Frequent PTEN genomic alterations and activated phosphatidylinositol 3-kinase pathway in basal-like breast cancer cells. - Wikidata - awgldk - TriplyDB

Frequent PTEN genomic alterations and activated phosphatidylinositol 3-kinase pathway in basal-like breast cancer cells.

Frequent PTEN genomic alterations and activated phosphatidylinositol 3-kinase pathway in basal-like breast cancer cells.

http://www.wikidata.org/entity/Q37131339

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PI3K/Akt/mTOR inhibitors in breast cancer Targeted Therapies in Triple-Negative Breast Cancer Identification of human triple-negative breast cancer subtypes and preclinical models for selection of targeted therapies Protein tyrosine phosphatase UBASH3B is overexpressed in triple-negative breast cancer and promotes invasion and metastasis.A phase II study of UCN-01 in combination with irinotecan in patients with metastatic triple negative breast cancer.PI3K pathway activation in high-grade ductal carcinoma in situ--implications for progression to invasive breast carcinoma.Targeting oncogenic vulnerabilities in triple negative breast cancer: biological bases and ongoing clinical studies.Asymmetric microarray data produces gene lists highly predictive of research literature on multiple cancer types.DNA methylome of familial breast cancer identifies distinct profiles defined by mutation status.EMA - A R package for Easy Microarray data analysis Patient-derived xenografts of triple-negative breast cancer reproduce molecular features of patient tumors and respond to mTOR inhibition.Realizing the promise of reverse phase protein arrays for clinical, translational, and basic research: a workshop report: the RPPA (Reverse Phase Protein Array) society.Identification and use of biomarkers in treatment strategies for triple-negative breast cancer subtypes.Potential prognostic value of heat-shock protein 90 in the presence of phosphatidylinositol-3-kinase overexpression or loss of PTEN, in invasive breast cancers.The application of gene co-expression network reconstruction based on CNVs and gene expression microarray data in breast cancer.Functional interaction between Env oncogene from Jaagsiekte sheep retrovirus and tumor suppressor Sprouty2.Genome wide proteomics of ERBB2 and EGFR and other oncogenic pathways in inflammatory breast cancer.t-DARPP regulates phosphatidylinositol-3-kinase-dependent cell growth in breast cancer.Molecular apocrine differentiation is a common feature of breast cancer in patients with germline PTEN mutations.Poly (ADP-ribose) polymerase as a novel therapeutic target in cancer.NormaCurve: a SuperCurve-based method that simultaneously quantifies and normalizes reverse phase protein array data.Claudin-low breast cancers: clinical, pathological, molecular and prognostic characterization Microarray phosphatome profiling of breast cancer patients unveils a complex phosphatase regulatory role of the MAPK and PI3K pathways in estrogen receptor-negative breast cancers Yiqi formula enhances the antitumor effects of erlotinib for treatment of triple-negative breast cancer xenografts.Minireview: Basal-like breast cancer: from molecular profiles to targeted therapies TTK/hMPS1 is an attractive therapeutic target for triple-negative breast cancer.Antagonism of EGFR and HER3 enhances the response to inhibitors of the PI3K-Akt pathway in triple-negative breast cancer.Ribavirin treatment effects on breast cancers overexpressing eIF4E, a biomarker with prognostic specificity for luminal B-type breast cancer.Pharmacological profiling of kinase dependency in cell lines across triple-negative breast cancer subtypes.A pharmacogenomic method for individualized prediction of drug sensitivity Association of high obesity with PAM50 breast cancer intrinsic subtypes and gene expression.β1 integrin mediates an alternative survival pathway in breast cancer cells resistant to lapatinib.Elevated PI3K signaling drives multiple breast cancer subtypes.Molecular basis of triple negative breast cancer and implications for therapy.MicroRNA profiling of the pubertal mouse mammary gland identifies miR-184 as a candidate breast tumour suppressor gene Triple-negative breast cancer: are we making headway at least?Phosphorylated mTOR expression correlates with poor outcome in early-stage triple negative breast carcinomas PTEN is required to maintain luminal epithelial homeostasis and integrity in the adult mammary gland Therapeutic potential of stem cells expressing suicide genes that selectively target human breast cancer cells: evidence that they exert tumoricidal effects via tumor tropism (review).Correlation of Notch1, pAKT and nuclear NF-κB expression in triple negative breast cancer.

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Frequent PTEN genomic alterations and activated phosphatidylinositol 3-kinase pathway in basal-like breast cancer cells.

http://www.wikidata.org/entity/Q37131339

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 03 December 2008

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Frequent PTEN genomic alterati ...... asal-like breast cancer cells.

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Frequent PTEN genomic alterati ...... asal-like breast cancer cells.

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type

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Frequent PTEN genomic alterati ...... asal-like breast cancer cells.

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Frequent PTEN genomic alterati ...... asal-like breast cancer cells.

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Frequent PTEN genomic alterati ...... asal-like breast cancer cells.

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Frequent PTEN genomic alterati ...... asal-like breast cancer cells.

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Breast Cancer Research

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Frequent PTEN genomic alterati ...... asal-like breast cancer cells.

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Audrey Tourdès

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Bérengère Marty

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Eléonore Gravier

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Fathia Djelti

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Francisco Cruzalegui

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Gordon C Tucker

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Guillem Rigaill

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Ingrid Lebigot

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Jean-Paul Thiery

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John A Hickman

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P2860

A transforming mutation in the pleckstrin homology domain of AKT1 in cancer

Trastuzumab after adjuvant chemotherapy in HER2-positive breast cancer.

Gene expression patterns of breast carcinomas distinguish tumor subclasses with clinical implications

Gene expression profiling predicts clinical outcome of breast cancer

Molecular portraits of human breast tumours

Repeated observation of breast tumor subtypes in independent gene expression data sets

PIK3CA mutations correlate with hormone receptors, node metastasis, and ERBB2, and are mutually exclusive with PTEN loss in human breast carcinoma

X chromosomal abnormalities in basal-like human breast cancer

The evolution of phosphatidylinositol 3-kinases as regulators of growth and metabolism

mTOR inhibition induces upstream receptor tyrosine kinase signaling and activates Akt

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scholarly article

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10.1186/BCR2204

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6

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10

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Anne Vincent-Salomon

Marc-Henri Stern

Emmanuel Barillot

Pierre Gestraud

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2008-12-03T00:00:00Z

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23570698

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