Immunologic and inflammatory mechanisms that drive asthma progression to remodeling - Wikidata - awgldk - TriplyDB

Immunologic and inflammatory mechanisms that drive asthma progression to remodeling

Immunologic and inflammatory mechanisms that drive asthma progression to remodeling

http://www.wikidata.org/entity/Q37105324

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Recent advances in mechanisms and treatments of airway remodeling in asthma: a message from the bench side to the clinic Airway Remodeling in Chronic Obstructive Pulmonary Disease and Asthma: the Role of Matrix Metalloproteinase-9 Pulmonary fibrosis: pathogenesis, etiology and regulation ADAM8: a new therapeutic target for asthma Absence of alpha 4 but not beta 2 integrins restrains development of chronic allergic asthma using mouse genetic models Resolution of remodeling in eosinophilic esophagitis correlates with epithelial response to topical corticosteroids.Asthma linked with rhinosinusitis: An extensive review.CTGF upregulation correlates with MMP-9 level in airway remodeling in a murine model of asthma.What targeting eosinophils has taught us about their role in diseases.Asthmatic airway epithelial cells differentially regulate fibroblast expression of extracellular matrix components.Asthma and pulmonary arterial hypertension: do they share a key mechanism of pathogenesis?Protein and microRNA biomarkers from lavage, urine, and serum in military personnel evaluated for dyspnea.Allergen-induced coexpression of bFGF and TGF-β1 by macrophages in a mouse model of airway remodeling: bFGF induces macrophage TGF-β1 expression in vitro.Mega-dose vitamin C attenuated lung inflammation in mouse asthma model.The irreversible component of persistent asthma.Inhibiting AKT phosphorylation employing non-cytotoxic anthraquinones ameliorates TH2 mediated allergic airways disease and rhinovirus exacerbation.Prostaglandin I₂promotes the development of IL-17-producing γδ T cells that associate with the epithelium during allergic lung inflammation.Airway remodeling in murine asthma correlates with a defect in PGE2 synthesis by lung fibroblasts.Dual Transcriptomic Profiling of Host and Microbiota during Health and Disease in Pediatric Asthma Innate lymphoid cells promote lung-tissue homeostasis after infection with influenza virus.Pediatric severe asthma with fungal sensitization is mediated by steroid-resistant IL-33 Innate lymphoid cells: critical regulators of allergic inflammation and tissue repair in the lung.Vitamin D and asthma.Cbl-b Deficiency in Mice Results in Exacerbation of Acute and Chronic Stages of Allergic Asthma.Effect of P2X4R on airway inflammation and airway remodeling in allergic airway challenge in mice.Correlation of hyaluronan deposition with infiltration of eosinophils and lymphocytes in a cockroach-induced murine model of asthma.Recurring BALB/c mouse lung inflammatory responses to episodic allergen exposure Assessment of murine lung mechanics outcome measures: alignment with those made in asthmatics.Fstl1 Promotes Asthmatic Airway Remodeling by Inducing Oncostatin M.Diagnostic, functional, and therapeutic roles of microRNA in allergic diseases.PI3K gamma-deficient mice have reduced levels of allergen-induced eosinophilic inflammation and airway remodeling.CD4+ cells are required for chronic eosinophilic lung inflammation but not airway remodeling.Immunization against TGF-β1 reduces collagen deposition but increases sustained inflammation in a murine asthma model Inhaled corticosteroids do not reduce initial high activity of matrix metalloproteinase (MMP)-9 in exhaled breath condensates of children with asthma exacerbation: a proof of concept study Relationships between eosinophilic inflammation, tissue remodeling, and fibrosis in eosinophilic esophagitis.Advances in mechanisms of asthma, allergy, and immunology in 2008.Investigation of the anti‑asthmatic activity of Oridonin on a mouse model of asthma.Antileukotriene reverts the early effects of inflammatory response of distal parenchyma in experimental chronic allergic inflammation.Bioaerosols from a food waste composting plant affect human airway epithelial cell remodeling genes.Epoxyeicosatrienoic acids are involved in the C(70) fullerene derivative-induced control of allergic asthma.

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Immunologic and inflammatory mechanisms that drive asthma progression to remodeling

http://www.wikidata.org/entity/Q37105324

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article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on March 2008

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Immunologic and inflammatory mechanisms that drive asthma progression to remodeling

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Immunologic and inflammatory mechanisms that drive asthma progression to remodeling.

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Immunologic and inflammatory mechanisms that drive asthma progression to remodeling

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Immunologic and inflammatory mechanisms that drive asthma progression to remodeling.

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Immunologic and inflammatory mechanisms that drive asthma progression to remodeling

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Immunologic and inflammatory mechanisms that drive asthma progression to remodeling.

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J.JACI.2008.01.031

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The Journal of Allergy and Clinical Immunology

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Immunologic and inflammatory mechanisms that drive asthma progression to remodeling

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David H Broide

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P2860

Siglec-8. A novel eosinophil-specific member of the immunoglobulin superfamily

Interleukin 25 regulates type 2 cytokine-dependent immunity and limits chronic inflammation in the gastrointestinal tract

Defining the in vivo function of Siglec-F, a CD33-related Siglec expressed on mouse eosinophils

Ligation of Siglec-8: a selective mechanism for induction of human eosinophil apoptosis

Association of the ADAM33 gene with asthma and bronchial hyperresponsiveness

Mouse Siglec-F and human Siglec-8 are functionally convergent paralogs that are selectively expressed on eosinophils and recognize 6'-sulfo-sialyl Lewis X as a preferred glycan ligand

Increased expression of ADAM33 and ADAM8 with disease progression in asthma

Siglecs and their roles in the immune system

Glycan array screening reveals a candidate ligand for Siglec-8

Inhaled corticosteroids decrease subepithelial collagen deposition by modulation of the balance between matrix metalloproteinase-9 and tissue inhibitor of metalloproteinase-1 expression in asthma

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560-70; quiz 571-2

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