Intermittent PTH stimulates periosteal bone formation by actions on post-mitotic preosteoblasts
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The role of estrogen and androgen receptors in bone health and diseaseDysapoptosis of osteoblasts and osteocytes increases cancellous bone formation but exaggerates cortical porosity with age.FoxO-mediated defense against oxidative stress in osteoblasts is indispensable for skeletal homeostasis in miceMolecular biology of bone remodeling: implications for new therapeutic targets for osteoporosis.Amelioration of type I diabetes-induced osteoporosis by parathyroid hormone is associated with improved osteoblast survivalGenetic and transcriptional control of bone formationThe effects of PTH, loading and surgical insult on cancellous bone at the bone-implant interface in the rabbit.Role of parathyroid hormone therapy in reversing radiation-induced nonunion and normalization of radiomorphometrics in a murine mandibular model of distraction osteogenesis.Decreased oxidative stress and greater bone anabolism in the aged, when compared to the young, murine skeleton with parathyroid hormone administration.Intermittent PTHrP(1-34) exposure augments chondrogenesis and reduces hypertrophy of mesenchymal stromal cells.PTH1-34 blocks radiation-induced osteoblast apoptosis by enhancing DNA repair through canonical Wnt pathwayAnabolic and catabolic regimens of human parathyroid hormone 1-34 elicit bone- and envelope-specific attenuation of skeletal effects in Sost-deficient mice.Teriparatide Therapy as an Adjuvant for Tissue Engineering and Integration of Biomaterials.Gsα enhances commitment of mesenchymal progenitors to the osteoblast lineage but restrains osteoblast differentiation in mice.Continuous elevation of PTH increases the number of osteoblasts via both osteoclast-dependent and -independent mechanisms.PTH receptor signaling in osteocytes governs periosteal bone formation and intracortical remodeling.Parathyroid hormone therapy mollifies radiation-induced biomechanical degradation in murine distraction osteogenesis.Parathyroid hormone regulates the distribution and osteoclastogenic potential of hematopoietic progenitors in the bone marrowBone mechanotransduction may require augmentation in order to strengthen the senescent skeleton.Systemic intermittent parathyroid hormone treatment improves osseointegration of press-fit inserted implants in cancellous boneIntermittent parathyroid hormone administration converts quiescent lining cells to active osteoblasts.Loss of Gsα in the Postnatal Skeleton Leads to Low Bone Mass and a Blunted Response to Anabolic Parathyroid Hormone TherapyAbility of circulating human hematopoietic lineage negative cells to support hematopoiesis.Teriparatide vs. alendronate as a treatment for osteoporosis: changes in biochemical markers of bone turnover, BMD and quality of lifeLong-term safety, efficacy, and patient acceptability of teriparatide in the management of glucocorticoid-induced osteoporosis.Mesenchymal progenitors residing close to the bone surface are functionally distinct from those in the central bone marrow.Bone fragility and decline in stem cells in prematurely aging DNA repair deficient trichothiodystrophy miceCell cycle and apoptosis regulatory protein (CARP)-1 is expressed in osteoblasts and regulated by PTHParathyroid hormone reverses radiation induced hypovascularity in a murine model of distraction osteogenesis.Commitment to the osteoblast lineage is not required for RANKL gene expression.Successful treatment of nonunion with teriparatide after failed ankle arthrodesis for Charcot arthropathyThe relevance of mouse models for investigating age-related bone loss in humans.Resorption controls bone anabolism driven by parathyroid hormone (PTH) receptor signaling in osteocytesManagement of glucocorticoids-induced osteoporosis: role of teriparatide.Osteosclerosis in two brothers with autosomal dominant pseudohypoparathyroidism type 1b: bone histomorphometric analysis.Adenylyl cyclase 6 mediates loading-induced bone adaptation in vivo.Parathyroid hormone analogues in the treatment of osteoporosis.Role of FGFs/FGFRs in skeletal development and bone regeneration.Gastric acid, calcium absorption, and their impact on bone health.Physiological Bone Remodeling: Systemic Regulation and Growth Factor Involvement.
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Intermittent PTH stimulates periosteal bone formation by actions on post-mitotic preosteoblasts
description
article científic
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article scientifique
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articolo scientifico
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artigo científico
@pt
bilimsel makale
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scientific article published on 22 October 2008
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Intermittent PTH stimulates pe ...... on post-mitotic preosteoblasts
@en
Intermittent PTH stimulates pe ...... n post-mitotic preosteoblasts.
@nl
type
label
Intermittent PTH stimulates pe ...... on post-mitotic preosteoblasts
@en
Intermittent PTH stimulates pe ...... n post-mitotic preosteoblasts.
@nl
prefLabel
Intermittent PTH stimulates pe ...... on post-mitotic preosteoblasts
@en
Intermittent PTH stimulates pe ...... n post-mitotic preosteoblasts.
@nl
P2093
P2860
P1433
P1476
Intermittent PTH stimulates pe ...... on post-mitotic preosteoblasts
@en
P2093
A Afshan Ali
Charles A O'Brien
Paula K Roberson
Robert L Jilka
Robert S Weinstein
Stavros C Manolagas
P2860
P304
P356
10.1016/J.BONE.2008.10.037
P577
2008-10-22T00:00:00Z