CCK Response Deficiency in Synphilin-1 Transgenic Mice.

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CCK Response Deficiency in Synphilin-1 Transgenic Mice.

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2015 nî lūn-bûn

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2015年の論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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CCK Response Deficiency in Synphilin-1 Transgenic Mice.

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CCK Response Deficiency in Synphilin-1 Transgenic Mice.

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label

CCK Response Deficiency in Synphilin-1 Transgenic Mice.

@ast

CCK Response Deficiency in Synphilin-1 Transgenic Mice.

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CCK Response Deficiency in Synphilin-1 Transgenic Mice.

@ast

CCK Response Deficiency in Synphilin-1 Transgenic Mice.

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P1476

CCK Response Deficiency in Synphilin-1 Transgenic Mice.

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P2093

Alexander Moghadam

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Dejun Yang

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Megan Smith

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Pique P Choi

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Tianxia Li

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Timothy H Moran

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Wanli W Smith

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P2860

Synphilin-1 associates with alpha-synuclein and promotes the formation of cytosolic inclusions

Parkin ubiquitinates the alpha-synuclein-interacting protein, synphilin-1: implications for Lewy-body formation in Parkinson disease

Synphilin-1 is developmentally localized to synaptic terminals, and its association with synaptic vesicles is modulated by alpha-synuclein

Synphilin-1 binds ATP and regulates intracellular energy status

The arcuate nucleus mediates GLP-1 receptor agonist liraglutide-dependent weight loss.

Synphilin-1 attenuates neuronal degeneration in the A53T alpha-synuclein transgenic mouse model

Amylin activates distributed CNS nuclei to control energy balance.

The direct and indirect controls of meal size.

Amylin, food intake, and obesity.

Peripheral and central GLP-1 receptor populations mediate the anorectic effects of peripherally administered GLP-1 receptor agonists, liraglutide and exendin-4.

P304

e0142314

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P31

scholarly article

P356

10.1371/JOURNAL.PONE.0142314

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P407

English

P433

http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

P577

2015-11-16T00:00:00Z

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P5875

284021352

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P698

26569394

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P932

4646498

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