Transgenic mice expressing dominant-negative bright exhibit defects in B1 B cells.
about
Signalling of the BCR is regulated by a lipid rafts-localised transcription factor, Bright.Global miRNA expression analysis identifies novel key regulators of plasma cell differentiation and malignant plasma cell.Spatial congregation of STAT binding directs selective nuclear architecture during T-cell functional differentiation.Disease activity in systemic lupus erythematosus correlates with expression of the transcription factor AT-rich-interactive domain 3A.The ARID family transcription factor bright is required for both hematopoietic stem cell and B lineage development.Lin28b promotes fetal B lymphopoiesis through the transcription factor Arid3aThe transcription factor Bright plays a role in marginal zone B lymphocyte development and autoantibody production.CCL5-independent helper T lymphocyte responses to immuno-dominant pneumococcal surface protein A epitopesTransgenic expression of survivin compensates for OX40-deficiency in driving Th2 development and allergic inflammation.The Bright Side of Hematopoiesis: Regulatory Roles of ARID3a/Bright in Human and Mouse Hematopoiesis.Roles of B-1 and B-2 cells in innate and acquired IgA-mediated immunity.Role of tissue-specific AT-rich DNA sequence-binding proteins in lymphocyte differentiation.Differential expression of the transcription factor ARID3a in lupus patient hematopoietic progenitor cells.Loss of Bright/ARID3a function promotes developmental plasticity.A Role for the Transcription Factor Arid3a in Mouse B2 Lymphocyte Expansion and Peritoneal B1a Generation.B-cell regulator of immunoglobulin heavy-chain transcription (Bright)/ARID3a is a direct target of the oncomir microRNA-125b in progenitor B-cells.
P2860
Q24310569-81DD2F67-3F94-4146-B0E0-C1FF4E782A27Q33741100-DA361C57-B330-4104-B88F-25A55EF548CBQ34501482-40DCDC3F-5790-40C3-A64E-1A8053FD8B4AQ34577521-12C31109-0A08-413D-B194-880D9C741183Q34742288-5B333473-20FA-407E-9D35-EA64B356D5A6Q35289270-D9269A67-0E4F-45AC-AE98-3CF9DF30CC1EQ35490321-425745B1-B36C-4348-8794-D0910B1CBF5AQ35717991-2E253946-E6C8-4AFB-A43A-E4B10A922AC0Q37423123-C40EDE26-B123-406A-947C-54ED5A37E9F0Q37644905-81F6E4C3-50E1-4E2A-8FC1-5081758EB607Q37781395-4934F8DD-46CF-44A4-B3BE-2F648AEEF5D1Q38221005-0576F136-C83C-4AE1-9820-7429740F5EB4Q41656076-76599735-3813-4381-B33F-FFF69C0CCCA1Q41841351-8C8AB180-CE2E-4601-96D0-97FD9762D7ADQ47286421-B3C9FE82-4D43-4CAE-9185-E5DAE86ADBA8Q53271931-51851145-BD3C-41E3-9008-A95A87A871BA
P2860
Transgenic mice expressing dominant-negative bright exhibit defects in B1 B cells.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年学术文章
@wuu
2008年学术文章
@zh-cn
2008年学术文章
@zh-hans
2008年学术文章
@zh-my
2008年学术文章
@zh-sg
2008年學術文章
@yue
2008年學術文章
@zh
2008年學術文章
@zh-hant
name
Transgenic mice expressing dominant-negative bright exhibit defects in B1 B cells.
@en
Transgenic mice expressing dominant-negative bright exhibit defects in B1 B cells.
@nl
type
label
Transgenic mice expressing dominant-negative bright exhibit defects in B1 B cells.
@en
Transgenic mice expressing dominant-negative bright exhibit defects in B1 B cells.
@nl
prefLabel
Transgenic mice expressing dominant-negative bright exhibit defects in B1 B cells.
@en
Transgenic mice expressing dominant-negative bright exhibit defects in B1 B cells.
@nl
P2093
P2860
P1476
Transgenic mice expressing dominant-negative bright exhibit defects in B1 B cells.
@en
P2093
Athenia L Oldham
Carol F Webb
Cathrine Miner
Jamee C Nixon
Scott Ferrell
Ute Hochgeschwender
P2860
P304
P356
10.4049/JIMMUNOL.181.10.6913
P407
P577
2008-11-01T00:00:00Z