Arsenic-based antineoplastic drugs and their mechanisms of action. - Wikidata - awgldk - TriplyDB

Arsenic-based antineoplastic drugs and their mechanisms of action.

Arsenic-based antineoplastic drugs and their mechanisms of action.

http://www.wikidata.org/entity/Q37145891

about

Metalloid compounds as drugs Candidate single nucleotide polymorphism markers for arsenic responsiveness of protein targets Posttranslational modification of human glyoxalase 1 indicates redox-dependent regulation An arsenic fluorescent compound as a novel probe to study arsenic-binding proteins.Inhibitor of kappaB kinase beta regulates redox homeostasis by controlling the constitutive levels of glutathione Genetic basis of arsenite and cadmium tolerance in Saccharomyces cerevisiae.Enhancement of arsenic trioxide cytotoxicity by dietary isothiocyanates in human leukemic cells via a reactive oxygen species-dependent mechanism.Mitochondrial proteomics of nasopharyngeal carcinoma metastasis High-throughput Giardia lamblia viability assay using bioluminescent ATP content measurements Discovery of novel antigiardiasis drug candidates.Aberrantly Expressed Genes in HaCaT Keratinocytes Chronically Exposed to Arsenic Trioxide.Genomic analysis of stress response against arsenic in Caenorhabditis elegans.Synergistic Apoptosis-Inducing Antileukemic Effects of Arsenic Trioxide and Mucuna macrocarpa Stem Extract in Human Leukemic Cells via a Reactive Oxygen Species-Dependent Mechanism.Targeting metabolism with arsenic trioxide and dichloroacetate in breast cancer cells.Autophagic degradation of the BCR-ABL oncoprotein and generation of antileukemic responses by arsenic trioxide Biological responses to arsenic compounds Zinc finger proteins as templates for metal ion exchange and ligand reactivity. Chemical and biological consequences.Therapeutic and analytical applications of arsenic binding to proteins.Hitting the Bull's-Eye in Metastatic Cancers-NSAIDs Elevate ROS in Mitochondria, Inducing Malignant Cell Death.The impact of arsenic trioxide and all-trans retinoic acid on p53 R273H-codon mutant glioblastoma.Robust structure and reactivity of aqueous arsenous acid-platinum(II) anticancer complexes Rho-kinase inhibitor Y-27632 attenuates arsenic trioxide toxicity in H9c2 cardiomyoblastoma cells.Remodulation of central carbon metabolic pathway in response to arsenite exposure in Rhodococcus sp. strain NAU-1.The inositol pyrophosphate pathway in health and diseases.

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P2860

Arsenic-based antineoplastic drugs and their mechanisms of action.

http://www.wikidata.org/entity/Q37145891

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on January 2008

@en

vedecký článok

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vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Arsenic-based antineoplastic drugs and their mechanisms of action.

@en

Arsenic-based antineoplastic drugs and their mechanisms of action.

@nl

type

label

Arsenic-based antineoplastic drugs and their mechanisms of action.

@en

Arsenic-based antineoplastic drugs and their mechanisms of action.

@nl

prefLabel

Arsenic-based antineoplastic drugs and their mechanisms of action.

@en

Arsenic-based antineoplastic drugs and their mechanisms of action.

@nl

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Metal-Based Drugs

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Arsenic-based antineoplastic drugs and their mechanisms of action.

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Stephen John Ralph

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P2860

Role of critical thiol groups on the matrix surface of the adenine nucleotide translocase in the mechanism of the mitochondrial permeability transition pore

Distinct gene expression profiles in immortalized human urothelial cells exposed to inorganic arsenite and its methylated trivalent metabolites

Functional and molecular characterization of the human neutral solute channel aquaporin-9.

The dynamic dimerization of the yeast ADP/ATP carrier in the inner mitochondrial membrane is affected by conserved cysteine residues.

Peroxiredoxin III, a mitochondrion-specific peroxidase, regulates apoptotic signaling by mitochondria

Arsenic in cancer therapy

Oxidation of a critical thiol residue of the adenine nucleotide translocator enforces Bcl-2-independent permeability transition pore opening and apoptosis

Arsenic-induced PML targeting onto nuclear bodies: implications for the treatment of acute promyelocytic leukemia

Chronic exposure to methylated arsenicals stimulates arsenic excretion pathways and induces arsenic tolerance in rat liver cells

Factor B is essential for ATP synthesis by mitochondria

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260146

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scholarly article

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10.1155/2008/260146

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2008

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2008-01-01T00:00:00Z

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5422327

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18431449

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2292810

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