Mislocalized scaffolding by the Na-H exchanger NHE1 dominantly inhibits fibronectin production and TGF-beta activation. - Wikidata - awgldk - TriplyDB

Mislocalized scaffolding by the Na-H exchanger NHE1 dominantly inhibits fibronectin production and TGF-beta activation.

Mislocalized scaffolding by the Na-H exchanger NHE1 dominantly inhibits fibronectin production and TGF-beta activation.

http://www.wikidata.org/entity/Q37158427

about

A human MAP kinase interactome Considering protonation as a posttranslational modification regulating protein structure and function.Aberrant dynamin 2-dependent Na(+) /H(+) exchanger-1 trafficking contributes to cardiomyocyte apoptosis.Decreased tumor progression and invasion by a novel anti-cell motility target for human colorectal carcinoma cells.Hypoxia increases the abundance but not the assembly of extracellular fibronectin during epithelial cell transdifferentiation.Oxidative Stress-Activated NHE1 Is Involved in High Glucose-Induced Apoptosis in Renal Tubular Epithelial Cells pH sensing by FAK-His58 regulates focal adhesion remodeling Roles of ion transport in control of cell motility.Cell volume regulation in epithelial physiology and cancer Glioma-mediated microglial activation promotes glioma proliferation and migration: roles of Na+/H+ exchanger isoform 1.Cancer-associated arginine-to-histidine mutations confer a gain in pH sensing to mutant proteins.

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P2860

Mislocalized scaffolding by the Na-H exchanger NHE1 dominantly inhibits fibronectin production and TGF-beta activation.

http://www.wikidata.org/entity/Q37158427

description

article científic

@ca

article scientifique

@fr

articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 18 February 2009

@en

vedecký článok

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vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Mislocalized scaffolding by th ...... ction and TGF-beta activation.

@en

Mislocalized scaffolding by th ...... ction and TGF-beta activation.

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type

label

Mislocalized scaffolding by th ...... ction and TGF-beta activation.

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Mislocalized scaffolding by th ...... ction and TGF-beta activation.

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prefLabel

Mislocalized scaffolding by th ...... ction and TGF-beta activation.

@en

Mislocalized scaffolding by th ...... ction and TGF-beta activation.

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MBC.E08-08-0842

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Molecular Biology of the Cell

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Mislocalized scaffolding by th ...... ction and TGF-beta activation.

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Anastasios Karydis

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Diane L Barber

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Maite Jimenez-Vidal

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P2860

Beyond ion translocation: structural functions of the sodium-hydrogen exchanger isoform-1

Expression profile of genes regulated by activity of the Na-H exchanger NHE1

Caveolin-1 regulates transforming growth factor (TGF)-beta/SMAD signaling through an interaction with the TGF-beta type I receptor

Role of transforming growth factor beta in human disease

Direct binding of the Na--H exchanger NHE1 to ERM proteins regulates the cortical cytoskeleton and cell shape independently of H(+) translocation

Inhibition of transforming growth factor (TGF)-beta1-induced extracellular matrix with a novel inhibitor of the TGF-beta type I receptor kinase activity: SB-431542

Making sense of latent TGFbeta activation

Growth factors induce phosphorylation of the Na+/H+ antiporter, glycoprotein of 110 kD

Positive feedback between Cdc42 activity and H+ efflux by the Na-H exchanger NHE1 for polarity of migrating cells

Fibronectin matrix assembly requires distinct contributions from Rho kinases I and -II

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2327-2336

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scholarly article

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10.1091/MBC.E08-08-0842

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8

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Sheryl P. Denker

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2009-02-18T00:00:00Z

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