The hypoxic cancer secretome induces pre-metastatic bone lesions through lysyl oxidase. - Wikidata - awgldk - TriplyDB

The hypoxic cancer secretome induces pre-metastatic bone lesions through lysyl oxidase.

The hypoxic cancer secretome induces pre-metastatic bone lesions through lysyl oxidase.

http://www.wikidata.org/entity/Q37126990

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Intravital imaging reveals new ancillary mechanisms co-opted by cancer cells to drive tumor progression Hypoxic control of metastasis A hypoxic ticket to the bone metastatic niche Tumor-associated Endo180 requires stromal-derived LOX to promote metastatic prostate cancer cell migration on human ECM surfaces.Medicine in the early twenty-first century: paradigm and anticipation - EPMA position paper 2016 Cell and Signal Components of the Microenvironment of Bone Metastasis Are Affected by Hypoxia Tumor cell dormancy Metastatic colonization by circulating tumour cells Implications of Hypoxia in Breast Cancer Metastasis to Bone The ever-expanding role of HIF in tumour and stromal biology The role of lysyl oxidase, the extracellular matrix and the pre-metastatic niche in bone metastasis Mitochondrial Akt Regulation of Hypoxic Tumor Reprogramming Breast cancer epidemic in the early twenty-first century: evaluation of risk factors, cumulative questionnaires and recommendations for preventive measures.Multi-Channel Optical Coherence Elastography Using Relative and Absolute Shear-Wave Time of Flight Integration of Breast Cancer Secretomes with Clinical Data Elucidates Potential Serum Markers for Disease Detection, Diagnosis, and Prognosis.Lysyl oxidase drives tumour progression by trapping EGF receptors at the cell surface.Roles of Copper-Binding Proteins in Breast Cancer.Blocking Surgically Induced Lysyl Oxidase Activity Reduces the Risk of Lung Metastases.Pre-clinical evaluation of small molecule LOXL2 inhibitors in breast cancer.Stromal PDGFR-α Activation Enhances Matrix Stiffness, Impedes Mammary Ductal Development, and Accelerates Tumor Growth.Structural ECM components in the premetastatic and metastatic niche.High Lysyl Oxidase (LOX) in the Non-Malignant Prostate Epithelium Predicts a Poor Outcome in Prostate Cancer Patient Managed by Watchful Waiting.Collaborative cross mice in a genetic association study reveal new candidate genes for bone microarchitecture.Fibromodulin Interacts with Collagen Cross-linking Sites and Activates Lysyl Oxidase.Cooperative Effect of miR-141-3p and miR-145-5p in the Regulation of Targets in Clear Cell Renal Cell Carcinoma.The unresolved role of systemic factors in bone metastasis.Rank ligand as a target in musculoskeletal neoplasms.Bone metastasis and the metastatic niche.Secretome identification of immune cell factors mediating metastatic cell homing.High SPARC Expression Starting from Dysplasia, Associated with Breast Carcinoma, Is Predictive for Bone Metastasis without Enhancement of Plasma Levels Forcing through Tumor Metastasis: The Interplay between Tissue Rigidity and Epithelial-Mesenchymal Transition.Functional osteoclastogenesis: the baseline variability in blood donor precursors is not associated with age and gender.Organotropic metastasis: role of tumor exosomes.Extracellular matrix mediators of metastatic cell colonization characterized using scaffold mimics of the pre-metastatic niche Tumour exosome integrins determine organotropic metastasis.Tumor-associated macrophages: unwitting accomplices in breast cancer malignancy.Emerging therapeutic targets in metastatic progression: A focus on breast cancer.An Arntl2-Driven Secretome Enables Lung Adenocarcinoma Metastatic Self-Sufficiency Heparanase 2 Attenuates Head and Neck Tumor Vascularity and Growth.Enzymatic and non-enzymatic functions of the lysyl oxidase family in bone

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The hypoxic cancer secretome induces pre-metastatic bone lesions through lysyl oxidase.

http://www.wikidata.org/entity/Q37126990

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

@tr

scientific article published on 27 May 2015

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

The hypoxic cancer secretome induces pre-metastatic bone lesions through lysyl oxidase.

@en

The hypoxic cancer secretome induces pre-metastatic bone lesions through lysyl oxidase.

@nl

type

label

The hypoxic cancer secretome induces pre-metastatic bone lesions through lysyl oxidase.

@en

The hypoxic cancer secretome induces pre-metastatic bone lesions through lysyl oxidase.

@nl

prefLabel

The hypoxic cancer secretome induces pre-metastatic bone lesions through lysyl oxidase.

@en

The hypoxic cancer secretome induces pre-metastatic bone lesions through lysyl oxidase.

@nl

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The hypoxic cancer secretome induces pre-metastatic bone lesions through lysyl oxidase

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Demelza Bird

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Erwin M Schoof

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Georgina Lang

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Hamish Forrest

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Holly R Evans

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Lara Perryman

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Norain Ab Latif

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Rune Linding

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P2860

VEGFR1-positive haematopoietic bone marrow progenitors initiate the pre-metastatic niche

Hypoxia-induced lysyl oxidase is a critical mediator of bone marrow cell recruitment to form the premetastatic niche

Gene expression programs in response to hypoxia: cell type specificity and prognostic significance in human cancers

T cells induce pre-metastatic osteolytic disease and help bone metastases establishment in a mouse model of metastatic breast cancer

A gene-expression signature as a predictor of survival in breast cancer

Expression of a P2X7 receptor by a subpopulation of human osteoblasts

Lysyl oxidase is essential for hypoxia-induced metastasis

Tumour-mediated upregulation of chemoattractants and recruitment of myeloid cells predetermines lung metastasis

Critical role for lysyl oxidase in mesenchymal stem cell-driven breast cancer malignancy

The role of lysyl oxidase in SRC-dependent proliferation and metastasis of colorectal cancer

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106-110

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10.1038/NATURE14492

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7554

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522

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Alison Gartland

Janine T. Erler

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2015-05-27T00:00:00Z

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