione peroxidase-1 exacerbates cerebral ischemia-reperfusion injury by reducing post-ischemic microvascular perfusion.Reactive oxygen species enhance insulin sensitivityInsulin-regulated aminopeptidase deficiency provides protection against ischemic stroke in mice.Neural injury following stroke: are Toll-like receptors the link between the immune system and the CNS?Overexpression of the chromosome 21 transcription factor Ets2 induces neuronal apoptosis.MyD88 is a critical regulator of hematopoietic cell-mediated neuroprotection seen after stroke.Impact of oxidative stress on neuronal survival.Reactive oxygen species and the modulation of stroke.Ablation of Type-1 IFN Signaling in Hematopoietic Cells Confers Protection Following Traumatic Brain InjuryToll-like receptors in the brain and their potential roles in neuropathology.Modulation of neuro-inflammation and vascular response by oxidative stress following cerebral ischemia-reperfusion injury.Deletion of the type-1 interferon receptor in APPSWE/PS1ΔE9 mice preserves cognitive function and alters glial phenotypeReduction of cerebral infarct volume by apocynin requires pretreatment and is absent in Nox2-deficient mice.Inflammation in epileptogenesis after traumatic brain injury.Type-1 interferons contribute to oxygen glucose deprivation induced neuro-inflammation in BE(2)M17 human neuroblastoma cells.Anti-lysophosphatidic acid antibodies improve traumatic brain injury outcomes.The contribution of astrocytes and microglia to traumatic brain injuryPerturbation of the transcriptome: implications of the innate immune system in Alzheimer's disease.The contribution of neuroinflammation to amyloid toxicity in Alzheimer's disease.Type-I interferon pathway in neuroinflammation and neurodegeneration: focus on Alzheimer's disease.Type-I interferon signalling through IFNAR1 plays a deleterious role in the outcome after stroke.Effects of GDNF-loaded injectable gelatin-based hydrogels on endogenous neural progenitor cell migration.Oxidation of Iron under Physiologically Relevant Conditions in Biological Fluids from Healthy and Alzheimer's Disease Subjects.Type-1 interferon signaling mediates neuro-inflammatory events in models of Alzheimer's disease.Type-I interferons mediate the neuroinflammatory response and neurotoxicity induced by rotenone.Type-1 interferons contribute to the neuroinflammatory response and disease progression of the MPTP mouse model of Parkinson's disease.A nonfibrin macromolecular cofactor for tPA-mediated plasmin generation following cellular injury.Evidence for the recruitment of autophagic vesicles in human brain after stroke.Suppressor of cytokine signaling 1 negatively regulates Toll-like receptor signaling by mediating Mal degradation.The σ 1 receptor agonist 4-PPBP elicits ERK1/2 phosphorylation in primary neurons: a possible mechanism of neuroprotective action.Glutathione peroxidase-1 protects against cigarette smoke-induced lung inflammation in mice.Mice lacking glutathione peroxidase-1 activity show increased TUNEL staining and an accelerated inflammatory response in brain following a cold-induced injury.
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description
hulumtues
@sq
researcher
@en
wetenschapper
@nl
հետազոտող
@hy
name
Peter J Crack
@nl
Peter J Crack
@sl
Peter J. Crack
@en
Peter J. Crack
@es
type
label
Peter J Crack
@nl
Peter J Crack
@sl
Peter J. Crack
@en
Peter J. Crack
@es
prefLabel
Peter J Crack
@nl
Peter J Crack
@sl
Peter J. Crack
@en
Peter J. Crack
@es
P106
P1153
6602479944
P21
P31
P496
0000-0002-5030-0330