Concentration dependent dual effect of thrombin in endothelial cells via Par-1 and Pi3 Kinase.
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Recent Progress in Research on the Pathogenesis of Pulmonary Thromboembolism: An Old Story with New Perspectives.Thrombin and activated protein C inhibit the expression of secretory group IIA phospholipase A(2) in the TNF-alpha-activated endothelial cells by EPCR and PAR-1 dependent mechanismsFactor VIIa binding to endothelial cell protein C receptor protects vascular barrier integrity in vivo.Thrombin upregulates the angiopoietin-Tie2 Axis: endothelial protein C receptor occupancy prevents the thrombin mobilization of angiopoietin 2 and P-selectin from Weibel-Palade bodies.The occupancy of endothelial protein C receptor by its ligand modulates the par-1 dependent signaling specificity of coagulation proteasesPolyphosphate suppresses complement via the terminal pathway.Thrombin down-regulates the TGF-beta-mediated synthesis of collagen and fibronectin by human proximal tubule epithelial cells through the EPCR-dependent activation of PAR-1.The roles of thrombin and protease-activated receptors in inflammation.The endothelial cell protein C receptor: cell surface conductor of cytoprotective coagulation factor signaling.Endothelial barrier dysfunction in septic shock.Thrombomodulin/activated protein C system in septic disseminated intravascular coagulationExtracellular granzyme K mediates endothelial activation through the cleavage of protease-activated receptor-1.Thrombin-Induced Podocyte Injury Is Protease-Activated Receptor Dependent.Coagulation and non-coagulation effects of thrombin.Concentration dependent anti-inflammatory effects thrombin on polyphosphate-mediated inflammatory responses in vitro and in vivo.Homeostatic effects of coagulation protease-dependent signaling and protease activated receptors.Protease-activated receptor-1 impairs host defense in murine pneumococcal pneumonia: a controlled laboratory study.Occupancy of human EPCR by protein C induces β-arrestin-2 biased PAR1 signaling by both APC and thrombin.Unexpected anti-hypertrophic responses to low-level stimulation of protease-activated receptors in adult rat cardiomyocytes.Effects of lower concentration thrombin on high-mobility group box 1 protein-mediated inflammatory responses.Thrombin as important factor for cutaneous wound healing: comparison of fibrin biomatrices in vitro and in a rat excisional wound healing model.Nephrin as a biomarker of sickle cell glomerulopathy in Malawi.Dual inhibition of PI3K and mTOR by VS-5584 suppresses thrombus formation.Paradoxical bleeding and thrombotic episodes of dysprothrombinaemia due to a homozygous Arg382His mutation.Inhibitory effect of thrombin on the expression of secretory group IIA phospholipase A₂.
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P2860
Concentration dependent dual effect of thrombin in endothelial cells via Par-1 and Pi3 Kinase.
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article científic
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article scientifique
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articolo scientifico
@it
artigo científico
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bilimsel makale
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scientific article published on June 2009
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Concentration dependent dual e ...... ells via Par-1 and Pi3 Kinase.
@en
Concentration dependent dual e ...... ells via Par-1 and Pi3 Kinase.
@nl
type
label
Concentration dependent dual e ...... ells via Par-1 and Pi3 Kinase.
@en
Concentration dependent dual e ...... ells via Par-1 and Pi3 Kinase.
@nl
prefLabel
Concentration dependent dual e ...... ells via Par-1 and Pi3 Kinase.
@en
Concentration dependent dual e ...... ells via Par-1 and Pi3 Kinase.
@nl
P2093
P2860
P356
P1476
Concentration dependent dual e ...... ells via Par-1 and Pi3 Kinase.
@en
P2093
Alireza R Rezaie
Jong-Sup Bae
Moon-Ki Park
Yong-Ung Kim
P2860
P304
P356
10.1002/JCP.21718
P577
2009-06-01T00:00:00Z