Pathogenic natural antibodies propagate cerebral injury following ischemic stroke in mice.
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Complement in the Homeostatic and Ischemic BrainB-1 Cell Heterogeneity and the Regulation of Natural and Antigen-Induced IgM ProductionDeficiency of complement receptors CR2/CR1 in Cr2⁻/⁻ mice reduces the extent of secondary brain damage after closed head injury.Natural IgM antibodies that bind neoepitopes exposed as a result of spinal cord injury , drive secondary injury by activating complementMembrane lipid interactions in intestinal ischemia/reperfusion-induced InjuryOxidative stress sensitizes retinal pigmented epithelial (RPE) cells to complement-mediated injury in a natural antibody-, lectin pathway-, and phospholipid epitope-dependent manner.Targeting pathogenic postischemic self-recognition by natural IgM to protect against posttransplantation cardiac reperfusion injuryThe Fifth Domain of Beta 2 Glycoprotein I Protects from Natural IgM Mediated Cardiac Ischaemia Reperfusion InjuryA Rich-Club Organization in Brain Ischemia Protein Interaction NetworkIgM exacerbates glomerular disease progression in complement-induced glomerulopathyThe alternative complement pathway propagates inflammation and injury in murine ischemic strokeNatural IgM Blockade Limits Infarct Expansion and Left Ventricular Dysfunction in a Swine Myocardial Infarct ModelTargeted inhibition of complement using complement receptor 2-conjugated inhibitors attenuates EAE.Method parameters' impact on mortality and variability in mouse stroke experiments: a meta-analysis.New therapeutic and diagnostic opportunities for injured tissue-specific targeting of complement inhibitors and imaging modalities.Lung ischemia reperfusion injury: a bench-to-bedside review.A Special Connection between γδ T Cells and Natural Antibodies?Transplantation and Damage-Associated Molecular Patterns (DAMPs).Injury site-specific targeting of complement inhibitors for treating stroke.Anti-Inflammatory Targets for the Treatment of Reperfusion Injury in Stroke.Clinical hypothermia temperatures increase complement activation and cell destruction via the classical pathway.Targeting mechanisms at sites of complement activation for imaging and therapy.Therapeutic hypothermia modulates complement factor C3a and C5a levels in a rat model of hypoxic ischemic encephalopathy.Natural IgM initiates an inflammatory response important for both hepatic ischemia reperfusion injury and regeneration.Inhibition of Complement Drives Increase in Early Growth Response Proteins and Neuroprotection Mediated by Salidroside After Cerebral Ischemia.Thromboembolic Model of Cerebral Ischemia and Reperfusion in Mice.Natural Autoantibodies: An Undersugn Hero of the Immune System and Autoimmune Disorders—A Point of View.
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P2860
Pathogenic natural antibodies propagate cerebral injury following ischemic stroke in mice.
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
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2011年论文
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2011年论文
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name
Pathogenic natural antibodies ...... owing ischemic stroke in mice.
@ast
Pathogenic natural antibodies ...... owing ischemic stroke in mice.
@en
type
label
Pathogenic natural antibodies ...... owing ischemic stroke in mice.
@ast
Pathogenic natural antibodies ...... owing ischemic stroke in mice.
@en
prefLabel
Pathogenic natural antibodies ...... owing ischemic stroke in mice.
@ast
Pathogenic natural antibodies ...... owing ischemic stroke in mice.
@en
P2093
P2860
P356
P1476
Pathogenic natural antibodies ...... owing ischemic stroke in mice.
@en
P2093
Andrew Elvington
Carl Atkinson
Liudmila Kulik
Mark S Kindy
Stephen Tomlinson
V Michael Holers
P2860
P304
P356
10.4049/JIMMUNOL.1102132
P407
P577
2011-12-23T00:00:00Z