Redistribution of the Lamin B1 genomic binding profile affects rearrangement of heterochromatic domains and SAHF formation during senescence. - Wikidata - awgldk - TriplyDB

Redistribution of the Lamin B1 genomic binding profile affects rearrangement of heterochromatic domains and SAHF formation during senescence.

Redistribution of the Lamin B1 genomic binding profile affects rearrangement of heterochromatic domains and SAHF formation during senescence.

http://www.wikidata.org/entity/Q37138585

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Old cells, new tricks: chromatin structure in senescence Epigenetic modulators, modifiers and mediators in cancer aetiology and progression Common features of chromatin in aging and cancer: cause or coincidence?Cell-based screen for altered nuclear phenotypes reveals senescence progression in polyploid cells after Aurora kinase B inhibition.Reorganization of chromosome architecture in replicative cellular senescence Physiological and Pathological Aging Affects Chromatin Dynamics, Structure and Function at the Nuclear Edge Autophagy mediates degradation of nuclear lamina NOTCH1 mediates a switch between two distinct secretomes during senescence.Specifying peripheral heterochromatin during nuclear lamina reassembly.Nucleolus association of chromosomal domains is largely maintained in cellular senescence despite massive nuclear reorganisation.Nonrandom domain organization of the Arabidopsis genome at the nuclear periphery.Nuclear lamina builds tissues from the stem cell niche.Three-dimensional genome architecture: players and mechanisms.HIRA orchestrates a dynamic chromatin landscape in senescence and is required for suppression of neoplasia.Role of lamin b1 in chromatin instability Wash interacts with lamin and affects global nuclear organization Identification of lamin B-regulated chromatin regions based on chromatin landscapes.JMJD3 promotes SAHF formation in senescent WI38 cells by triggering an interplay between demethylation and phosphorylation of RB protein.A novel role for the condensin II complex in cellular senescence.Unfolding the story of chromatin organization in senescent cells.Distinct features of lamin A-interacting chromatin domains mapped by ChIP-sequencing from sonicated or micrococcal nuclease-digested chromatin Mapping H4K20me3 onto the chromatin landscape of senescent cells indicates a function in control of cell senescence and tumor suppression through preservation of genetic and epigenetic stability.Downregulation of miR-130b~301b cluster is mediated by aberrant promoter methylation and impairs cellular senescence in prostate cancer.Closing the (nuclear) envelope on the genome: how nuclear lamins interact with promoters and modulate gene expression.B-type lamins in health and disease.Cellular senescence and its effector programs.Chromatin maintenance and dynamics in senescence: a spotlight on SAHF formation and the epigenome of senescent cells.Diverse lamin-dependent mechanisms interact to control chromatin dynamics. Focus on laminopathies Nuclear lamina remodelling and its implications for human disease.The redundancy of the mammalian heterochromatic compartment.Recent advances in understanding nuclear size and shape.Autophagy-mediated degradation of nuclear envelope proteins during oncogene-induced senescence.Global reorganization of the nuclear landscape in senescent cells Epigenome rejuvenation: HP1β mobility as a measure of pluripotent and senescent chromatin ground states The Chromatin Landscape of Cellular Senescence.The molecular basis of the organization of repetitive DNA-containing constitutive heterochromatin in mammals.Senescent cells harbour features of the cancer epigenome.Mechanisms of oncogene-induced genomic instability.Messenger RNA processing is altered in autosomal dominant leukodystrophy Enriched domain detector: a program for detection of wide genomic enrichment domains robust against local variations.

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Redistribution of the Lamin B1 genomic binding profile affects rearrangement of heterochromatic domains and SAHF formation during senescence.

http://www.wikidata.org/entity/Q37138585

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article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on August 2013

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Redistribution of the Lamin B1 ...... F formation during senescence.

@en

Redistribution of the Lamin B1 ...... F formation during senescence.

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type

label

Redistribution of the Lamin B1 ...... F formation during senescence.

@en

Redistribution of the Lamin B1 ...... F formation during senescence.

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prefLabel

Redistribution of the Lamin B1 ...... F formation during senescence.

@en

Redistribution of the Lamin B1 ...... F formation during senescence.

@nl

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Genes & Development

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Redistribution of the Lamin B1 ...... F formation during senescence.

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Andrew R J Young

http://www.w3.org/2001/XMLSchema#string

Heung Chong

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Hiroshi Kimura

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Kosuke Tomimatsu

http://www.w3.org/2001/XMLSchema#string

Mahito Sadaie

http://www.w3.org/2001/XMLSchema#string

Masako Narita

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Pedro A Pérez-Mancera

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Tamir Chandra

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Thomas Carroll

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Interaction between an integral protein of the nuclear envelope inner membrane and human chromodomain proteins homologous to Drosophila HP1

Oncogenic ras provokes premature cell senescence associated with accumulation of p53 and p16INK4a

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Hutchinson-Gilford progeria mutant lamin A primarily targets human vascular cells as detected by an anti-Lamin A G608G antibody.

The p400 E1A-associated protein is a novel component of the p53 --> p21 senescence pathway

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1800-1808

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scholarly article

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10.1101/GAD.217281.113

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16

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27

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Dorothy C Bennett

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2013-08-01T00:00:00Z

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256074824

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23964094

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3759696

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