The AGE/RAGE axis in diabetes-accelerated atherosclerosis.
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The effect of soluble RAGE on inhibition of angiotensin II-mediated atherosclerosis in apolipoprotein E deficient miceRosiglitazone via upregulation of Akt/eNOS pathways attenuates dysfunction of endothelial progenitor cells, induced by advanced glycation end products.Modulation of cell proliferation, survival and gene expression by RAGE and TLR signaling in cells of the innate and adaptive immune response: role of p38 MAPK and NF-KB.Ghrelin inhibits AGEs-induced apoptosis in human endothelial cells involving ERK1/2 and PI3K/Akt pathways.Diabetes alters activation and repression of pro- and anti-inflammatory signaling pathways in the vasculature.Relating form and function of EF-hand calcium binding proteins.Selective Inhibition on RAGE-binding AGEs Required by Bioactive Peptide Alpha-S2 Case in Protein from Goat Ethawah Breed Milk: Study of Biological Modeling.Consequences of Aberrant Insulin Regulation in the Brain: Can Treating Diabetes be Effective for Alzheimer's Disease.Advanced glycation end products (AGEs) in relation to atherosclerotic lipid profiles in middle-aged and elderly diabetic patientsEsculin improves dyslipidemia, inflammation and renal damage in streptozotocin-induced diabetic rats.Shear stress modulates RAGE-mediated inflammation in a model of diabetes-induced metabolic stress.Advanced glycation end products impair the migration, adhesion and secretion potentials of late endothelial progenitor cells.The pathogenic role of Maillard reaction in the aging eye.AGEs Decreased SIRT3 Expression and SIRT3 Activation Protected AGEs-Induced EPCs' Dysfunction and Strengthened Anti-oxidant Capacity.Glycolaldehyde-modified β-lactoglobulin AGEs are unable to stimulate inflammatory signaling pathways in RAGE-expressing human cell lines.The Influence of Glycated Hemoglobin on the Cross Susceptibility Between Type 1 Diabetes Mellitus and Periodontal Disease.Perturbation of human coronary artery endothelial cell redox state and NADPH generation by methylglyoxal.Mechanisms linking brain insulin resistance to Alzheimer's disease.Advanced glycation end-products induce heparanase expression in endothelial cells by the receptor for advanced glycation end products and through activation of the FOXO4 transcription factor.
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P2860
The AGE/RAGE axis in diabetes-accelerated atherosclerosis.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on March 2008
@en
vedecký článok
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vetenskaplig artikel
@sv
videnskabelig artikel
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vědecký článek
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name
The AGE/RAGE axis in diabetes-accelerated atherosclerosis.
@en
The AGE/RAGE axis in diabetes-accelerated atherosclerosis.
@nl
type
label
The AGE/RAGE axis in diabetes-accelerated atherosclerosis.
@en
The AGE/RAGE axis in diabetes-accelerated atherosclerosis.
@nl
prefLabel
The AGE/RAGE axis in diabetes-accelerated atherosclerosis.
@en
The AGE/RAGE axis in diabetes-accelerated atherosclerosis.
@nl
P2860
P1476
The AGE/RAGE axis in diabetes-accelerated atherosclerosis
@en
P2093
Aino Soro-Paavonen
Karin Jandeleit-Dahm
P2860
P304
P356
10.1111/J.1440-1681.2007.04875.X
P50
P577
2008-03-01T00:00:00Z