Identification of a specific self-reactive IgM antibody that initiates intestinal ischemia/reperfusion injury. - Wikidata - awgldk - TriplyDB

Identification of a specific self-reactive IgM antibody that initiates intestinal ischemia/reperfusion injury.

Identification of a specific self-reactive IgM antibody that initiates intestinal ischemia/reperfusion injury.

http://www.wikidata.org/entity/Q37095150

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IgM production by bone marrow plasmablasts contributes to long-term protection against intracellular bacterial infection Intestinal alkaline phosphatase is a gut mucosal defense factor maintained by enteral nutrition Natural antibody and complement mediate neutralization of influenza virus in the absence of prior immunity Beta-glucan enhances complement-mediated hematopoietic recovery after bone marrow injury Role of Natural Autoantibodies and Natural IgM Anti-Leucocyte Autoantibodies in Health and Disease Life and death at the mucosal-luminal interface: New perspectives on human intestinal ischemia-reperfusion Complement in the Homeostatic and Ischemic Brain How dying cells alert the immune system to danger Natural IgM anti-leukocyte autoantibodies attenuate excess inflammation mediated by innate and adaptive immune mechanisms involving Th-17 Analysis of factorial time-course microarrays with application to a clinical study of burn injury.Pathogenic natural antibodies recognizing annexin IV are required to develop intestinal ischemia-reperfusion injury.B lymphocytes trigger monocyte mobilization and impair heart function after acute myocardial infarction.Mobilization studies in complement-deficient mice reveal that optimal AMD3100 mobilization of hematopoietic stem cells depends on complement cascade activation by AMD3100-stimulated granulocytes.Natural IgM antibodies that bind neoepitopes exposed as a result of spinal cord injury , drive secondary injury by activating complement Human intestinal ischemia-reperfusion-induced inflammation characterized: experiences from a new translational model.Inhibition of rat gut reperfusion injury with an agent developed for the mouse. Evidence that amplification of injury by innate immunity is conserved between two animal species.Poor Mobilization in T-Cell-Deficient Nude Mice Is Explained by Defective Activation of Granulocytes and Monocytes.Targeted complement inhibition by C3d recognition ameliorates tissue injury without apparent increase in susceptibility to infection.Blockade of self-reactive IgM significantly reduces injury in a murine model of acute myocardial infarction Innate immunity alone is not sufficient for chronic rejection but predisposes healed allografts to T cell-mediated pathology Targeting of mannan-binding lectin-associated serine protease-2 confers protection from myocardial and gastrointestinal ischemia/reperfusion injury.Non-MHC antigenic targets of the humoral immune response in transplantation The role of platelet factor 4 in local and remote tissue damage in a mouse model of mesenteric ischemia/reperfusion injury.Attenuation of the effects of rat hemorrhagic shock with a reperfusion injury-inhibiting agent specific to mice.Domain V peptides inhibit beta2-glycoprotein I-mediated mesenteric ischemia/reperfusion-induced tissue damage and inflammation.Sca-1 influences the innate immune response during skeletal muscle regeneration.Oxidative stress sensitizes retinal pigmented epithelial (RPE) cells to complement-mediated injury in a natural antibody-, lectin pathway-, and phospholipid epitope-dependent manner.In situ IgM production and clonal expansion of B-1 cells in peritoneal cavity promote elimination of C. albicans infection in IgH transgenic mice with VH derived from a natural antibody.The inflammatory response to cell death.TLR2 modulates antibodies required for intestinal ischemia/reperfusion-induced damage and inflammation Natural and disease-specific autoantibodies in chronic obstructive pulmonary disease Targeting pathogenic postischemic self-recognition by natural IgM to protect against posttransplantation cardiac reperfusion injury A complement-IL-4 regulatory circuit controls liver regeneration.Innate response to self-antigen significantly exacerbates burn wound depth.Pathogenic natural antibodies propagate cerebral injury following ischemic stroke in mice.Somatostatin Improved B Cells Mature in Macaques during Intestinal Ischemia-Reperfusion.Manipulation of the glycan-specific natural antibody repertoire for immunotherapy Natural and adaptive IgM antibodies in the recognition of tumor-associated antigens of breast cancer (Review).The Fifth Domain of Beta 2 Glycoprotein I Protects from Natural IgM Mediated Cardiac Ischaemia Reperfusion Injury Immune response to lipoproteins in atherosclerosis.

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Identification of a specific self-reactive IgM antibody that initiates intestinal ischemia/reperfusion injury.

http://www.wikidata.org/entity/Q37095150

description

article científic

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artigo científico

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bilimsel makale

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scientific article published on 03 March 2004

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Identification of a specific s ...... l ischemia/reperfusion injury.

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Identification of a specific s ...... l ischemia/reperfusion injury.

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Identification of a specific s ...... l ischemia/reperfusion injury.

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Identification of a specific s ...... l ischemia/reperfusion injury.

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Identification of a specific s ...... l ischemia/reperfusion injury.

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Identification of a specific s ...... l ischemia/reperfusion injury.

@nl

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PNAS.0400347101

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Proceedings of the National Academy of Sciences of the United States of America

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Identification of a specific s ...... l ischemia/reperfusion injury.

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Elisabeth M Alicot

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Francis D Moore

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Herbert B Hechtman

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Isaac Chiu

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Michael C Carroll

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Min Xu

http://www.w3.org/2001/XMLSchema#string

Ming Zhang

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Minghe Ma

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Nicola Verna

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Rachel Hung

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P2860

Origin of murine B cell lineages

Natural antibodies and complement link innate and acquired immunity.

B-1 cell definition.

B-1 cell origins and VH repertoire determination.

Reactive species mechanisms of cellular hypoxia-reoxygenation injury.

Reperfusion injury of ischemic skeletal muscle is mediated by natural antibody and complement.

B-1 and B-2 cell-derived immunoglobulin M antibodies are nonredundant components of the protective response to influenza virus infection

Effects of breeding environments on generation and activation of autoreactive B-1 cells in anti-red blood cell autoantibody transgenic mice.

Frequent occurrence of identical heavy and light chain Ig rearrangements.

Anti-red blood cell autoantibody transgenic mice: murine model of autoimmune hemolytic anemia.

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3886-3891

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scholarly article

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10.1073/PNAS.0400347101

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11

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101

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2004-03-03T00:00:00Z

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8679913

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14999103

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reperfusion injury

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374339

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