Commitment to the osteoblast lineage is not required for RANKL gene expression. - Wikidata - awgldk - TriplyDB

Commitment to the osteoblast lineage is not required for RANKL gene expression.

Commitment to the osteoblast lineage is not required for RANKL gene expression.

http://www.wikidata.org/entity/Q37176364

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Control of RANKL gene expression The Modulatory Effects of Mesenchymal Stem Cells on Osteoclastogenesis The osteocyte: key player in regulating bone turnover Matrix-embedded cells control osteoclast formation Identification of the GATA factor TRPS1 as a repressor of the osteocalcin promoter.Disruption of PTH receptor 1 in T cells protects against PTH-induced bone loss Direct recruitment of polycomb repressive complex 1 to chromatin by core binding transcription factors.Decreased oxidative stress and greater bone anabolism in the aged, when compared to the young, murine skeleton with parathyroid hormone administration.Identification of a subpopulation of marrow MSC-derived medullary adipocytes that express osteoclast-regulating molecules: marrow adipocytes express osteoclast mediators.Physiological functions of osteoblast lineage and T cell-derived RANKL in bone homeostasis.Osteocyte RANKL: new insights into the control of bone remodeling MKP-1 is essential for canonical vitamin D-induced signaling through nuclear import and regulates RANKL expression and function.Osteocyte control of osteoclastogenesis.Osteocyte control of bone remodeling: is sclerostin a key molecular coordinator of the balanced bone resorption-formation cycles?The effects of tumour necrosis factor-α on bone cells involved in periodontal alveolar bone loss; osteoclasts, osteoblasts and osteocytes.Murine Rankl-/- Mesenchymal Stromal Cells Display an Osteogenic Differentiation Defect Improved by a RANKL-Expressing Lentiviral Vector.Serotonin: a novel bone mass controller may have implications for alveolar bone Osteoimmunology: The Conceptual Framework Unifying the Immune and Skeletal Systems.Parathyroid Hormone Signaling in Osteocytes

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Commitment to the osteoblast lineage is not required for RANKL gene expression.

http://www.wikidata.org/entity/Q37176364

description

article científic

@ca

article scientifique

@fr

articolo scientifico

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artigo científico

@pt

bilimsel makale

@tr

scientific article published on 11 March 2009

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Commitment to the osteoblast lineage is not required for RANKL gene expression.

@en

Commitment to the osteoblast lineage is not required for RANKL gene expression.

@nl

type

label

Commitment to the osteoblast lineage is not required for RANKL gene expression.

@en

Commitment to the osteoblast lineage is not required for RANKL gene expression.

@nl

prefLabel

Commitment to the osteoblast lineage is not required for RANKL gene expression.

@en

Commitment to the osteoblast lineage is not required for RANKL gene expression.

@nl

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Journal of Biological Chemistry

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Commitment to the osteoblast lineage is not required for RANKL gene expression.

@en

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Bjorn R Olsen

http://www.w3.org/2001/XMLSchema#string

Carlo Galli

http://www.w3.org/2001/XMLSchema#string

Charles A O'Brien

http://www.w3.org/2001/XMLSchema#string

Qiang Fu

http://www.w3.org/2001/XMLSchema#string

Robert L Jilka

http://www.w3.org/2001/XMLSchema#string

Stavros C Manolagas

http://www.w3.org/2001/XMLSchema#string

Wenfang Wang

http://www.w3.org/2001/XMLSchema#string

P2860

Birth and Death of Bone Cells: Basic Regulatory Mechanisms and Implications for the Pathogenesis and Treatment of Osteoporosis 1

Osteoprotegerin ligand is a cytokine that regulates osteoclast differentiation and activation

Osteoclast differentiation factor is a ligand for osteoprotegerin/osteoclastogenesis-inhibitory factor and is identical to TRANCE/RANKL

Cbfa1, a candidate gene for cleidocranial dysplasia syndrome, is essential for osteoblast differentiation and bone development

Osteoimmunology: interplay between the immune system and bone metabolism

Dimerization with PEBP2beta protects RUNX1/AML1 from ubiquitin-proteasome-mediated degradation

Cbfbeta interacts with Runx2 and has a critical role in bone development

OPGL is a key regulator of osteoclastogenesis, lymphocyte development and lymph-node organogenesis

The core-binding factor beta subunit is required for bone formation and hematopoietic maturation

Targeted disruption of Cbfa1 results in a complete lack of bone formation owing to maturational arrest of osteoblasts

P304

12654-12662

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scholarly article

P356

10.1074/JBC.M806628200

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19

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284

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2009-03-11T00:00:00Z

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19279010

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2675994

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