Inflammatory cytokines in the pathophysiology of hypertension during preeclampsia.
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Cytokines as key players in the pathophysiology of preeclampsiaHypertensive disorders of pregnancyMolecular and vascular targets in the pathogenesis and management of the hypertension associated with preeclampsia.Altered matrix metalloproteinase-2 and -9 expression/activity links placental ischemia and anti-angiogenic sFlt-1 to uteroplacental and vascular remodeling and collagen deposition in hypertensive pregnancy.Recombinant vascular endothelial growth factor 121 infusion lowers blood pressure and improves renal function in rats with placentalischemia-induced hypertension.Polymorphisms in inflammatory genes are associated with term small for gestational age and preeclampsia.Tissue transglutaminase contributes to the pathogenesis of preeclampsia and stabilizes placental angiotensin receptor type 1 by ubiquitination-preventing isopeptide modification.Vascular and cellular calcium in normal and hypertensive pregnancyRisk factors and mediators of the vascular dysfunction associated with hypertension in pregnancy.Circulating and Vascular Bioactive Factors during Hypertension in Pregnancy.Hypertension, inflammation and T lymphocytes are increased in a rat model of HELLP syndromeA common polymorphism in NR1H2 (LXRbeta) is associated with preeclampsia.Interleukin-1β-31C/T and -511T/C polymorphisms were associated with preeclampsia in Chinese Han populationThe possible role of extravillous trophoblast-derived exosomes on the uterine spiral arterial remodeling under both normal and pathological conditionsRegulation of sFlt-1 and VEGF secretion by adenosine under hypoxic conditions in rat placental villous explants.Maternal serum levels of TNF-alpha and IL-6 long after delivery in preeclamptic and normotensive pregnant womenCD4(+) T Cells Play a Critical Role in Mediating Hypertension in Response to Placental Ischemia.Vitamin D Supplementation Suppresses Hypoxia-Stimulated Placental Cytokine Secretion, Hypertension and CD4(+) T Cell Stimulation in Response to Placental Ischemia.Human infectious diseases and risk of preeclampsia: an updated review of the literature.Increased expression of matrix metalloproteinase-1 in systemic vessels of preeclamptic women: a critical mediator of vascular dysfunction.Hypertension in response to placental ischemia during pregnancy: role of B lymphocytes.Prenatal stress and developmental programming of human health and disease risk: concepts and integration of empirical findingsMesenchymal stem cells ameliorate Th1-induced pre-eclampsia-like symptoms in mice via the suppression of TNF-α expression.CD4+ T-helper cells stimulated in response to placental ischemia mediate hypertension during pregnancyThe genetics of pre-eclampsia and other hypertensive disorders of pregnancy17-hydroxyprogesterone caproate significantly improves clinical characteristics of preeclampsia in the reduced uterine perfusion pressure rat modelUnravelling the theories of pre-eclampsia: are the protective pathways the new paradigm?Placental expression of miR-517a/b and miR-517c contributes to trophoblast dysfunction and preeclampsiaSingle administration of ultra-low-dose lipopolysaccharide in rat early pregnancy induces TLR4 activation in the placenta contributing to preeclampsia.Impact of obesity on childhood kidneyPrevention of Defective Placentation and Pregnancy Loss by Blocking Innate Immune Pathways in a Syngeneic Model of Placental Insufficiency.Expression profile of microRNAs and mRNAs in human placentas from pregnancies complicated by preeclampsia and preterm labor.Fetal programming of body composition, obesity, and metabolic function: the role of intrauterine stress and stress biology.Moderate Exercise Attenuates Lipopolysaccharide-Induced Inflammation and Associated Maternal and Fetal Morbidities in Pregnant Rats.Autoantibody-mediated angiotensin receptor activation contributes to preeclampsia through tumor necrosis factor-alpha signaling.Hypertension in response to CD4(+) T cells from reduced uterine perfusion pregnant rats is associated with activation of the endothelin-1 system.A Common Profile of Disordered Angiogenic Factor Production and the Exacerbation of Inflammation in Early Preeclampsia, Late Preeclampsia, and Intrauterine Growth RestrictionPlacental secretion of interleukin-1 and interleukin-1 receptor antagonist in preeclampsia: effect of magnesium sulfate.Timing of ischemic insult alters fetal growth trajectory, maternal angiogenic balance, and markers of renal oxidative stress in the pregnant rat.Control of soluble fms-like tyrosine-1 (sFlt-1) production response to placental ischemia/hypoxia: role of tumor necrosis factor-α.
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Inflammatory cytokines in the pathophysiology of hypertension during preeclampsia.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on December 2007
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
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vědecký článek
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name
Inflammatory cytokines in the pathophysiology of hypertension during preeclampsia.
@en
Inflammatory cytokines in the pathophysiology of hypertension during preeclampsia.
@nl
type
label
Inflammatory cytokines in the pathophysiology of hypertension during preeclampsia.
@en
Inflammatory cytokines in the pathophysiology of hypertension during preeclampsia.
@nl
prefLabel
Inflammatory cytokines in the pathophysiology of hypertension during preeclampsia.
@en
Inflammatory cytokines in the pathophysiology of hypertension during preeclampsia.
@nl
P2093
P921
P1476
Inflammatory cytokines in the pathophysiology of hypertension during preeclampsia.
@en
P2093
Babbette D LaMarca
Jeffrey S Gilbert
Joey P Granger
Michael J Ryan
Sydney R Murphy
P2860
P2888
P304
P356
10.1007/S11906-007-0088-1
P577
2007-12-01T00:00:00Z