Protein kinase A anchoring via AKAP150 is essential for TRPV1 modulation by forskolin and prostaglandin E2 in mouse sensory neurons.
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The mechanism of μ-opioid receptor (MOR)-TRPV1 crosstalk in TRPV1 activation involves morphine anti-nociception, tolerance and dependenceTRPV1 and SP: key elements for sepsis outcome?TNFα induces co-trafficking of TRPV1/TRPA1 in VAMP1-containing vesicles to the plasmalemma via Munc18-1/syntaxin1/SNAP-25 mediated fusion.The protein phosphatase 2A regulatory subunits B'beta and B'delta mediate sustained TrkA neurotrophin receptor autophosphorylation and neuronal differentiationNerve growth factor mediates a switch in intracellular signaling for PGE2-induced sensitization of sensory neurons from protein kinase A to Epacβ-Arrestin-2 desensitizes the transient receptor potential vanilloid 1 (TRPV1) channel.AKAP150-mediated TRPV1 sensitization is disrupted by calcium/calmodulin.A-kinase anchoring in dendritic cells is required for antigen presentationMitochondrial Ca2+ cycling facilitates activation of the transcription factor NFAT in sensory neurons.Nociceptive sensitization by complement C5a and C3a in mouse.The G2A receptor (GPR132) contributes to oxaliplatin-induced mechanical pain hypersensitivity.A-Kinase Anchoring Protein 79/150 Scaffolds Transient Receptor Potential A 1 Phosphorylation and Sensitization by Metabotropic Glutamate Receptor Activation.Laser modulation of heat and capsaicin receptor TRPV1 leads to thermal antinociception.The basal thermal sensitivity of the TRPV1 ion channel is determined by PKCβII.[EXPRESS] Epac and Nociceptor Sensitization.Prostaglandin E₂ regulates AMPA receptor phosphorylation and promotes membrane insertion in preoptic area neurons and glia during sexual differentiationAfferent nerves regulating the cough reflex: mechanisms and mediators of cough in disease.Ionic mechanisms underlying inflammatory mediator-induced sensitization of dural afferents.Mapping the binding site of TRPV1 on AKAP79: implications for inflammatory hyperalgesia.Red fluorescent genetically encoded Ca2+ indicators for use in mitochondria and endoplasmic reticulumNerve growth factor (NGF) regulates activity of nuclear factor of activated T-cells (NFAT) in neurons via the phosphatidylinositol 3-kinase (PI3K)-Akt-glycogen synthase kinase 3β (GSK3β) pathwayTargeting protein-protein interactions within the cyclic AMP signaling system as a therapeutic strategy for cardiovascular disease.PP2B/calcineurin-mediated desensitization of TRPV1 does not require AKAP150.TRPV1 activation is required for hypertonicity-stimulated inflammatory cytokine release in human corneal epithelial cells.Peripheral group I metabotropic glutamate receptor activation leads to muscle mechanical hyperalgesia through TRPV1 phosphorylation in the rat.Subgroup-elimination transcriptomics identifies signaling proteins that define subclasses of TRPV1-positive neurons and a novel paracrine circuit.A-kinase anchoring protein 150 mediates transient receptor potential family V type 1 sensitivity to phosphatidylinositol-4,5-bisphosphateResolving TRPV1- and TNF-α-mediated spinal cord synaptic plasticity and inflammatory pain with neuroprotectin D1.Modality-specific mechanisms of protein kinase C-induced hypersensitivity of TRPV1: S800 is a polymodal sensitization site.TRPV1 channel inhibition contributes to the antinociceptive effects of Croton macrostachyus extract in miceNew insights into protease-activated receptor 4 signaling pathways in the pathogenesis of inflammation and neuropathic pain: a literature reviewThe C-type natriuretic peptide induces thermal hyperalgesia through a noncanonical Gβγ-dependent modulation of TRPV1 channelEstrogen modulation of visceral nociceptors.Persistent Electrical Activity in Primary Nociceptors after Spinal Cord Injury Is Maintained by Scaffolded Adenylyl Cyclase and Protein Kinase A and Is Associated with Altered Adenylyl Cyclase Regulation.Scaffolding by A-kinase anchoring protein enhances functional coupling between adenylyl cyclase and TRPV1 channel.The contribution of Kv2.2-mediated currents decreases during the postnatal development of mouse dorsal root ganglion neuronsThe Complement System Component C5a Produces Thermal Hyperalgesia via Macrophage-to-Nociceptor Signaling That Requires NGF and TRPV1Abnormal trigeminal sensory processing in obese mice.The glutamatergic nature of TRPV1-expressing neurons in the spinal dorsal hornSustained morphine treatment augments capsaicin-evoked calcitonin gene-related peptide release from primary sensory neurons in a protein kinase A- and Raf-1-dependent manner.
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Protein kinase A anchoring via AKAP150 is essential for TRPV1 modulation by forskolin and prostaglandin E2 in mouse sensory neurons.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
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artigo científico
@pt
bilimsel makale
@tr
scientific article published on May 2008
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
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vědecký článek
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name
Protein kinase A anchoring via ...... n E2 in mouse sensory neurons.
@en
Protein kinase A anchoring via ...... n E2 in mouse sensory neurons.
@nl
type
label
Protein kinase A anchoring via ...... n E2 in mouse sensory neurons.
@en
Protein kinase A anchoring via ...... n E2 in mouse sensory neurons.
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prefLabel
Protein kinase A anchoring via ...... n E2 in mouse sensory neurons.
@en
Protein kinase A anchoring via ...... n E2 in mouse sensory neurons.
@nl
P2093
P2860
P1476
Protein kinase A anchoring via ...... n E2 in mouse sensory neurons.
@en
P2093
Blake Nichols
G Stanley McKnight
Johannes W Hell
Katrin Schnizler
Leonid P Shutov
Michael J Van Kanegan
Michelle A Merrill
Stefan Strack
Yuriy M Usachev
P2860
P304
P356
10.1523/JNEUROSCI.0233-08.2008
P407
P577
2008-05-01T00:00:00Z