Intrameal hepatic portal and intraperitoneal infusions of glucagon-like peptide-1 reduce spontaneous meal size in the rat via different mechanisms.
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Long-acting glucagon-like peptide-1 receptor agonists have direct access to and effects on pro-opiomelanocortin/cocaine- and amphetamine-stimulated transcript neurons in the mouse hypothalamusDo glucagonomas always produce glucagon?The L-Cell in Nutritional Sensing and the Regulation of AppetiteCentral nervous system mechanisms linking the consumption of palatable high-fat diets to the defense of greater adiposityGLP-1, the gut-brain, and brain-periphery axesTherapy for obesity based on gastrointestinal hormonesEffects of GLP-1 on appetite and weightPhysiology of proglucagon peptides: role of glucagon and GLP-1 in health and diseaseNutritional modulation of endogenous glucagon-like peptide-1 secretion: a reviewMicrobial regulation of GLP-1 and L-cell biologyGut-brain connection: The neuroprotective effects of the anti-diabetic drug liraglutideGlucagon-like peptide 1 interacts with ghrelin and leptin to regulate glucose metabolism and food intake through vagal afferent neuron signalingPeripheral signals mediate the beneficial effects of gastric surgery in obesityThe glucagon-like peptide 1 analogue, exendin-4, attenuates the rewarding properties of psychostimulant drugs in miceThe arcuate nucleus mediates GLP-1 receptor agonist liraglutide-dependent weight loss.Endogenous leptin signaling in the caudal nucleus tractus solitarius and area postrema is required for energy balance regulation.Post-oral infusion sites that support glucose-conditioned flavor preferences in rats.Meal-induced hormone responses in a rat model of Roux-en-Y gastric bypass surgery.Gut hormones such as amylin and GLP-1 in the control of eating and energy expenditure.Role of capsaicin-sensitive peripheral sensory neurons in anorexic responses to intravenous infusions of cholecystokinin, peptide YY-(3-36), and glucagon-like peptide-1 in ratsVagal afferent mediates the anorectic effect of peripheral secretin.Possible role of intestinal fatty acid oxidation in the eating-inhibitory effect of the PPAR-α agonist Wy-14643 in high-fat diet fed rats.Intracellular signals mediating the food intake-suppressive effects of hindbrain glucagon-like peptide-1 receptor activationThe glucagon-like peptide 1 analogue Exendin-4 attenuates the nicotine-induced locomotor stimulation, accumbal dopamine release, conditioned place preference as well as the expression of locomotor sensitization in mice.Obesity surgery and gut-brain communication.Liraglutide, leptin and their combined effects on feeding: additive intake reduction through common intracellular signalling mechanisms.Peripheral and central GLP-1 receptor populations mediate the anorectic effects of peripherally administered GLP-1 receptor agonists, liraglutide and exendin-4.Abdominal surgery inhibits circulating acyl ghrelin and ghrelin-O-acyltransferase levels in rats: role of the somatostatin receptor subtype 2.Mechanisms responsible for excess weight loss after bariatric surgeryThe gut sensor as regulator of body weight.The common hepatic branch of the vagus is not required to mediate the glycemic and food intake suppressive effects of glucagon-like-peptide-1.Jejunal linoleic acid infusions require GLP-1 receptor signaling to inhibit food intake: implications for the effectiveness of Roux-en-Y gastric bypass.Glucagon-like peptide-1 receptor agonists suppress water intake independent of effects on food intake.Hippocampal GLP-1 receptors influence food intake, meal size, and effort-based responding for food through volume transmissionVagal innervation of the hepatic portal vein and liver is not necessary for Roux-en-Y gastric bypass surgery-induced hypophagia, weight loss, and hypermetabolism.GLP-1 neurons in the nucleus of the solitary tract project directly to the ventral tegmental area and nucleus accumbens to control for food intake.Regulation of energy balance by a gut-brain axis and involvement of the gut microbiota.CCK Response Deficiency in Synphilin-1 Transgenic Mice.Ghrelin signaling is not essential for sugar or fat conditioned flavor preferences in mice.Circulating GLP-1 and CCK-8 reduce food intake by capsaicin-insensitive, nonvagal mechanisms.
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P2860
Intrameal hepatic portal and intraperitoneal infusions of glucagon-like peptide-1 reduce spontaneous meal size in the rat via different mechanisms.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
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scientific article published on 23 October 2008
@en
vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Intrameal hepatic portal and i ...... rat via different mechanisms.
@en
Intrameal hepatic portal and i ...... rat via different mechanisms.
@nl
type
label
Intrameal hepatic portal and i ...... rat via different mechanisms.
@en
Intrameal hepatic portal and i ...... rat via different mechanisms.
@nl
prefLabel
Intrameal hepatic portal and i ...... rat via different mechanisms.
@en
Intrameal hepatic portal and i ...... rat via different mechanisms.
@nl
P2093
P2860
P356
P1433
P1476
Intrameal hepatic portal and i ...... rat via different mechanisms.
@en
P2093
Elisabeth B Rüttimann
Jacquelien J Hillebrand
Myrtha Arnold
Nori Geary
P2860
P304
P356
10.1210/EN.2008-1221
P407
P577
2008-10-23T00:00:00Z