Disrupted female reproductive physiology following neonatal exposure to phytoestrogens or estrogen specific ligands is associated with decreased GnRH activation and kisspeptin fiber density in the hypothalamus.

Disrupted female reproductive physiology following neonatal exposure to phytoestrogens or estrogen specific ligands is associated with decreased GnRH activation and kisspeptin fiber density in the hypothalamus.

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http://www.wikidata.org/entity/Q37108482

Q37108482

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Long-term effects of environmental endocrine disruptors on reproductive physiology and behavior Neonatal bisphenol-a exposure alters rat reproductive development and ovarian morphology without impairing activation of gonadotropin-releasing hormone neurons The Deep Correlation between Energy Metabolism and Reproduction: A View on the Effects of Nutrition for Women Fertility Reproductive consequences of developmental phytoestrogen exposure Epigenetic effects of endocrine-disrupting chemicals on female reproduction: an ovarian perspective The pros and cons of phytoestrogens Soy but not bisphenol A (BPA) induces hallmarks of polycystic ovary syndrome (PCOS) and related metabolic co-morbidities in rats Neuroendocrine targets of endocrine disruptors Sexual differentiation and development of forebrain reproductive circuits Neonatal exposure to 17α-ethinyl estradiol affects kisspeptin expression and LH-surge level in female rats.BAX-dependent and BAX-independent regulation of Kiss1 neuron development in mice The impact of neonatal bisphenol-A exposure on sexually dimorphic hypothalamic nuclei in the female rat.Neonatal exposure to genistein adversely impacts the ontogeny of hypothalamic kisspeptin signaling pathways and ovarian development in the peripubertal female rat Endocrine disruption of brain sexual differentiation by developmental PCB exposure Gonadal and nongonadal regulation of sex differences in hypothalamic Kiss1 neurones The development of kisspeptin circuits in the Mammalian brain.Systemic compensatory response to neonatal estradiol exposure does not prevent depletion of the oocyte pool in the rat.Circulating levels of genistein in the neonate, apart from dose and route, predict future adverse female reproductive outcomes Early life exposure to endocrine-disrupting chemicals causes lifelong molecular reprogramming of the hypothalamus and premature reproductive aging.Endocrine disrupters: a review of some sources, effects, and mechanisms of actions on behaviour and neuroendocrine systems.Neonatal Bisphenol A exposure alters sexually dimorphic gene expression in the postnatal rat hypothalamus.Influence of ERβ selective agonism during the neonatal period on the sexual differentiation of the rat hypothalamic-pituitary-gonadal (HPG) axis.Assessment of epigenetic contributions to sexually-dimorphic Kiss1 expression in the anteroventral periventricular nucleus of mice Disrupted organization of RFamide pathways in the hypothalamus is associated with advanced puberty in female rats neonatally exposed to bisphenol A.Effects of phytoestrogen on sexual development.Early-life soy exposure and age at menarche Effect of Soyabean Isoflavones Exposure on Onset of Puberty, Serum Hormone Concentration and Gene Expression in Hypothalamus, Pituitary Gland and Ovary of Female Bama Miniature Pigs.Soy but not bisphenol A (BPA) or the phytoestrogen genistin alters developmental weight gain and food intake in pregnant rats and their offspring.Development and Aging of the Kisspeptin-GPR54 System in the Mammalian Brain: What are the Impacts on Female Reproductive Function?Impact of neonatal exposure to the ERalpha agonist PPT, bisphenol-A or phytoestrogens on hypothalamic kisspeptin fiber density in male and female rats Emerging concepts on the epigenetic and transcriptional regulation of the Kiss1 gene.Neonatal agonism of ERalpha masculinizes serotonergic (5-HT) projections to the female rat ventromedial nucleus of the hypothalamus (VMN) but does not impair lordosis.Developmental profile and sexually dimorphic expression of kiss1 and kiss1r in the fetal mouse brain.Human infertility: are endocrine disruptors to blame?Sexually dimorphic expression of hypothalamic estrogen receptors α and β and Kiss1 in neonatal male and female rats.Neuropeptides and enzymes are targets for the action of endocrine disrupting chemicals in the vertebrate brain.Metabolism and health effects of phyto-estrogens.Endocrine disruption by dietary phyto-oestrogens: impact on dimorphic sexual systems and behaviours.Exposure to phytoestrogens in utero and age at menarche in a contemporary British cohort.Soy-based Infant Formula Feeding and Heavy Menstrual Bleeding Among Young African American Women.

P2860

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P2860

Disrupted female reproductive physiology following neonatal exposure to phytoestrogens or estrogen specific ligands is associated with decreased GnRH activation and kisspeptin fiber density in the hypothalamus.

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http://www.wikidata.org/entity/Q37108482

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article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 06 July 2008

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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