Roles for cytosolic NADPH redox in regulating pulmonary artery relaxation by thiol oxidation-elicited subunit dimerization of protein kinase G1α
about
Redox regulation of cGMP-dependent protein kinase Iα in the cardiovascular system20-HETE-induced mitochondrial superoxide production and inflammatory phenotype in vascular smooth muscle is prevented by glucose-6-phosphate dehydrogenase inhibition.Dehydroepiandrosterone promotes pulmonary artery relaxation by NADPH oxidation-elicited subunit dimerization of protein kinase G 1α.Novel role for protein kinase g oxidative activation in the vasodilator and antihypertensive actions of hydrogen sulfide.Redox Mechanisms Influencing cGMP Signaling in Pulmonary Vascular Physiology and Pathophysiology.
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Roles for cytosolic NADPH redox in regulating pulmonary artery relaxation by thiol oxidation-elicited subunit dimerization of protein kinase G1α
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 24 May 2013
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Roles for cytosolic NADPH redo ...... rization of protein kinase G1α
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Roles for cytosolic NADPH redo ...... ization of protein kinase G1α.
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type
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Roles for cytosolic NADPH redo ...... rization of protein kinase G1α
@en
Roles for cytosolic NADPH redo ...... ization of protein kinase G1α.
@nl
prefLabel
Roles for cytosolic NADPH redo ...... rization of protein kinase G1α
@en
Roles for cytosolic NADPH redo ...... ization of protein kinase G1α.
@nl
P2093
P2860
P1476
Roles for cytosolic NADPH redo ...... rization of protein kinase G1α
@en
P2093
Boon Hwa Neo
Dhara Patel
Michael S Wolin
Sharath Kandhi
P2860
P304
P356
10.1152/AJPHEART.01010.2011
P577
2013-05-24T00:00:00Z